Long noncoding RNA MAPKAPK5-AS1 promoted lipopolysaccharide-induced inflammatory damage in the myocardium by sponging microRNA-124-3p/E2F3
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Weiwei Chen | Guangyuan Gao | M. Yan | Ming Yu | Kaiyao Shi | P. Yang | Ping Yang
[1] Yan Zhou,et al. IncRNA MAPKAPK5-AS1 promotes proliferation and migration of thyroid cancer cell lines by targeting miR-519e-5p/YWHAH , 2020, European journal of histochemistry : EJH.
[2] D. Pei,et al. Long noncoding RNA MAPKAPK5-AS1 promotes colorectal cancer progression by cis-regulating the nearby gene MK5 and acting as a let-7f-1-3p sponge , 2020, Journal of Experimental & Clinical Cancer Research.
[3] Ying Han,et al. lncRNA RMRP Prevents Mitochondrial Dysfunction and Cardiomyocyte Apoptosis via the miR-1-5p/hsp70 Axis in LPS-Induced Sepsis Mice , 2020, Inflammation.
[4] Xinhua Zhang,et al. Knockdown of lncRNA HOXA-AS2 Inhibits Viability, Migration and Invasion of Osteosarcoma Cells by miR-124-3p/E2F3 , 2019, OncoTargets and therapy.
[5] C. Jung,et al. MicroRNAs in Inflammatory Heart Diseases and Sepsis-Induced Cardiac Dysfunction: A Potential Scope for the Future? , 2019, Cells.
[6] T. Sun,et al. FAM46C inhibits lipopolysaccharides-induced myocardial dysfunction via downregulating cellular adhesion molecules and inhibiting apoptosis. , 2019, Life sciences.
[7] Y. Long,et al. Role of the mTOR Signalling Pathway in Human Sepsis-Induced Myocardial Dysfunction. , 2019, The Canadian journal of cardiology.
[8] Hongyan Zhang,et al. Identification of lung-adenocarcinoma-related long non-coding RNAs by random walking on a competing endogenous RNA network. , 2019, Annals of translational medicine.
[9] Jian-song Lin,et al. Role of TXNIP/NLRP3 in sepsis-induced myocardial dysfunction , 2019, International journal of molecular medicine.
[10] X. Teng,et al. Hydrogen Sulfide Protects Against Sepsis‐Induced Myocardial Dysfunction by Inhibiting inflammation and Endoplasmic Reticulum Stress , 2019, The FASEB Journal.
[11] Wen-jie Chen,et al. An autophagy‐related long non‐coding RNA signature for glioma , 2019, FEBS open bio.
[12] C. Sohn,et al. Troponin Testing for Assessing Sepsis-Induced Myocardial Dysfunction in Patients with Septic Shock , 2019, Journal of clinical medicine.
[13] Dinesh Kumar Yadav,et al. Prevalence and Outcome of Sepsis-induced Myocardial Dysfunction in Children with ‘Sepsis’ ‘With’ and ‘Without Shock’—A Prospective Observational Study , 2018, Journal of tropical pediatrics.
[14] W. Zollitsch,et al. Waste management in recirculating aquaculture system through bacteria dissimilation and plant assimilation , 2018, Aquaculture International.
[15] Cuiyu Huang,et al. Circulating lncRNA NEAT1 correlates with increased risk, elevated severity and unfavorable prognosis in sepsis patients , 2018, The American journal of emergency medicine.
[16] Lu Zhang,et al. LncRNA H19 functions as an Aquaporin 1 competitive endogenous RNA to regulate microRNA-874 expression in LPS sepsis. , 2018, Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie.
[17] Jian Xu,et al. miR-146a Attenuates Sepsis-Induced Myocardial Dysfunction by Suppressing IRAK1 and TRAF6 via Targeting ErbB4 Expression , 2018, Oxidative medicine and cellular longevity.
[18] K. Walley. Sepsis-induced myocardial dysfunction , 2018, Current opinion in critical care.
[19] M. Shehata,et al. The role of brain natriuretic peptide in correlation to mitral annular plane systolic excursion in predicting sepsis-induced myocardial dysfunction , 2018 .
[20] Xiang-Hu He,et al. LncRNA MALAT1 regulates sepsis‐induced cardiac inflammation and dysfunction via interaction with miR‐125b and p38 MAPK/NF&kgr;B , 2018, International immunopharmacology.
[21] G. Fan,et al. MiR-210 protects cardiomyocytes from OGD/R injury by inhibiting E2F3. , 2018, European review for medical and pharmacological sciences.
[22] S. Kannan,et al. PUB004 Inhibition of iPLA 2 Regulates E2F3 Pathway and Cell Cycle Checkpoints to Induce Apoptosis in Non-Small Cell Lung Cancer Cells , 2017 .
[23] W. Du,et al. Rb‐independent E2F3 promotes cell proliferation and alters expression of genes involved in metabolism and inflammation , 2017, FEBS open bio.
[24] Song-Tao Shou,et al. Sepsis-Induced Cardiomyopathy: Mechanisms and Treatments , 2017, Front. Immunol..
[25] Junhang Liu,et al. miRNA-214 Protects Sepsis-Induced Myocardial Injury , 2017, Shock.
[26] Kaijiang Yu,et al. Long non‐coding RNA PVT1 promote LPS‐induced septic acute kidney injury by regulating TNF&agr; and JNK/NF‐&kgr;B pathways in HK‐2 cells , 2017, International immunopharmacology.
[27] Yumao Huang,et al. MicroRNA‐135a is up‐regulated and aggravates myocardial depression in sepsis via regulating p38 MAPK/NF‐&kgr;B pathway , 2017, International immunopharmacology.
[28] F. Gao,et al. Interleukin-27 is elevated in sepsis-induced myocardial dysfunction and mediates inflammation. , 2016, Cytokine.
[29] J. Kalbfleisch,et al. MicroRNA-125b Prevents Cardiac Dysfunction in Polymicrobial Sepsis by Targeting TRAF6-Mediated Nuclear Factor κB Activation and p53-Mediated Apoptotic Signaling. , 2016, The Journal of infectious diseases.
[30] Xinli Li,et al. Inhibition of miR-155 Protects Against LPS-induced Cardiac Dysfunction and Apoptosis in Mice , 2016, Molecular therapy. Nucleic acids.
[31] Xiuxiu Lv,et al. Pathophysiology of sepsis-induced myocardial dysfunction , 2016, Military Medical Research.
[32] Takashi Ito,et al. Sepsis-induced myocardial dysfunction: pathophysiology and management , 2016, Journal of Intensive Care.
[33] Hongwei Wu,et al. LncRNA-HOTAIR promotes TNF-α production in cardiomyocytes of LPS-induced sepsis mice by activating NF-κB pathway. , 2016, Biochemical and biophysical research communications.
[34] Maria-Teresa Piccoli,et al. Noncoding RNAs as regulators of cardiomyocyte proliferation and death. , 2015, Journal of molecular and cellular cardiology.
[35] Masaharu Kataoka,et al. Non-Coding RNAs Including miRNAs and lncRNAs in Cardiovascular Biology and Disease , 2014, Cells.
[36] Ferhaan Ahmad,et al. E2F3 plays an essential role in cardiac development and function , 2008, Cell cycle.
[37] Avadhesh C. Sharma. SEPSIS-INDUCED MYOCARDIAL DYSFUNCTION , 2006, Shock.
[38] J. Nevins,et al. E2F: a link between the Rb tumor suppressor protein and viral oncoproteins. , 1992, Science.
[39] S. Weintraub,et al. Retinoblastoma protein switches the E2F site from positive to negative element , 1992, Nature.
[40] Qinghua Wu,et al. Infusion of esmolol attenuates lipopolysaccharide-induced myocardial dysfunction. , 2016, The Journal of surgical research.