Serum Levels of IL-17 and IL-22 Are Reduced by Etanercept, but not by Acitretin, in Patients with Psoriasis: a Randomized-Controlled Trial

[1]  Lisa C. Zaba,et al.  Psoriasis vulgaris lesions contain discrete populations of Th1 and Th17 T cells. , 2008, The Journal of investigative dermatology.

[2]  K. Jażdżewski,et al.  Interleukin (IL)-23 receptor is a major susceptibility gene for Graves' ophthalmopathy: the IL-23/T-helper 17 axis extends to thyroid autoimmunity. , 2008, The Journal of clinical endocrinology and metabolism.

[3]  Lisa C. Zaba,et al.  Amelioration of epidermal hyperplasia by TNF inhibition is associated with reduced Th17 responses , 2007, The Journal of experimental medicine.

[4]  F. Morel,et al.  A role for T cell‐derived interleukin 22 in psoriatic skin inflammation , 2007, Clinical and experimental immunology.

[5]  L. Cosmi,et al.  Phenotypic and functional features of human Th17 cells , 2007, The Journal of experimental medicine.

[6]  Jia-wen Li,et al.  Expression of Th17 cytokines in skin lesions of patients with psoriasis , 2007, Journal of Huazhong University of Science and Technology. Medical sciences = Hua zhong ke ji da xue xue bao. Yi xue Ying De wen ban = Huazhong keji daxue xuebao. Yixue Yingdewen ban.

[7]  A. Gottlieb,et al.  Mechanisms of action of etanercept in psoriasis. , 2007, The journal of investigative dermatology. Symposium proceedings.

[8]  B. Chong,et al.  Immunobiologics in the treatment of psoriasis. , 2007, Clinical immunology.

[9]  J. Koo,et al.  An attempt to formulate an evidence-based strategy in the management of moderate-to-severe psoriasis: a review of the efficacy and safety of biologics and prebiologic options , 2007, Expert opinion on pharmacotherapy.

[10]  R. D. Hatton,et al.  IL-17 family cytokines and the expanding diversity of effector T cell lineages. , 2007, Annual review of immunology.

[11]  James G. Krueger,et al.  Pathogenesis and therapy of psoriasis , 2007, Nature.

[12]  P. Valdez,et al.  Interleukin-22, a TH17 cytokine, mediates IL-23-induced dermal inflammation and acanthosis , 2007, Nature.

[13]  O. Boyman,et al.  The pathogenic role of tissue-resident immune cells in psoriasis. , 2007, Trends in immunology.

[14]  K. Kikly,et al.  The IL-23/Th(17) axis: therapeutic targets for autoimmune inflammation. , 2006, Current opinion in immunology.

[15]  P. Kerkhof.  Update on retinoid therapy of psoriasis in: an update on the use of retinoids in dermatology. , 2006 .

[16]  S. Nakae,et al.  IL-17 Plays an Important Role in the Development of Experimental Autoimmune Encephalomyelitis1 , 2006, The Journal of Immunology.

[17]  K. Asadullah,et al.  IL‐22 regulates the expression of genes responsible for antimicrobial defense, cellular differentiation, and mobility in keratinocytes: a potential role in psoriasis , 2006, European journal of immunology.

[18]  J. Bos,et al.  In Vitro and In Situ Expression of IL-23 by Keratinocytes in Healthy Skin and Psoriasis Lesions: Enhanced Expression in Psoriatic Skin , 2006, The Journal of Immunology.

[19]  O. Arican,et al.  Serum Levels of TNF-α, IFN-γ, IL-6, IL-8, IL-12, IL-17, and IL-18 in Patients With Active Psoriasis and Correlation With Disease Severity , 2005, Mediators of inflammation.

[20]  A. Gurney,et al.  IL-22 Inhibits Epidermal Differentiation and Induces Proinflammatory Gene Expression and Migration of Human Keratinocytes1 , 2005, The Journal of Immunology.

[21]  M. Goldman,et al.  IL‐23 up‐regulates IL‐10 and induces IL‐17 synthesis by polyclonally activated naive T cells in human , 2005, European journal of immunology.

[22]  R. Wu,et al.  IL-17 Markedly Up-Regulates β-Defensin-2 Expression in Human Airway Epithelium via JAK and NF-κB Signaling Pathways1 , 2004, The Journal of Immunology.

[23]  K. Asadullah,et al.  IL-22 increases the innate immunity of tissues. , 2004, Immunity.

[24]  A. Bowcock,et al.  Psoriasis vulgaris: cutaneous lymphoid tissue supports T-cell activation and "Type 1" inflammatory gene expression. , 2004, Trends in immunology.

[25]  M. Dhodapkar,et al.  Increased Expression of Interleukin 23 p19 and p40 in Lesional Skin of Patients with Psoriasis Vulgaris , 2004, The Journal of experimental medicine.

[26]  J. Voorhees,et al.  Molecular mechanisms of retinoid actions in skin , 1996, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[27]  W. Jochum,et al.  T cells involved in psoriasis vulgaris belong to the Th1 subset. , 1994, The Journal of investigative dermatology.

[28]  Jiawen Li,et al.  The expression of interleukin-17, interferon-gamma, and macrophage inflammatory protein-3 alpha mRNA in patients with psoriasis vulgaris. , 2004, Journal of Huazhong University of Science and Technology. Medical sciences = Hua zhong ke ji da xue xue bao. Yi xue Ying De wen ban = Huazhong keji daxue xuebao. Yixue Yingdewen ban.