Long noncoding RNA MAPKAPK5-AS1 promoted lipopolysaccharide-induced inflammatory damage in the myocardium by sponging microRNA-124-3p/E2F3

[1]  Yan Zhou,et al.  IncRNA MAPKAPK5-AS1 promotes proliferation and migration of thyroid cancer cell lines by targeting miR-519e-5p/YWHAH , 2020, European journal of histochemistry : EJH.

[2]  D. Pei,et al.  Long noncoding RNA MAPKAPK5-AS1 promotes colorectal cancer progression by cis-regulating the nearby gene MK5 and acting as a let-7f-1-3p sponge , 2020, Journal of Experimental & Clinical Cancer Research.

[3]  Ying Han,et al.  lncRNA RMRP Prevents Mitochondrial Dysfunction and Cardiomyocyte Apoptosis via the miR-1-5p/hsp70 Axis in LPS-Induced Sepsis Mice , 2020, Inflammation.

[4]  Xinhua Zhang,et al.  Knockdown of lncRNA HOXA-AS2 Inhibits Viability, Migration and Invasion of Osteosarcoma Cells by miR-124-3p/E2F3 , 2019, OncoTargets and therapy.

[5]  C. Jung,et al.  MicroRNAs in Inflammatory Heart Diseases and Sepsis-Induced Cardiac Dysfunction: A Potential Scope for the Future? , 2019, Cells.

[6]  T. Sun,et al.  FAM46C inhibits lipopolysaccharides-induced myocardial dysfunction via downregulating cellular adhesion molecules and inhibiting apoptosis. , 2019, Life sciences.

[7]  Y. Long,et al.  Role of the mTOR Signalling Pathway in Human Sepsis-Induced Myocardial Dysfunction. , 2019, The Canadian journal of cardiology.

[8]  Hongyan Zhang,et al.  Identification of lung-adenocarcinoma-related long non-coding RNAs by random walking on a competing endogenous RNA network. , 2019, Annals of translational medicine.

[9]  Jian-song Lin,et al.  Role of TXNIP/NLRP3 in sepsis-induced myocardial dysfunction , 2019, International journal of molecular medicine.

[10]  X. Teng,et al.  Hydrogen Sulfide Protects Against Sepsis‐Induced Myocardial Dysfunction by Inhibiting inflammation and Endoplasmic Reticulum Stress , 2019, The FASEB Journal.

[11]  Wen-jie Chen,et al.  An autophagy‐related long non‐coding RNA signature for glioma , 2019, FEBS open bio.

[12]  C. Sohn,et al.  Troponin Testing for Assessing Sepsis-Induced Myocardial Dysfunction in Patients with Septic Shock , 2019, Journal of clinical medicine.

[13]  Dinesh Kumar Yadav,et al.  Prevalence and Outcome of Sepsis-induced Myocardial Dysfunction in Children with ‘Sepsis’ ‘With’ and ‘Without Shock’—A Prospective Observational Study , 2018, Journal of tropical pediatrics.

[14]  W. Zollitsch,et al.  Waste management in recirculating aquaculture system through bacteria dissimilation and plant assimilation , 2018, Aquaculture International.

[15]  Cuiyu Huang,et al.  Circulating lncRNA NEAT1 correlates with increased risk, elevated severity and unfavorable prognosis in sepsis patients , 2018, The American journal of emergency medicine.

[16]  Lu Zhang,et al.  LncRNA H19 functions as an Aquaporin 1 competitive endogenous RNA to regulate microRNA-874 expression in LPS sepsis. , 2018, Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie.

[17]  Jian Xu,et al.  miR-146a Attenuates Sepsis-Induced Myocardial Dysfunction by Suppressing IRAK1 and TRAF6 via Targeting ErbB4 Expression , 2018, Oxidative medicine and cellular longevity.

[18]  K. Walley Sepsis-induced myocardial dysfunction , 2018, Current opinion in critical care.

[19]  M. Shehata,et al.  The role of brain natriuretic peptide in correlation to mitral annular plane systolic excursion in predicting sepsis-induced myocardial dysfunction , 2018 .

[20]  Xiang-Hu He,et al.  LncRNA MALAT1 regulates sepsis‐induced cardiac inflammation and dysfunction via interaction with miR‐125b and p38 MAPK/NF&kgr;B , 2018, International immunopharmacology.

[21]  G. Fan,et al.  MiR-210 protects cardiomyocytes from OGD/R injury by inhibiting E2F3. , 2018, European review for medical and pharmacological sciences.

[22]  S. Kannan,et al.  PUB004 Inhibition of iPLA 2 Regulates E2F3 Pathway and Cell Cycle Checkpoints to Induce Apoptosis in Non-Small Cell Lung Cancer Cells , 2017 .

[23]  W. Du,et al.  Rb‐independent E2F3 promotes cell proliferation and alters expression of genes involved in metabolism and inflammation , 2017, FEBS open bio.

[24]  Song-Tao Shou,et al.  Sepsis-Induced Cardiomyopathy: Mechanisms and Treatments , 2017, Front. Immunol..

[25]  Junhang Liu,et al.  miRNA-214 Protects Sepsis-Induced Myocardial Injury , 2017, Shock.

[26]  Kaijiang Yu,et al.  Long non‐coding RNA PVT1 promote LPS‐induced septic acute kidney injury by regulating TNF&agr; and JNK/NF‐&kgr;B pathways in HK‐2 cells , 2017, International immunopharmacology.

[27]  Yumao Huang,et al.  MicroRNA‐135a is up‐regulated and aggravates myocardial depression in sepsis via regulating p38 MAPK/NF‐&kgr;B pathway , 2017, International immunopharmacology.

[28]  F. Gao,et al.  Interleukin-27 is elevated in sepsis-induced myocardial dysfunction and mediates inflammation. , 2016, Cytokine.

[29]  J. Kalbfleisch,et al.  MicroRNA-125b Prevents Cardiac Dysfunction in Polymicrobial Sepsis by Targeting TRAF6-Mediated Nuclear Factor κB Activation and p53-Mediated Apoptotic Signaling. , 2016, The Journal of infectious diseases.

[30]  Xinli Li,et al.  Inhibition of miR-155 Protects Against LPS-induced Cardiac Dysfunction and Apoptosis in Mice , 2016, Molecular therapy. Nucleic acids.

[31]  Xiuxiu Lv,et al.  Pathophysiology of sepsis-induced myocardial dysfunction , 2016, Military Medical Research.

[32]  Takashi Ito,et al.  Sepsis-induced myocardial dysfunction: pathophysiology and management , 2016, Journal of Intensive Care.

[33]  Hongwei Wu,et al.  LncRNA-HOTAIR promotes TNF-α production in cardiomyocytes of LPS-induced sepsis mice by activating NF-κB pathway. , 2016, Biochemical and biophysical research communications.

[34]  Maria-Teresa Piccoli,et al.  Noncoding RNAs as regulators of cardiomyocyte proliferation and death. , 2015, Journal of molecular and cellular cardiology.

[35]  Masaharu Kataoka,et al.  Non-Coding RNAs Including miRNAs and lncRNAs in Cardiovascular Biology and Disease , 2014, Cells.

[36]  Ferhaan Ahmad,et al.  E2F3 plays an essential role in cardiac development and function , 2008, Cell cycle.

[37]  Avadhesh C. Sharma SEPSIS-INDUCED MYOCARDIAL DYSFUNCTION , 2006, Shock.

[38]  J. Nevins,et al.  E2F: a link between the Rb tumor suppressor protein and viral oncoproteins. , 1992, Science.

[39]  S. Weintraub,et al.  Retinoblastoma protein switches the E2F site from positive to negative element , 1992, Nature.

[40]  Qinghua Wu,et al.  Infusion of esmolol attenuates lipopolysaccharide-induced myocardial dysfunction. , 2016, The Journal of surgical research.