Follistatin-Like 1 Is an Akt-Regulated Cardioprotective Factor That Is Secreted by the Heart
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[1] S. Houser,et al. Pim-1 regulates cardiomyocyte survival downstream of Akt , 2006, Nature Medicine.
[2] Y. Taniyama,et al. Elevated myocardial Akt signaling ameliorates doxorubicin-induced congestive heart failure and promotes heart growth. , 2002, Journal of molecular and cellular cardiology.
[3] K. Tsuchida,et al. Expression of a TGF-beta1 inducible gene, TSC-36, causes growth inhibition in human lung cancer cell lines. , 2000, Cancer letters.
[4] Orton,et al. INTRAVENOUS NESIRITIDE, A NATRIURETIC PEPTIDE, IN THE TREATMENT OF DECOMPENSATED CONGESTIVE HEART FAILURE , 2000 .
[5] R. Kitsis,et al. The MEK1–ERK1/2 signaling pathway promotes compensated cardiac hypertrophy in transgenic mice , 2000, The EMBO journal.
[6] Y. Fujio,et al. Bone Morphogenetic Protein-2 Inhibits Serum Deprivation-induced Apoptosis of Neonatal Cardiac Myocytes through Activation of the Smad1 Pathway* , 2001, The Journal of Biological Chemistry.
[7] N. Minamino,et al. Cloning and sequence analysis of cDNA encoding a precursor for human brain natriuretic peptide. , 1988, Biochemical and biophysical research communications.
[8] S. Engelhardt,et al. A Secretion Trap Screen in Yeast Identifies Protease Inhibitor 16 as a Novel Antihypertrophic Protein Secreted From the Heart , 2007, Circulation.
[9] S. Houser,et al. Nuclear Targeting of Akt Enhances Ventricular Function and Myocyte Contractility , 2005, Circulation research.
[10] S. Cook,et al. Myostatin Regulates Cardiomyocyte Growth Through Modulation of Akt Signaling , 2006, Circulation research.
[11] W. Van Hul,et al. Extracellular regulation of BMP signaling in vertebrates: a cocktail of modulators. , 2002, Developmental biology.
[12] Masahiro Ito,et al. Akt3 overexpression in the heart results in progression from adaptive to maladaptive hypertrophy. , 2005, Journal of molecular and cellular cardiology.
[13] J. Ingwall,et al. Paracrine action accounts for marked protection of ischemic heart by Akt-modified mesenchymal stem cells , 2005, Nature Medicine.
[14] T. Flynn,et al. The amino acid sequence of an atrial peptide with potent diuretic and natriuretic properties. , 1983, Biochemical and biophysical research communications.
[15] R. Kloner,et al. Alterations in myostatin expression are associated with changes in cardiac left ventricular mass but not ejection fraction in the mouse. , 2007, The Journal of endocrinology.
[16] Robert H Christenson,et al. National Academy of Clinical Biochemistry Laboratory Medicine practice guidelines: Clinical utilization of cardiac biomarker testing in heart failure. , 2007, Circulation.
[17] Y. Fujio,et al. Isolation and characterization of the murine cardiotrophin-1 gene: expression and norepinephrine-induced transcriptional activation. , 2000, Journal of molecular and cellular cardiology.
[18] H. Drexler,et al. The Transforming Growth Factor-&bgr; Superfamily Member Growth-Differentiation Factor-15 Protects the Heart From Ischemia/Reperfusion Injury , 2006 .
[19] I. Shiojima,et al. Microarray analysis of Akt1 activation in transgenic mouse hearts reveals transcript expression profiles associated with compensatory hypertrophy and failure. , 2006, Physiological genomics.
[20] R. Califf,et al. Prognostic Value of Growth-Differentiation Factor-15 in Patients With Non–ST-Elevation Acute Coronary Syndrome , 2007, Circulation.
[21] R. Kitsis,et al. Akt promotes survival of cardiomyocytes in vitro and protects against ischemia-reperfusion injury in mouse heart. , 2000, Circulation.
[22] P. Ponikowski,et al. Prognostic utility of growth differentiation factor-15 in patients with chronic heart failure. , 2007, Journal of the American College of Cardiology.
[23] S. Cook,et al. Phenotypic Spectrum Caused by Transgenic Overexpression of Activated Akt in the Heart* , 2002, The Journal of Biological Chemistry.
[24] R. Kitsis,et al. Intracoronary, adenovirus-mediated Akt gene transfer in heart limits infarct size following ischemia-reperfusion injury in vivo. , 2000, Journal of molecular and cellular cardiology.
[25] K. Nose,et al. Decrease in the expression of a novel TGF beta1-inducible and ras-recision gene, TSC-36, in human cancer cells. , 1997, Cancer letters.
[26] T. Abraham,et al. Myostatin does not regulate cardiac hypertrophy or fibrosis , 2007, Neuromuscular Disorders.
[27] I. Shiojima,et al. Disruption of coordinated cardiac hypertrophy and angiogenesis contributes to the transition to heart failure. , 2005, The Journal of clinical investigation.
[28] K. Patel,et al. Developmental expression of myostatin in cardiomyocytes and its effect on foetal and neonatal rat cardiomyocyte proliferation. , 2007, Cardiovascular research.
[29] J. Ross,et al. Akt induces enhanced myocardial contractility and cell size in vivo in transgenic mice , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[30] I. Shiojima,et al. Vascular Endothelial Growth Factor Blockade Promotes the Transition From Compensatory Cardiac Hypertrophy to Failure in Response to Pressure Overload , 2006, Hypertension.
[31] T. Hewett,et al. GDF15/MIC-1 Functions As a Protective and Antihypertrophic Factor Released From the Myocardium in Association With SMAD Protein Activation , 2006, Circulation research.
[32] Ronald J Moore,et al. Human plasma N-glycoproteome analysis by immunoaffinity subtraction, hydrazide chemistry, and mass spectrometry. , 2005, Journal of proteome research.
[33] T. Yue,et al. Inhibition of extracellular signal-regulated kinase enhances Ischemia/Reoxygenation-induced apoptosis in cultured cardiac myocytes and exaggerates reperfusion injury in isolated perfused heart. , 2000, Circulation research.
[34] D. Das,et al. Retracted: Cardioprotective effect of resveratrol via HO-1 expression involves p38 map kinase and PI-3-kinase signaling, but does not involve NFκB , 2006 .
[35] A. D. de Bold,et al. A rapid and potent natriuretic response to intravenous injection of atrial myocardial extract in rats. Reprinted from Life Sci. 28:89-94, 1981. , 2001, Journal of the American Society of Nephrology : JASN.
[36] S. Kihara,et al. Adiponectin accumulates in myocardial tissue that has been damaged by ischemia-reperfusion injury via leakage from the vascular compartment. , 2007, Cardiovascular research.
[37] H. Drexler,et al. The transforming growth factor-beta superfamily member growth-differentiation factor-15 protects the heart from ischemia/reperfusion injury. , 2006, Circulation research.
[38] K. Kangawa,et al. Adrenomedullin: a possible autocrine or paracrine inhibitor of hypertrophy of cardiomyocytes. , 1998, Hypertension.
[39] Y. Ahn,et al. Nuclear Targeting of Akt Enhances Kinase Activity and Survival of Cardiomyocytes , 2003, Circulation research.
[40] G. Semenza,et al. Complete loss of ischaemic preconditioning-induced cardioprotection in mice with partial deficiency of HIF-1 alpha. , 2008, Cardiovascular research.
[41] I. Shiojima,et al. Regulation of cardiac growth and coronary angiogenesis by the Akt/PKB signaling pathway. , 2006, Genes & development.
[42] K. Tsuchida,et al. Expression of a TGF-β1 inducible gene, TSC-36, causes growth inhibition in human lung cancer cell lines , 2000 .
[43] N. Minamino,et al. Cloning and sequence analysis of cDNA encoding a precursor for porcine brain natriuretic peptide. , 1988, Biochemical and biophysical research communications.
[44] J. Vass,et al. Regulation of a multigenic invasion programme by the transcription factor, AP-1: re-expression of a down-regulated gene, TSC-36, inhibits invasion , 2000, Oncogene.
[45] A. D. de Bold,et al. A rapid and potent natriuretic response to intravenous injection of atrial myocardial extract in rats. , 1981, Life sciences.