The Role of NO Synthase, MPT pore, and Protein Kinase A in the Cardioprotective Effect of the Opioid Receptor Agonist Deltorphin II

[1]  S. Popov,et al.  δ2-Opioid Receptors as a Target in Designing New Cardioprotective Drugs: the Role of Protein Kinase C, AMPK, and Sarcolemmal KATP Channels , 2022, Bulletin of Experimental Biology and Medicine.

[2]  P. Oeltgen,et al.  Activation of peripheral δ2-opioid receptor prevents reperfusion heart injury. , 2021, European journal of pharmacology.

[3]  S. Sapozhnikov,et al.  Influence of Total Ischemic Time on Clinical Outcomes in Patients with ST-Segment Elevation Myocardial Infarction. , 2021, Kardiologiia.

[4]  P. Oeltgen,et al.  Pharmacology of mitochondrial permeability transition pore inhibitors , 2019, Drug development research.

[5]  A. Heinen,et al.  Morphine-Induced Preconditioning: Involvement of Protein Kinase A and Mitochondrial Permeability Transition Pore , 2016, PloS one.

[6]  G. Heusch.  Molecular basis of cardioprotection: signal transduction in ischemic pre-, post-, and remote conditioning. , 2015, Circulation research.

[7]  A. Hsu,et al.  Acute and Chronic Cardioprotection by the Enkephalin Analogue, Eribis Peptide 94, is Mediated via Activation of Nitric Oxide Synthase and Adenosine Triphosphate-Regulated Potassium Channels , 2012, Pharmacology.

[8]  P. Oeltgen,et al.  Activation of peripheral delta2 opioid receptors increases cardiac tolerance to ischemia/reperfusion injury Involvement of protein kinase C, NO-synthase, KATP channels and the autonomic nervous system. , 2009, Life sciences.

[9]  A. Hsu,et al.  Ischemic preconditioning and morphine-induced cardioprotection involve the delta (delta)-opioid receptor in the intact rat heart. , 1997, Journal of molecular and cellular cardiology.

[10]  D. Hausenloy,et al.  The mitochondrial permeability transition pore and its role in myocardial ischemia reperfusion injury. , 2015, Journal of molecular and cellular cardiology.

[11]  J. Herget,et al.  Cardioprotective effect of chronic hypoxia is blunted by concomitant hypercapnia. , 2003, Physiological research.