CD44-deficiency on hematopoietic cells limits T-cell number but does not protect against atherogenesis in LDL receptor-deficient mice.

[1]  D. Rader,et al.  CD44 Expressed on Both Bone Marrow–Derived and Non–Bone Marrow–Derived Cells Promotes Atherogenesis in ApoE-Deficient Mice , 2008, Arteriosclerosis, thrombosis, and vascular biology.

[2]  Yun‐Sil Lee,et al.  Hyaluronic acid targets CD44 and inhibits FcepsilonRI signaling involving PKCdelta, Rac1, ROS, and MAPK to exert anti-allergic effect. , 2008, Molecular immunology.

[3]  B. Coll,et al.  The results in rodent models of atherosclerosis are not interchangeable: the influence of diet and strain. , 2007, Atherosclerosis.

[4]  J. Nettleship,et al.  Physiological Testosterone Replacement Therapy Attenuates Fatty Streak Formation and Improves High-Density Lipoprotein Cholesterol in the Tfm Mouse: An Effect That Is Independent of the Classic Androgen Receptor , 2007, Circulation.

[5]  J. Borén,et al.  Retention of Low-Density Lipoprotein in Atherosclerotic Lesions of the Mouse: Evidence for a Role of Lipoprotein Lipase , 2007, Circulation research.

[6]  D. Rader,et al.  CD44 Regulates Vascular Gene Expression in a Proatherogenic Environment , 2007, Arteriosclerosis, thrombosis, and vascular biology.

[7]  R. Bucala,et al.  CD44 Is a Negative Regulator of Acute Pulmonary Inflammation and Lipopolysaccharide-TLR Signaling in Mouse Macrophages1 , 2007, The Journal of Immunology.

[8]  Christian Weber,et al.  Ccr5 But Not Ccr1 Deficiency Reduces Development of Diet-Induced Atherosclerosis in Mice , 2006, Arteriosclerosis, thrombosis, and vascular biology.

[9]  A. Tedgui,et al.  Role of Bone Marrow–Derived CC-Chemokine Receptor 5 in the Development of Atherosclerosis of Low-Density Lipoprotein Receptor Knockout Mice , 2006, Arteriosclerosis, thrombosis, and vascular biology.

[10]  A. Zernecke,et al.  Deficiency in CCR5 but not CCR1 protects against neointima formation in atherosclerosis-prone mice: involvement of IL-10. , 2006, Blood.

[11]  D. Accili,et al.  Macrophage insulin receptor deficiency increases ER stress-induced apoptosis and necrotic core formation in advanced atherosclerotic lesions. , 2006, Cell metabolism.

[12]  P. Libby,et al.  Cystatin C Deficiency Increases Elastic Lamina Degradation and Aortic Dilatation in Apolipoprotein E–Null Mice , 2005, Circulation research.

[13]  Adil I. Khan,et al.  Role of CD44 and Hyaluronan in Neutrophil Recruitment1 , 2004, The Journal of Immunology.

[14]  P. Libby,et al.  Enhanced expression of CD44 variants in human atheroma and abdominal aortic aneurysm: possible role for a feedback loop in endothelial cells. , 2004, The American journal of pathology.

[15]  J. Borén,et al.  Lack of Complement Factor C3, but Not Factor B, Increases Hyperlipidemia and Atherosclerosis in Apolipoprotein E−/− Low-Density Lipoprotein Receptor−/− Mice , 2004, Arteriosclerosis, thrombosis, and vascular biology.

[16]  K. Reue,et al.  Cholesterol and Cholate Components of an Atherogenic Diet Induce Distinct Stages of Hepatic Inflammatory Gene Expression* , 2003, Journal of Biological Chemistry.

[17]  Georg Kraal,et al.  Inhibition of NF-kappaB activation in macrophages increases atherosclerosis in LDL receptor-deficient mice. , 2003, The Journal of clinical investigation.

[18]  J. Sleeman,et al.  Targeting dendritic cells with CD44 monoclonal antibodies selectively inhibits the proliferation of naive CD4+ T‐helper cells by induction of FAS‐independent T‐cell apoptosis , 2003, Immunology.

[19]  S. Hart,et al.  Role of macrophage CD44 in the disposal of inflammatory cell corpses. , 2002, Clinical science.

[20]  D. Rader,et al.  The adhesion receptor CD44 promotes atherosclerosis by mediating inflammatory cell recruitment and vascular cell activation. , 2001, The Journal of clinical investigation.

[21]  L. Lindbom,et al.  Importance of Primary Capture and L-Selectin–Dependent Secondary Capture in Leukocyte Accumulation in Inflammation and Atherosclerosis in Vivo , 2001, The Journal of experimental medicine.

[22]  M. Zöller,et al.  CD44 supports T cell proliferation and apoptosis by apposition of protein kinases , 2000, European journal of immunology.

[23]  L. Curtiss,et al.  Apolipoprotein E and atherosclerosis. , 2000, Current opinion in lipidology.

[24]  R. Ross,et al.  Proteoglycan distribution in lesions of atherosclerosis depends on lesion severity, structural characteristics, and the proximity of platelet-derived growth factor and transforming growth factor-beta. , 1998, The American journal of pathology.

[25]  M. Burdick,et al.  Hyaluronan (HA) fragments induce chemokine gene expression in alveolar macrophages. The role of HA size and CD44. , 1996, The Journal of clinical investigation.

[26]  Y. Wu,et al.  Identification of a costimulatory molecule rapidly induced by CD40L as CD44H , 1996, The Journal of experimental medicine.

[27]  Jonathan D. Smith,et al.  Apolipoprotein E allele–specific antioxidant activity and effects on cytotoxicity by oxidative insults and β–amyloid peptides , 1996, Nature Genetics.

[28]  L. Curtiss,et al.  Apolipoprotein E is a biologically active constituent of the normal immunoregulatory lipoprotein, LDL-In. , 1986, Journal of immunology.

[29]  B. Fagerberg,et al.  Augmented levels of CD44 in macrophages from atherosclerotic subjects: a possible IL-6-CD44 feedback loop? , 2007, Atherosclerosis.

[30]  K. Gee,et al.  Recent advances in the regulation of CD44 expression and its role in inflammation and autoimmune diseases. , 2004, Archivum immunologiae et therapiae experimentalis.