Estimation of the multiple testing burden for genomewide association studies of nearly all common variants
暂无分享,去创建一个
[1] Z. Šidák. Rectangular Confidence Regions for the Means of Multivariate Normal Distributions , 1967 .
[2] E. Lander,et al. Genetic dissection of complex traits: guidelines for interpreting and reporting linkage results , 1995, Nature Genetics.
[3] N Risch,et al. The Future of Genetic Studies of Complex Human Diseases , 1996, Science.
[4] P. Donnelly,et al. Inferring coalescence times from DNA sequence data. , 1997, Genetics.
[5] M. Daly,et al. Linkage thresholds for two-stage genome scans. , 1998, American journal of human genetics.
[6] †The International HapMap Consortium. The International HapMap Project , 2003, Nature.
[7] Aravinda Chakravarti,et al. Exhaustive allelic transmission disequilibrium tests as a new approach to genome-wide association studies , 2004, Nature Genetics.
[8] Frank Dudbridge,et al. Efficient computation of significance levels for multiple associations in large studies of correlated data, including genomewide association studies. , 2004, American journal of human genetics.
[9] D. Thomas,et al. Two‐Stage sampling designs for gene association studies , 2004, Genetic epidemiology.
[10] S. Gabriel,et al. Efficiency and power in genetic association studies , 2005, Nature Genetics.
[11] M. Daly,et al. Genome-wide association studies for common diseases and complex traits , 2005, Nature Reviews Genetics.
[12] M. Olivier. A haplotype map of the human genome , 2003, Nature.
[13] M. Olivier. A haplotype map of the human genome. , 2003, Nature.
[14] J. Ott,et al. Complement Factor H Polymorphism in Age-Related Macular Degeneration , 2005, Science.
[15] Frank Dudbridge. A note on permutation tests in multistage association scans. , 2006, American journal of human genetics.
[16] M. Daly,et al. Evaluating and improving power in whole-genome association studies using fixed marker sets , 2006, Nature Genetics.
[17] Lon R Cardon,et al. Evaluating coverage of genome-wide association studies , 2006, Nature Genetics.
[18] Christian Gieger,et al. A common genetic variant in the NOS1 regulator NOS1AP modulates cardiac repolarization , 2006, Nature Genetics.
[19] M. Daly,et al. Biases and reconciliation in estimates of linkage disequilibrium in the human genome. , 2006, American journal of human genetics.
[20] G. Abecasis,et al. Joint analysis is more efficient than replication-based analysis for two-stage genome-wide association studies , 2006, Nature Genetics.
[21] M. McCarthy,et al. Replication of Genome-Wide Association Signals in UK Samples Reveals Risk Loci for Type 2 Diabetes , 2007, Science.
[22] William Stafford Noble,et al. Identification and analysis of functional elements in 1% of the human genome by the ENCODE pilot project , 2007, Nature.
[23] Marcia M. Nizzari,et al. Genome-Wide Association Analysis Identifies Loci for Type 2 Diabetes and Triglyceride Levels , 2007, Science.
[24] Simon C. Potter,et al. Genome-wide association study of 14,000 cases of seven common diseases and 3,000 shared controls , 2007, Nature.
[25] G. Abecasis,et al. A Genome-Wide Association Study of Type 2 Diabetes in Finns Detects Multiple Susceptibility Variants , 2007, Science.