Role of nitric oxide, tetrahydrobiopterin and peroxynitrite in glucose toxicity-associated contractile dysfunction in ventricular myocytes

[1]  Xueliang Du,et al.  Role of oxygen derived radicals for vascular dysfunction in the diabetic heart: Prevention by α-tocopherol? , 1998, Molecular and Cellular Biochemistry.

[2]  A. Koller,et al.  Lack of Nitric Oxide Mediation of Flow-Dependent Arteriolar Dilation in Type I Diabetes Is Restored by Sepiapterin , 2003, Journal of Vascular Research.

[3]  S. Stolc,et al.  In vivo treatment with stobadine prevents lipid peroxidation, protein glycation and calcium overload but does not ameliorate Ca2+ -ATPase activity in heart and liver of streptozotocin-diabetic rats: comparison with vitamin E. , 2002, Biochimica et biophysica acta.

[4]  L. Wold,et al.  IGF-I attenuates diabetes-induced cardiac contractile dysfunction in ventricular myocytes. , 2002, American journal of physiology. Endocrinology and metabolism.

[5]  S. Stolc,et al.  In vivo treatment with stobadine prevents lipid peroxidation, protein glycation and calcium overload but does not ameliorate Ca 2+ -ATPase activity in heart and liver of streptozotocin-diabetic rats: comparison with vitamin E The ADIC (Antioxidants in Diabetes-Induced Complications) Study Group , 2002 .

[6]  Jun Ren,et al.  Ceramide attenuates high glucose-induced cardiac contractile abnormalities in cultured adult rat ventricular myocytes. , 2002, Cellular and molecular biology.

[7]  K. J. Lee,et al.  Influence of ovariectomy on ventricular myocyte contraction in simulated diabetes. , 2001, Journal of Biomedical Sciences.

[8]  A. Patruno,et al.  Endothelial nitric oxide synthase (eNOS) expression and localization in healthy and diabetic rat hearts. , 2001, Annals of clinical and laboratory science.

[9]  D. Harrison,et al.  Endothelial Regulation of Vasomotion in ApoE-Deficient Mice: Implications for Interactions Between Peroxynitrite and Tetrahydrobiopterin , 2001, Circulation.

[10]  P. Rösen,et al.  Influence of diabetes on cardiac nitric oxide synthase expression and activity. , 2000, Biochimica et biophysica acta.

[11]  L. Wold,et al.  Leptin Attenuates Cardiac Contraction in Rat Ventricular Myocytes: Role of NO , 2000, Hypertension.

[12]  S. Yuan,et al.  Protein Kinase C Activation Contributes to Microvascular Barrier Dysfunction in the Heart at Early Stages of Diabetes , 2000, Circulation research.

[13]  Guoyao Wu,et al.  Impaired nitric oxide production in coronary endothelial cells of the spontaneously diabetic BB rat is due to tetrahydrobiopterin deficiency. , 2000, The Biochemical journal.

[14]  G. Kojda,et al.  Interactions between NO and reactive oxygen species: pathophysiological importance in atherosclerosis, hypertension, diabetes and heart failure. , 1999, Cardiovascular research.

[15]  L. Mazzanti,et al.  Evidence for iNOS-dependent peroxynitrite production in diabetic platelets , 1999, Diabetologia.

[16]  G. Pieper Review of alterations in endothelial nitric oxide production in diabetes: protective role of arginine on endothelial dysfunction. , 1998, Hypertension.

[17]  S. Massry,et al.  High glucose concentration causes a rise in [Ca2+]i of cardiac myocytes. , 1998, Kidney international.

[18]  G. Gintant,et al.  High extracellular glucose impairs cardiac E-C coupling in a glycosylation-dependent manner. , 1997, The American journal of physiology.

[19]  F. Romano,et al.  Inhibition of nitric oxide synthase by L-NAME improves ventricular performance in streptozotocin-diabetic rats. , 1997, Journal of molecular and cellular cardiology.

[20]  H. Trachtman,et al.  High glucose inhibits nitric oxide production in cultured rat mesangial cells. , 1997, Journal of the American Society of Nephrology : JASN.

[21]  T. Lüscher,et al.  High glucose increases nitric oxide synthase expression and superoxide anion generation in human aortic endothelial cells. , 1997, Circulation.

[22]  C. Ballard-Croft,et al.  Mechanisms underlying depressed Na+/Ca2+ exchanger activity in the diabetic heart. , 1997, Cardiovascular research.

[23]  G. Lopaschuk,et al.  Peroxynitrite impairs cardiac contractile function by decreasing cardiac efficiency. , 1997, The American journal of physiology.

[24]  A. Davidoff,et al.  Diabetes rapidly induces contractile dysfunctions in isolated ventricular myocytes. , 1997, The American journal of physiology.

[25]  A. Davidoff,et al.  Low insulin and high glucose induce abnormal relaxation in cultured adult rat ventricular myocytes. , 1997, The American journal of physiology.

[26]  G. Pieper Acute amelioration of diabetic endothelial dysfunction with a derivative of the nitric oxide synthase cofactor, tetrahydrobiopterin. , 1997, Journal of cardiovascular pharmacology.

[27]  J. Nadler,et al.  Evidence that nitric oxide increases glucose transport in skeletal muscle. , 1997, Journal of applied physiology.

[28]  J. Sowers,et al.  Troglitazone Attenuates High-Glucose–Induced Abnormalities in Relaxation and Intracellular Calcium in Rat Ventricular Myocytes , 1996, Diabetes.

[29]  M. Suematsu,et al.  Modulation of mitochondrion-mediated oxidative stress by nitric oxide in human placental trophoblastic cells. , 1996, The American journal of physiology.

[30]  W. Graier,et al.  High D-Glucose–Induced Changes in Endothelial Ca2+/EDRF Signaling are Due to Generation of Superoxide Anions , 1996, Diabetes.

[31]  P. Rösen,et al.  Impairment of endothelium dependent relaxation in the diabetic rat heart: mechanisms and implications. , 1996, Diabetes research and clinical practice.

[32]  D. Lagadic-Gossmann,et al.  Altered Ca2+ handling in ventricular myocytes isolated from diabetic rats. , 1996, The American journal of physiology.

[33]  M. Brownlee,et al.  BCL-2 expression or antioxidants prevent hyperglycemia-induced formation of intracellular advanced glycation endproducts in bovine endothelial cells. , 1996, The Journal of clinical investigation.

[34]  B. Freeman,et al.  Nitric oxide regulation of superoxide-dependent lung injury: oxidant-protective actions of endogenously produced and exogenously administered nitric oxide. , 1996, Free radical biology & medicine.

[35]  J. Sowers,et al.  Diabetes mellitus and associated hypertension, vascular disease, and nephropathy. An update. , 1995, Hypertension.

[36]  P. Hofmann,et al.  Effects of diabetes on isometric tension as a function of [Ca2+] and pH in rat skinned cardiac myocytes. , 1995, The American journal of physiology.

[37]  M. Latronico,et al.  Administration of a nitric oxide synthase inhibitor does not suppress low-dose streptozotocin-induced diabetes in mice , 1995, International journal of pancreatology : official journal of the International Association of Pancreatology.

[38]  M. Barbagallo,et al.  Glucose-induced alterations of cytosolic free calcium in cultured rat tail artery vascular smooth muscle cells. , 1995, The Journal of clinical investigation.

[39]  S. Moncada,et al.  The role of nitric oxide in cardiac depression induced by interleukin‐1β and tumour necrosis factor‐α , 1995 .

[40]  M. Brownlee,et al.  Nonenzymatic glycosylation in vitro and in bovine endothelial cells alters basic fibroblast growth factor activity. A model for intracellular glycosylation in diabetes. , 1994, The Journal of clinical investigation.

[41]  Y. Zou,et al.  Enhanced cellular oxidant stress by the interaction of advanced glycation end products with their receptors/binding proteins. , 1994, The Journal of biological chemistry.

[42]  D. Levy,et al.  Echocardiographic evidence for the existence of a distinct diabetic cardiomyopathy (the Framingham Heart Study). , 1991, The American journal of cardiology.

[43]  J. Lancaster,et al.  Synthesis of nitric oxide from a terminal guanidino nitrogen atom of L-arginine: A molecular mechanism regulating cellular proliferation that targets intracellular iron , 1990 .

[44]  H. Kolb,et al.  Suppression of low-dose streptozotocin-induced diabetes by immunomodulatory lectins. , 1986, Diabetes research.