Possible role of fructosamine 3-kinase genotyping for the management of diabetic patients
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M. Ferrari | A. Mosca | R. Paleari | A. Lapolla | N. Chilelli | A. Ambrosi | F. Avemaria | P. Carrera | G. Sartore
[1] Michael Q. Zhang,et al. Integrative analysis of 111 reference human epigenomes , 2015, Nature.
[2] Y. J. Kim,et al. Multiple Nonglycemic Genomic Loci Are Newly Associated With Blood Level of Glycated Hemoglobin in East Asians , 2014, Diabetes.
[3] Nirmal Singh,et al. Advanced Glycation End Products and Diabetic Complications , 2014, The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology.
[4] Leif Groop,et al. Genetics of diabetes – Are we missing the genes or the disease? , 2014, Molecular and Cellular Endocrinology.
[5] A. Lapolla,et al. AGEs, rather than hyperglycemia, are responsible for microvascular complications in diabetes: a "glycoxidation-centric" point of view. , 2013, Nutrition, metabolism, and cardiovascular diseases : NMCD.
[6] E. Schaftingen,et al. Enzymatic repair of Amadori products , 2012, Amino Acids.
[7] M. Ezzati,et al. National, regional, and global trends in fasting plasma glucose and diabetes prevalence since 1980: systematic analysis of health examination surveys and epidemiological studies with 370 country-years and 2·7 million participants , 2011, The Lancet.
[8] Å. Lernmark,et al. Position Statement Executive Summary: Guidelines and Recommendations for Laboratory Analysis in the Diagnosis and Management of Diabetes Mellitus , 2011, Diabetes Care.
[9] Paul J Thornalley,et al. Glyoxalase in diabetes, obesity and related disorders. , 2011, Seminars in cell & developmental biology.
[10] Teri A Manolio,et al. Genomewide association studies and assessment of the risk of disease. , 2010, The New England journal of medicine.
[11] E. Schaftingen,et al. Effects of fructosamine-3-kinase deficiency on function and survival of mouse pancreatic islets after prolonged culture in high glucose or ribose concentrations. , 2010, American journal of physiology. Endocrinology and metabolism.
[12] B. Melegh,et al. A Polymorphism within the Fructosamine-3-kinase Gene is Associated with HbA1c Levels and the Onset of Type 2 Diabetes Mellitus , 2009, Experimental and clinical endocrinology & diabetes : official journal, German Society of Endocrinology [and] German Diabetes Association.
[13] R. Cohen,et al. Biologic Variability in Plasma Glucose, Hemoglobin A1c, and Advanced Glycation End Products Associated with Diabetes Complications , 2009, Journal of diabetes science and technology.
[14] J. Shaw,et al. International Expert Committee Report on the Role of the A1C Assay in the Diagnosis of Diabetes , 2009, Diabetes Care.
[15] Eric P. Smith,et al. Frequency of HbA1c discordance in estimating blood glucose control , 2008, Current opinion in clinical nutrition and metabolic care.
[16] D. Vertommen,et al. Increased protein glycation in fructosamine 3-kinase-deficient mice. , 2006, The Biochemical journal.
[17] D. Vertommen,et al. Variability in erythrocyte fructosamine 3-kinase activity in humans correlates with polymorphisms in the FN3K gene and impacts on haemoglobin glycation at specific sites. , 2006, Diabetes & metabolism.
[18] F. Opperdoes,et al. Tissue Distribution and Evolution of Fructosamine 3-Kinase and Fructosamine 3-Kinase-related Protein* , 2004, Journal of Biological Chemistry.
[19] B. Szwergold,et al. The expression of the genes for fructosamine-3-kinase and fructosamine-3-kinase-related protein appears to be constitutive and unaffected by environmental signals. , 2004, Biochemical and biophysical research communications.
[20] D. Vertommen,et al. Identification of Fructosamine Residues Deglycated by Fructosamine-3-kinase in Human Hemoglobin* , 2004, Journal of Biological Chemistry.
[21] F. Collard,et al. A mammalian protein homologous to fructosamine-3-kinase is a ketosamine-3-kinase acting on psicosamines and ribulosamines but not on fructosamines. , 2003, Diabetes.
[22] V. Monnier,et al. Enzymatic deglycation with amadoriase enzymes from Aspergillus sp. as a potential strategy against the complications of diabetes and aging. , 2003, Biochemical Society transactions.
[23] G. King,et al. Molecular understanding of hyperglycemia's adverse effects for diabetic complications. , 2002, JAMA.
[24] F. Collard,et al. Identification of a Pathway for the Utilization of the Amadori Product Fructoselysine in Escherichia coli * , 2002, The Journal of Biological Chemistry.
[25] F. Collard,et al. Fructosamine 3-kinase is involved in an intracellular deglycation pathway in human erythrocytes. , 2002, The Biochemical journal.
[26] R. Dean,et al. Inactivation of cellular enzymes by carbonyls and protein-bound glycation/glycoxidation products. , 2002, Archives of biochemistry and biophysics.
[27] M. Brownlee. Biochemistry and molecular cell biology of diabetic complications , 2001, Nature.
[28] B. Szwergold,et al. Human fructosamine-3-kinase: purification, sequencing, substrate specificity, and evidence of activity in vivo. , 2001, Diabetes.
[29] L. Bry,et al. Effects of hemoglobin variants and chemically modified derivatives on assays for glycohemoglobin. , 2001, Clinical chemistry.
[30] E. Schaftingen,et al. Identification, cloning, and heterologous expression of a mammalian fructosamine-3-kinase. , 2000, Diabetes.
[31] R. Holman,et al. Association of glycaemia with macrovascular and microvascular complications of type 2 diabetes (UKPDS 35): prospective observational study , 2000, BMJ : British Medical Journal.
[32] P. Zimmet,et al. Definition, diagnosis and classification of diabetes mellitus and its complications. Part 1: diagnosis and classification of diabetes mellitus. Provisional report of a WHO Consultation , 1998, Diabetic medicine : a journal of the British Diabetic Association.
[33] T. Brown,et al. Fructose metabolism in the human erythrocyte. Phosphorylation to fructose 3-phosphate. , 1992, The Biochemical journal.
[34] T. Brown,et al. Identification of sorbitol 3-phosphate and fructose 3-phosphate in normal and diabetic human erythrocytes. , 1990, The Journal of biological chemistry.
[35] E. Beutler,et al. Mannose metabolism in the human erythrocyte. , 1969, The Journal of clinical investigation.
[36] V. Bartáková,et al. Genetic variability in enzymes of metabolic pathways conferring protection against non-enzymatic glycation versus diabetes-related morbidity and mortality , 2014, Clinical chemistry and laboratory medicine.
[37] T. Zima,et al. Fructosamine 3-kinase and glyoxalase I polymorphisms and their association with soluble RAGE and adhesion molecules in diabetes. , 2014, Physiological research.
[38] A. Mosca,et al. Genetic variability of the fructosamine 3-kinase gene in diabetic patients , 2011, Clinical chemistry and laboratory medicine.
[39] Juliane,et al. Edinburgh Research Explorer Common variants at 10 genomic loci influence hemoglobin A(C) levels via glycemic and nonglycemic pathways , 2010 .
[40] P. Zimmet,et al. Diagnosis and classification of diabetes mellitus , 2002 .
[41] A. Cerami,et al. Protein glycation, diabetes, and aging. , 2001, Recent progress in hormone research.
[42] G. P. Ellis,et al. The Maillard reaction. , 1959, Advances in carbohydrate chemistry.