Assessment of "microvascular no-reflow phenomenon" using technetium-99m macroaggregated albumin scintigraphy in patients with acute myocardial infarction.
暂无分享,去创建一个
Y. Shimono | M. Kondo | A. Nakano | D. Saito
[1] Y. Taniyama,et al. Myocardial perfusion patterns related to thrombolysis in myocardial infarction perfusion grades after coronary angioplasty in patients with acute anterior wall myocardial infarction. , 1996, Circulation.
[2] D. Ramage,et al. Leukocyte adhesion to the coronary microvasculature during ischemia and reperfusion in an in vivo canine model. , 1996, Circulation.
[3] M. Hori,et al. Clinical implications of the 'no reflow' phenomenon. A predictor of complications and left ventricular remodeling in reperfused anterior wall myocardial infarction. , 1996, Circulation.
[4] E. Zajac,et al. Ability of the no-reflow phenomenon during an acute myocardial infarction to predict left ventricular dysfunction at one-month follow-up. , 1995, The American journal of cardiology.
[5] R. Jeremy,et al. Relation between ischemia time, infarct size, and left ventricular function in humans. , 1995, Circulation.
[6] I. Morishima,et al. Clinical significance of no-reflow phenomenon observed on angiography after successful treatment of acute myocardial infarction with percutaneous transluminal coronary angioplasty. , 1995, American heart journal.
[7] M. Hori,et al. Temporal changes in myocardial perfusion patterns in patients with reperfused anterior wall myocardial infarction. Their relation to myocardial viability. , 1995, Circulation.
[8] M. Hori,et al. Progressive decreases in coronary vein flow during reperfusion in acute myocardial infarction: clinical documentation of the no reflow phenomenon after successful thrombolysis. , 1994, Journal of the American College of Cardiology.
[9] J. Copeland,et al. Microvascular compression during myocardial ischemia: mechanistic basis for no-reflow phenomenon. , 1994, The American journal of physiology.
[10] S. Glantz. It is all in the numbers. , 1993, Journal of the American College of Cardiology.
[11] R. Kloner. Does reperfusion injury exist in humans? , 1993, Journal of the American College of Cardiology.
[12] Y. Shimono,et al. Is ST segment re-elevation associated with reperfusion an indicator of marked myocardial damage after thrombolysis? , 1993, Journal of the American College of Cardiology.
[13] K. Messmer,et al. Microvascular ischemia-reperfusion injury in striated muscle: significance of "reflow paradox". , 1992, The American journal of physiology.
[14] K. Messmer,et al. Microvascular ischemia-reperfusion injury in striated muscle: significance of "no reflow". , 1992, The American journal of physiology.
[15] A. Kitabatake,et al. Lack of Myocardial Perfusion Immediately After Successful Thrombolysis: A Predictor of Poor Recovery of Left Ventricular Function in Anterior Myocardial Infarction , 1992, Circulation.
[16] L. Becker,et al. Progressive impairment of regional myocardial perfusion after initial restoration of postischemic blood flow. , 1989, Circulation.
[17] J. Covell,et al. Granulocytes cause reperfusion ventricular dysfunction after 15-minute ischemia in the dog. , 1987, Circulation research.
[18] P. Serruys,et al. Which patients benefit most from early thrombolytic therapy with intracoronary streptokinase? , 1986, Circulation.
[19] G. Schmid-Schönbein,et al. Accumulation of polymorphonuclear leukocytes during 3-h experimental myocardial ischemia. , 1986, The American journal of physiology.
[20] A prospective trial of intravenous streptokinase in acute myocardial infarction (I.S.A.M.). Mortality, morbidity, and infarct size at 21 days. , 1986, The New England journal of medicine.
[21] W. Ganz,et al. The thrombolysis in myocardial infarction (TIMI) trial. , 1985, The New England journal of medicine.
[22] J. Schofer,et al. Scintigraphic evidence of the "no reflow" phenomenon in human beings after coronary thrombolysis. , 1985, Journal of the American College of Cardiology.
[23] G. Schmid-Schönbein,et al. Leukocyte capillary plugging in myocardial ischemia and reperfusion in the dog. , 1983, The American journal of pathology.
[24] E. Braunwald,et al. Ultrastructural Evidence of Microvascular Damage and Myocardial Cell Injury After Coronary Artery Occlusion: Which Comes First? , 1980, Circulation.
[25] S. Glantz,et al. Quantitative Left Ventricular Wall Motion Analysis: A Comparison of Area, Chord Radial Methods , 1979, Circulation.
[26] M. P. Judkins,et al. Particulate myocardial perfusion scintigraphy: its clinical usefulness in evaluation of coronary artery disease. , 1977, Seminars in nuclear medicine.
[27] J. Ritchie,et al. Myocardial Imaging with Radionuclide-Labeled Particles , 1976 .
[28] J. Murray,et al. Myocardial perfusion imaging with 99mTc or 113mIn macroaggregated albumin: Correlation of the perfusion image with clinical, angiographic, surgical, and histologic findings , 1975 .
[29] J. Murray,et al. Myocardial perfusion imaging with 99m-Tc or 113m-In macroaggregated albumin: correlation of the perfusion image with clinical, angiographic, surgical, and histologic findings. , 1975, American Heart Journal.
[30] R. Kloner,et al. The "no-reflow" phenomenon after temporary coronary occlusion in the dog. , 1974, The Journal of clinical investigation.
[31] A. L. Simon,et al. Myocardial Perfusion Imaging with Radioactive‐Labeled Particles Injected Directly into the Coronary Circulation of Patients with Coronary Artery Disease , 1971, Circulation.
[32] S. Konno,et al. The direct diagnosis of human myocardial ischemia using 131I-MAA via the selective coronary catheter. Preliminary report. , 1970, American heart journal.
[33] S. Rosalki. An improved procedure for serum creatine phosphokinase determination. , 1967, The Journal of laboratory and clinical medicine.