TL1A/DR3 axis involvement in the inflammatory cytokine network during pulmonary sarcoidosis
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F. Cinetto | F. Calzetti | M. Cassatella | C. Agostini | NICOLA TAMASSIA | F. Calabrese | M. Facco | A. Cabrelle | G. Semenzato | A. Teramo | S. Carraro | V. Martini
[1] M. Judson. The Clinical Features of Sarcoidosis: A Comprehensive Review , 2015, Clinical Reviews in Allergy & Immunology.
[2] M. Koenders,et al. The role of the Th17 cytokines IL-17 and IL-22 in Rheumatoid Arthritis pathogenesis and developments in cytokine immunotherapy. , 2015, Cytokine.
[3] R. Vandenbroucke,et al. Is there new hope for therapeutic matrix metalloproteinase inhibition? , 2014, Nature Reviews Drug Discovery.
[4] Hui Liu,et al. TNF-like ligand 1A is associated with the pathogenesis of psoriasis vulgaris and contributes to IL-17 production in PBMCs , 2014, Archives of Dermatological Research.
[5] R. Baughman,et al. Worsening of pulmonary sarcoidosis , 2014, Current opinion in pulmonary medicine.
[6] V. Kähäri,et al. Matrix metalloproteinases in inflammation. , 2014, Biochimica et biophysica acta.
[7] R. Polverosi,et al. Typical and atypical pattern of pulmonary sarcoidosis at high-resolution CT: relation to clinical evolution and therapeutic procedures , 2014, La radiologia medica.
[8] Kori L Wallace,et al. Immunopathology of inflammatory bowel disease. , 2014, World journal of gastroenterology.
[9] Minoru Nakamura,et al. The Role of TL1A and DR3 in Autoimmune and Inflammatory Diseases , 2013, Mediators of inflammation.
[10] E. Podack,et al. Immunobiology of TNFSF15 and TNFRSF25 , 2013, Immunologic research.
[11] L. Jia,et al. The tumor necrosis factor-like cytokine 1A/death receptor 3 cytokine system in intestinal inflammation , 2013, Current opinion in gastroenterology.
[12] W. Drake,et al. Reversal of Global CD4+ Subset Dysfunction Is Associated with Spontaneous Clinical Resolution of Pulmonary Sarcoidosis , 2013, The Journal of Immunology.
[13] X. Sun,et al. Elevated serum and synovial fluid TNF-like ligand 1A (TL1A) is associated with autoantibody production in patients with rheumatoid arthritis , 2013, Scandinavian journal of rheumatology.
[14] Arianne C. Richard,et al. TL1A and DR3, a TNF family ligand‐receptor pair that promotes lymphocyte costimulation, mucosal hyperplasia, and autoimmune inflammation , 2011, Immunological reviews.
[15] T. Ślebioda,et al. Sustained TL1A expression modulates effector and regulatory T-cell responses and drives intestinal goblet cell hyperplasia , 2011, Mucosal Immunology.
[16] C. Hunter,et al. Naive and activated T cells display differential responsiveness to TL1A that affects Th17 generation, maintenance, and proliferation , 2011, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[17] G. Fadini,et al. Sarcoidosis is a Th1/Th17 multisystem disorder , 2010, Thorax.
[18] T. Hibi,et al. TL1A produced by lamina propria macrophages induces Th1 and Th17 immune responses in cooperation with IL‐23 in patients with Crohn's disease , 2010, Inflammatory bowel diseases.
[19] M. Oukka. Th17 cells in immunity and autoimmunity , 2008, Annals of the rheumatic diseases.
[20] M. Cassatella,et al. Pulmonary Sarcoidosis , 2007, Seminars in respiratory and critical care medicine.
[21] A. Mantovani,et al. Soluble TNF-Like Cytokine (TL1A) Production by Immune Complexes Stimulated Monocytes in Rheumatoid Arthritis1 , 2007, The Journal of Immunology.
[22] F. Cominelli,et al. Role of TL1A and its receptor DR3 in two models of chronic murine ileitis. , 2006, Proceedings of the National Academy of Sciences of the United States of America.
[23] B. Beghé,et al. Role for CXCR6 and its ligand CXCL16 in the pathogenesis of T-cell alveolitis in sarcoidosis. , 2005, American journal of respiratory and critical care medicine.
[24] B. Kwon,et al. Involvement of TL1A and DR3 in induction of pro-inflammatory cytokines and matrix metalloproteinase-9 in atherogenesis. , 2005, Cytokine.
[25] 修一 野村,et al. 亜鉛不足が原因の味覚障害患者に亜鉛の経口投与で唾液中のTIMP-1 (tissue inhibitor of metalloproteinase-1) , 2003 .
[26] K. Prikk,et al. Matrix metalloproteinases and tissue inhibitor of metalloproteinase-1 in sarcoidosis and IPF , 2002, European Respiratory Journal.
[27] Jun Zhang,et al. TL1A is a TNF-like ligand for DR3 and TR6/DcR3 and functions as a T cell costimulator. , 2002, Immunity.
[28] J. Bousquet,et al. Balance in asthma between matrix metalloproteinases and their inhibitors. , 1999, The Journal of allergy and clinical immunology.
[29] L. Trentin,et al. Regulation of alveolar macrophage-T cell interactions during Th1-type sarcoid inflammatory process. , 1999, American journal of physiology. Lung cellular and molecular physiology.
[30] M. Chilosi,et al. Involvement of the IP-10 chemokine in sarcoid granulomatous reactions. , 1998, Journal of immunology.
[31] N. MacIntyre,et al. Diffusing capacity of the lung for carbon monoxide. , 1997, Respiratory care clinics of North America.
[32] R. Zambello,et al. Role of IL-15, IL-2, and their receptors in the development of T cell alveolitis in pulmonary sarcoidosis. , 1996, Journal of immunology.
[33] L. Trentin,et al. CD8 alveolitis in sarcoidosis: incidence, phenotypic characteristics, and clinical features. , 1993, The American journal of medicine.
[34] D. Casara,et al. Pulmonary alveolar macrophages from patients with active sarcoidosis express type IV collagenolytic proteinase. An enzymatic mechanism for influx of mononuclear phagocytes at sites of disease activity. , 1989, The Journal of clinical investigation.
[35] O. Sharma,et al. Pathogenesis of sarcoidosis. , 1987, The Western journal of medicine.
[36] W. Shelley. On the Pathogenesis of Sarcoidosis , 1971, Southern medical journal.
[37] Y. C. Lee,et al. Imbalance between matrix metalloproteinase-9 and tissue inhibitor of metalloproteinase-1 in toluene diisocyanate-induced asthma. , 2004, Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology.