Autophagy contributes to inflammation in patients with TNFR-associated periodic syndrome (TRAPS)
暂无分享,去创建一个
A. Ballabio | R. Ravazzolo | D. Bani | P. Castagnola | A. Fraldi | A. Martini | E. Di Zanni | I. Ceccherini | T. Bachetti | A. Omenetti | M. Gattorno | S. Chiesa | A. D’Osualdo
[1] A. Sher,et al. Activation of autophagy by inflammatory signals limits IL-1β production by targeting ubiquitinated inflammasomes for destruction , 2012, Nature Immunology.
[2] R. Ravazzolo,et al. In vitro drug treatments reduce the deleterious effects of aggregates containing polyAla expanded PHOX2B proteins , 2012, Neurobiology of Disease.
[3] M. D. Turner,et al. Tumour necrosis factor receptor trafficking dysfunction opens the TRAPS door to pro-inflammatory cytokine secretion , 2011, Bioscience reports.
[4] E. Di Zanni,et al. Toward a therapeutic strategy for polyalanine expansions disorders: in vivo and in vitro models for drugs analysis. , 2011, European journal of paediatric neurology : EJPN : official journal of the European Paediatric Neurology Society.
[5] Ralph A. Nixon,et al. Autophagy failure in Alzheimer's disease—locating the primary defect , 2011, Neurobiology of Disease.
[6] M. Ohno,et al. Therapeutic effects of remediating autophagy failure in a mouse model of Alzheimer disease by enhancing lysosomal proteolysis , 2011, Autophagy.
[7] Oliver Sawodny,et al. Modeling the TNFα-Induced Apoptosis Pathway in Hepatocytes , 2011, PloS one.
[8] M. Sormani,et al. Long-Term Clinical Profile of Children With the Low-Penetrance R92Q Mutation of the TNFRSF1A Gene , 2011, Arthritis and rheumatism.
[9] Daniel L. Kastner,et al. Mitochondrial reactive oxygen species promote production of proinflammatory cytokines and are elevated in TNFR1-associated periodic syndrome (TRAPS) , 2011, The Journal of experimental medicine.
[10] S. Ryter,et al. Autophagy proteins regulate innate immune responses by inhibiting the release of mitochondrial DNA mediated by the NALP3 inflammasome. , 2011, Nature immunology.
[11] I. Kourtzelis,et al. Evidence for the involvement of mTOR inhibition and basal autophagy in familial Mediterranean fever phenotype. , 2011, Human immunology.
[12] J. Tschopp,et al. Autophagy Controls IL-1β Secretion by Targeting Pro-IL-1β for Degradation , 2011, The Journal of Biological Chemistry.
[13] G. Hitman,et al. Differential cytokine secretion results from p65 and c-Rel NF-κB subunit signaling in peripheral blood mononuclear cells of TNF receptor-associated periodic syndrome patients. , 2011, Cellular immunology.
[14] A. Ballabio,et al. Defective CFTR induces aggresome formation and lung inflammation in cystic fibrosis through ROS-mediated autophagy inhibition , 2010, Nature Cell Biology.
[15] E. Di Zanni,et al. In vitro treatments with ceftriaxone promote elimination of mutant glial fibrillary acidic protein and transcription down-regulation. , 2010, Experimental cell research.
[16] D. Kastner,et al. Concerted action of wild-type and mutant TNF receptors enhances inflammation in TNF receptor 1-associated periodic fever syndrome , 2010, Proceedings of the National Academy of Sciences.
[17] D. Sulzer,et al. CARGO RECOGNITION FAILURE IS RESPONSIBLE FOR INEFFICIENT AUTOPHAGY IN HUNTINGTON’S DISEASE , 2010, Nature Neuroscience.
[18] K. Wada,et al. Induction of molecular chaperones as a therapeutic strategy for the polyglutamine diseases. , 2010, Current pharmaceutical biotechnology.
[19] Valerio Embrione,et al. A Gene Network Regulating Lysosomal Biogenesis and Function , 2009, Science.
[20] M. Diaz-Meco,et al. p62 at the Crossroads of Autophagy, Apoptosis, and Cancer , 2009, Cell.
[21] S. Akira,et al. Loss of the autophagy protein Atg16L1 enhances endotoxin-induced IL-1β production , 2008, Nature.
[22] 井田 弘明,et al. 小児医学最近の進歩 TNF receptor-associated periodic syndrome (TRAPS)の全国疫学調査 , 2008 .
[23] D. Mann,et al. Oxidative Stress Promotes Ligand-independent and Enhanced Ligand-dependent Tumor Necrosis Factor Receptor Signaling* , 2008, Journal of Biological Chemistry.
[24] A. Plebani,et al. Persistent efficacy of anakinra in patients with tumor necrosis factor receptor-associated periodic syndrome. , 2008, Arthritis and rheumatism.
[25] T. Suuronen,et al. Innate immunity meets with cellular stress at the IKK complex: regulation of the IKK complex by HSP70 and HSP90. , 2008, Immunology letters.
[26] T. Ueno,et al. Consideration about negative controls for LC3 and expression vectors for four colored fluorescent protein-LC3 negative controls , 2008, Autophagy.
[27] G. Hitman,et al. A novel TNFRSF1A splice mutation associated with increased nuclear factor κappaB (NF-κB) transcription factor activation in patients with tumour necrosis factor receptor associated periodic syndrome (TRAPS) , 2007, Annals of the rheumatic diseases.
[28] G. Hitman,et al. Abnormal tumor necrosis factor receptor I cell surface expression and NF-kappaB activation in tumor necrosis factor receptor-associated periodic syndrome. , 2008, Arthritis and rheumatism.
[29] A. Martini,et al. Pattern of interleukin-1beta secretion in response to lipopolysaccharide and ATP before and after interleukin-1 blockade in patients with CIAS1 mutations. , 2007, Arthritis and rheumatism.
[30] D. Stolz,et al. Linking of autophagy to ubiquitin-proteasome system is important for the regulation of endoplasmic reticulum stress and cell viability. , 2007, The American journal of pathology.
[31] Tariq Ahmad,et al. Confirmation of the role of ATG16l1 as a Crohn's disease susceptibility gene , 2007, Inflammatory bowel diseases.
[32] P. Tighe,et al. Mutant tumor necrosis factor receptor associated with tumor necrosis factor receptor-associated periodic syndrome is altered antigenically and is retained within patients' leukocytes. , 2007, Arthritis and rheumatism.
[33] C. Bauvy,et al. Regulation of Autophagy by NF-kappaB Transcription Factor and Reactives Oxygen Species , 2007, Autophagy.
[34] C. Cheng,et al. Geldanamycin Interferes with the 90-kDa Heat Shock Protein, Affecting Lipopolysaccharide-Mediated Interleukin-1 Expression and Apoptosis within Macrophages , 2007, Molecular Pharmacology.
[35] R. Ravazzolo,et al. Geldanamycin promotes nuclear localisation and clearance of PHOX2B misfolded proteins containing polyalanine expansions. , 2007, The international journal of biochemistry & cell biology.
[36] G. Xiao,et al. Hsp90 inhibition results in autophagy-mediated proteasome-independent degradation of IκB kinase (IKK) , 2006, Cell Research.
[37] Dangsheng Li. Selective degradation of the IκB kinase (IKK) by autophagy , 2006, Cell Research.
[38] D. Kastner,et al. Abnormal disulfide-linked oligomerization results in ER retention and altered signaling by TNFR1 mutants in TNFR1-associated periodic fever syndrome (TRAPS). , 2006, Blood.
[39] P. Tighe,et al. Modeling of tumor necrosis factor receptor superfamily 1A mutants associated with tumor necrosis factor receptor-associated periodic syndrome indicates misfolding consistent with abnormal function. , 2006, Arthritis and rheumatism.
[40] R. Ravazzolo,et al. Neutrophils from patients with TNFRSF1A mutations display resistance to tumor necrosis factor-induced apoptosis: pathogenetic and clinical implications. , 2006, Arthritis and rheumatism.
[41] Dangsheng Li. Selective degradation of the IkappaB kinase (IKK) by autophagy. , 2006, Cell research.
[42] Daniel J. Klionsky,et al. Autophagy in Health and Disease: A Double-Edged Sword , 2004, Science.
[43] J. Tschopp,et al. Life and death decisions: secondary complexes and lipid rafts in TNF receptor family signal transduction. , 2004, Immunity.
[44] James Lowe,et al. Role of ubiquitin-mediated proteolysis in the pathogenesis of neurodegenerative disorders , 2003, Ageing Research Reviews.
[45] J. Tschopp,et al. Induction of TNF Receptor I-Mediated Apoptosis via Two Sequential Signaling Complexes , 2003, Cell.
[46] D. Kastner,et al. The TNF Receptor-Associated Periodic Syndrome (TRAPS): Emerging Concepts of an Autoinflammatory Disorder , 2002, Medicine.
[47] J. Piette,et al. The enlarging clinical, genetic, and population spectrum of tumor necrosis factor receptor-associated periodic syndrome. , 2002, Arthritis and rheumatism.
[48] D. Kastner,et al. The tumor-necrosis-factor receptor-associated periodic syndrome: new mutations in TNFRSF1A, ancestral origins, genotype-phenotype studies, and evidence for further genetic heterogeneity of periodic fevers. , 2001, American journal of human genetics.
[49] D. Kastner,et al. TNFRSF1A mutations and autoinflammatory syndromes. , 2000, Current opinion in immunology.
[50] Ian Todd,et al. Germline Mutations in the Extracellular Domains of the 55 kDa TNF Receptor, TNFR1, Define a Family of Dominantly Inherited Autoinflammatory Syndromes , 1999, Cell.
[51] P. Allen,et al. Inhibition of autophagy abrogates tumour necrosis factor α induced apoptosis in human T‐lymphoblastic leukaemic cells , 1997, British journal of haematology.
[52] P. Toghill,et al. Familial Hibernian fever. , 1982, The Quarterly journal of medicine.