Glycine plays a crucial role as a co-agonist of NMDA receptors in the neuronal circuit generating body movements in rat fetuses

[1]  Natascha Schaefer,et al.  Glycine receptor mouse mutants: model systems for human hyperekplexia , 2013, British journal of pharmacology.

[2]  Y. Ben-Ari,et al.  Timing of developmental sequences in different brain structures: physiological and pathological implications , 2012, The European journal of neuroscience.

[3]  F. Andermann,et al.  Pathophysiological Mechanisms of Dominant and Recessive GLRA1 Mutations in Hyperekplexia , 2010, The Journal of Neuroscience.

[4]  H. Nishimaru,et al.  The role of inhibitory neurotransmission in locomotor circuits of the developing mammalian spinal cord , 2009, Acta physiologica.

[5]  P. Branchereau,et al.  NKCC1 cotransporter inactivation underlies embryonic development of chloride‐mediated inhibition in mouse spinal motoneuron , 2008, The Journal of physiology.

[6]  K. Sillar,et al.  The contribution of the NMDA receptor glycine site to rhythm generation during fictive swimming in Xenopus laevis tadpoles , 2007, The European journal of neuroscience.

[7]  K. Obata,et al.  Respiratory activity in brainstem of fetal mice lacking glutamate decarboxylase 65/67 and vesicular GABA transporter , 2007, Neuroscience.

[8]  Kazuo Umezawa,et al.  Adrenaline contributes to prenatal respiratory maturation in rat medulla–spinal cord preparation , 2006, Brain Research.

[9]  Fred H. Gage,et al.  Cholinergic Input Is Required during Embryonic Development to Mediate Proper Assembly of Spinal Locomotor Circuits , 2005, Neuron.

[10]  R. Lim,et al.  Blockade of glycine transporter-1 (GLYT-1) potentiates NMDA receptor-mediated synaptic transmission in hypoglossal motorneurons. , 2004, Journal of neurophysiology.

[11]  M. Hanson,et al.  Normal Patterns of Spontaneous Activity Are Required for Correct Motor Axon Guidance and the Expression of Specific Guidance Molecules , 2004, Neuron.

[12]  D. Richter,et al.  Inactivation of the Glycine Transporter 1 Gene Discloses Vital Role of Glial Glycine Uptake in Glycinergic Inhibition , 2003, Neuron.

[13]  J. Greer,et al.  Ontogeny of rhythmic motor patterns generated in the embryonic rat spinal cord. , 2003, Journal of neurophysiology.

[14]  N. Kudo,et al.  Basis of Changes in Left–Right Coordination of Rhythmic Motor Activity during Development in the Rat Spinal Cord , 2002, The Journal of Neuroscience.

[15]  P. Branchereau,et al.  Descending 5-Hydroxytryptamine Raphe Inputs Repress the Expression of Serotonergic Neurons and Slow the Maturation of Inhibitory Systems in Mouse Embryonic Spinal Cord , 2002, The Journal of Neuroscience.

[16]  I. Homma,et al.  Development of the rat respiratory neuron network during the late fetal period , 2002, Neuroscience Research.

[17]  N. Kudo,et al.  Developmental changes in 5-hydroxytryptamine-induced rhythmic activity in the spinal cord of rat fetuses in vitro , 2001, Neuroscience Letters.

[18]  F. Clarac,et al.  Perinatal development of lumbar motoneurons and their inputs in the rat , 2000, Brain Research Bulletin.

[19]  J. Cazalets,et al.  Regulation by glycine, Mg2+ and polyamines of the N-methyl-d-aspartate-induced locomotion in the neonatal rat spinal cord in vitro , 1999, Neuroscience.

[20]  N. Kudo,et al.  Rostrocaudal progression in the development of periodic spontaneous activity in fetal rat spinal motor circuits in vitro. , 1999, Journal of neurophysiology.

[21]  K. Ballanyi,et al.  Synaptic inhibition in the isolated respiratory network of neonatal rats , 1998, The European journal of neuroscience.

[22]  Ikuo Homma,et al.  The adrenergic modulation of firings of respiratory rhythm-generating neurons in medulla-spinal cord preparation from newborn rat , 1998, Experimental Brain Research.

[23]  J. Feldman,et al.  Respiratory rhythm generation and synaptic inhibition of expiratory neurons in pre-Bötzinger complex: differential roles of glycinergic and GABAergic neural transmission. , 1997, Journal of neurophysiology.

[24]  N. Kudo,et al.  Development of locomotor activity induced by NMDA receptor activation in the lumbar spinal cord of the rat fetus studied in vitro. , 1996, Brain research. Developmental brain research.

[25]  N. Kudo,et al.  Spontaneous motoneuronal activity mediated by glycine and GABA in the spinal cord of rat fetuses in vitro. , 1996, The Journal of physiology.

[26]  T. Suzue.  Mouse fetuses in late gestation maintained in vitro by a transplacental perfusion method and their physiological activities , 1994, Neuroscience Research.

[27]  G. Giacoia,et al.  Hyperekplexia associated with apnea and sudden infant death syndrome. , 1994, Archives of pediatrics & adolescent medicine.

[28]  N. Kudo,et al.  Development of descending fibers to the rat embryonic spinal cord , 1993, Neuroscience Research.

[29]  J. C. Smith,et al.  Respiratory and locomotor patterns generated in the fetal rat brain stem-spinal cord in vitro. , 1992, Journal of neurophysiology.

[30]  N. Kudo,et al.  N-Methyl-d,l-aspartate-induced locomotor activity in a spinal cord-indlimb muscles preparation of the newborn rat studied in vitro , 1987, Neuroscience Letters.

[31]  P. Ascher,et al.  Glycine potentiates the NMDA response in cultured mouse brain neurons , 1987, Nature.

[32]  T. Suzue,et al.  Respiratory rhythm generation in the in vitro brain stem‐spinal cord preparation of the neonatal rat. , 1984, The Journal of physiology.

[33]  M G Rosen,et al.  Classification of human fetal movement. , 1976, American Journal of Obstetrics and Gynecology.

[34]  R. Thomas,et al.  Genotype-phenotype correlations in hyperekplexia: apnoeas, learning difficulties and speech delay. , 2013, Brain : a journal of neurology.

[35]  G. Hilaire,et al.  In vitro study of central respiratory-like activity of the fetal rat , 2004, Experimental Brain Research.

[36]  V. Hamburger,et al.  Prenatal development of spontaneous and evoked activity in the rat (Rattus norvegicus albinus). , 1971, Behaviour.