FADD expression and caspase activation in B‐cell lymphomas resistant to Fas‐mediated apoptosis
暂无分享,去创建一个
E. Devilard | R. Bouabdallah | F. Birg | L. Xerri | A. Stoppa | J. Hassoun
[1] P. Guldberg,et al. Somatic Fas mutations in non-Hodgkin's lymphoma: association with extranodal disease and autoimmunity. , 1998, Blood.
[2] M. Hutchinson,et al. Processing/activation of caspases, -3 and -7 and -8 but not caspase-2, in the induction of apoptosis in B-chronic lymphocytic leukemia cells , 1998, Leukemia.
[3] E. Devilard,et al. Sensitivity to Fas-mediated apoptosis is null or weak in B-cell non-Hodgkin's lymphomas and is moderately increased by CD40 ligation. , 1998, British Journal of Cancer.
[4] Marc C. Jacob,et al. Tumor B cells from non-Hodgkin's lymphoma are resistant to CD95 (Fas/Apo-1)-mediated apoptosis. , 1998, Blood.
[5] M. Peter,et al. Bcl-xL Acts Downstream of Caspase-8 Activation by the CD95 Death-inducing Signaling Complex* , 1998, The Journal of Biological Chemistry.
[6] S. Nagata,et al. Cleavage of CAD inhibitor in CAD activation and DNA degradation during apoptosis , 1998, Nature.
[7] E. Devilard,et al. Malignant and reactive cells from human lymphomas frequently express Fas ligand but display a different sensitivity to Fas-mediated apoptosis , 1997, Leukemia.
[8] M. Peter,et al. FLICE Is Predominantly Expressed as Two Functionally Active Isoforms, Caspase-8/a and Caspase-8/b* , 1997, The Journal of Biological Chemistry.
[9] Y. Tsujimoto,et al. Evidence against a functional site for Bcl-2 downstream of caspase cascade in preventing apoptosis , 1997, Oncogene.
[10] E. Devilard,et al. Cysteine protease CPP32, but not Ich1-L, is expressed in germinal center B cells and their neoplastic counterparts. , 1997, Human pathology.
[11] N. Thornberry,et al. Caspases: killer proteases. , 1997, Trends in biochemical sciences.
[12] Margot Thome,et al. Inhibition of death receptor signals by cellular FLIP , 1997, Nature.
[13] Matthias Mann,et al. FLICE is activated by association with the CD95 death‐inducing signaling complex (DISC) , 1997, The EMBO journal.
[14] R. Gascoyne,et al. Immunolocalization of the ICE/Ced-3–Family Protease, CPP32 (Caspase-3), in Non-Hodgkin's Lymphomas, Chronic Lymphocytic Leukemias, and Reactive Lymph Nodes , 1997 .
[15] D. Colomer,et al. Involvement of CED-3/ICE proteases in the apoptosis of B-chronic lymphocytic leukemia cells. , 1997, Blood.
[16] M. Peter,et al. Resistance of cultured peripheral T cells towards activation‐induced cell death involves a lack of recruitment of FLICE (MACH/caspase 8) to the CD95 death‐inducing signaling complex , 1997, European journal of immunology.
[17] G. Freeman,et al. Role of the CD40 and CD95 (APO‐1/Fas) antigens in the apoptosis of human B‐cell malignancies , 1997, British journal of haematology.
[18] W. Dalton,et al. Selection for drug resistance results in resistance to Fas-mediated apoptosis. , 1997, Blood.
[19] J. Hoflack,et al. Characterization of anti-peptide antibodies directed towards the automodification domain and apoptotic fragment of poly (ADP-ribose) polymerase. , 1997, Biochimica et biophysica acta.
[20] Z. Ao,et al. A mitochondrial membrane protein defined by a novel monoclonal antibody is preferentially detected in apoptotic cells. , 1996, Journal of immunology.
[21] David Wallach,et al. Involvement of MACH, a Novel MORT1/FADD-Interacting Protease, in Fas/APO-1- and TNF Receptor–Induced Cell Death , 1996, Cell.
[22] Matthias Mann,et al. FLICE, A Novel FADD-Homologous ICE/CED-3–like Protease, Is Recruited to the CD95 (Fas/APO-1) Death-Inducing Signaling Complex , 1996, Cell.
[23] I. Herr,et al. Involvement of the CD95 (APO–1/Fas) receptor/ligand system in drug–induced apoptosis in leukemia cells , 1996, Nature Medicine.
[24] N. Thornberry,et al. Apopain/CPP32 cleaves proteins that are essential for cellular repair: a fundamental principle of apoptotic death , 1996, The Journal of experimental medicine.
[25] S. Nagata,et al. Sequential activation of ICE-like and CPP32-like proteases during Fas-mediated apoptosis , 1996, Nature.
[26] A. Chinnaiyan,et al. FADD/MORT1 Is a Common Mediator of CD95 (Fas/APO-1) and Tumor Necrosis Factor Receptor-induced Apoptosis (*) , 1996, The Journal of Biological Chemistry.
[27] D. Olive,et al. Comparison of CD28‐B7.1 and B7.2 functional interaction in resting human T cells: Phosphatidylinositol 3‐kinase association to CD28 and cytokine production , 1996, European journal of immunology.
[28] C. Schneider,et al. Microfilament reorganization during apoptosis: the role of Gas2, a possible substrate for ICE‐like proteases. , 1995, The EMBO journal.
[29] F. Birg,et al. Search for rearrangements and/or allelic loss of the fas/APO-1 gene in 101 human lymphomas. , 1995, American journal of clinical pathology.
[30] F. Birg,et al. Frequent expression of FAS/APO‐1 in Hodgkin's disease and anaplastic large cell lymphomas FAS/APO‐1 in Hodgkin's disease and anaplastic large cell lymphomas , 1995, Histopathology.
[31] Patrick R. Griffin,et al. Identification and inhibition of the ICE/CED-3 protease necessary for mammalian apoptosis , 1995, Nature.
[32] Muneesh Tewari,et al. Yama/CPP32β, a mammalian homolog of CED-3, is a CrmA-inhibitable protease that cleaves the death substrate poly(ADP-ribose) polymerase , 1995, Cell.
[33] Arul M. Chinnaiyan,et al. FADD, a novel death domain-containing protein, interacts with the death domain of fas and initiates apoptosis , 1995, Cell.
[34] W. Fiers,et al. Requirement of an ICE/CED-3 protease for Fas/APO-1-mediated apoptosis , 1995, Nature.
[35] E. Alnemri,et al. CPP32, a novel human apoptotic protein with homology to Caenorhabditis elegans cell death protein Ced-3 and mammalian interleukin-1 beta-converting enzyme. , 1994, The Journal of biological chemistry.
[36] Y. Lazebnik,et al. Cleavage of poly(ADP-ribose) polymerase by a proteinase with properties like ICE , 1994, Nature.
[37] H Stein,et al. A revised European-American classification of lymphoid neoplasms: a proposal from the International Lymphoma Study Group. , 1994, Blood.
[38] R. Armitage. Tumor necrosis factor receptor superfamily members and their ligands. , 1994, Current opinion in immunology.
[39] D. Olive,et al. Binding of phosphatidyl-inositol-3-OH kinase to CD28 is required for T-cell signalling , 1994, Nature.
[40] P. Möller,et al. Coregulation of the APO-1 antigen with intercellular adhesion molecule-1 (CD54) in tonsillar B cells and coordinate expression in follicular center B cells and in follicle center and mediastinal B-cell lymphomas. , 1993, Blood.
[41] M. Pawlita,et al. Purification and molecular cloning of the APO-1 cell surface antigen, a member of the tumor necrosis factor/nerve growth factor receptor superfamily. Sequence identity with the Fas antigen. , 1992, The Journal of biological chemistry.
[42] Atsushi Hase,et al. The polypeptide encoded by the cDNA for human cell surface antigen Fas can mediate apoptosis , 1991, Cell.
[43] J. Sklar,et al. Cloning and structural analysis of cDNAs for bcl-2 and a hybrid bcl-2/immunoglobulin transcript resulting from the t(14;18) translocation , 1986, Cell.
[44] S. Korsmeyer,et al. Cloning the chromosomal breakpoint of t(14;18) human lymphomas: clustering around Jh on chromosome 14 and near a transcriptional unit on 18 , 1985, Cell.