Assessment of "microvascular no-reflow phenomenon" using technetium-99m macroaggregated albumin scintigraphy in patients with acute myocardial infarction.

[1]  Y. Taniyama,et al.  Myocardial perfusion patterns related to thrombolysis in myocardial infarction perfusion grades after coronary angioplasty in patients with acute anterior wall myocardial infarction. , 1996, Circulation.

[2]  D. Ramage,et al.  Leukocyte adhesion to the coronary microvasculature during ischemia and reperfusion in an in vivo canine model. , 1996, Circulation.

[3]  M. Hori,et al.  Clinical implications of the 'no reflow' phenomenon. A predictor of complications and left ventricular remodeling in reperfused anterior wall myocardial infarction. , 1996, Circulation.

[4]  E. Zajac,et al.  Ability of the no-reflow phenomenon during an acute myocardial infarction to predict left ventricular dysfunction at one-month follow-up. , 1995, The American journal of cardiology.

[5]  R. Jeremy,et al.  Relation between ischemia time, infarct size, and left ventricular function in humans. , 1995, Circulation.

[6]  I. Morishima,et al.  Clinical significance of no-reflow phenomenon observed on angiography after successful treatment of acute myocardial infarction with percutaneous transluminal coronary angioplasty. , 1995, American heart journal.

[7]  M. Hori,et al.  Temporal changes in myocardial perfusion patterns in patients with reperfused anterior wall myocardial infarction. Their relation to myocardial viability. , 1995, Circulation.

[8]  M. Hori,et al.  Progressive decreases in coronary vein flow during reperfusion in acute myocardial infarction: clinical documentation of the no reflow phenomenon after successful thrombolysis. , 1994, Journal of the American College of Cardiology.

[9]  J. Copeland,et al.  Microvascular compression during myocardial ischemia: mechanistic basis for no-reflow phenomenon. , 1994, The American journal of physiology.

[10]  S. Glantz It is all in the numbers. , 1993, Journal of the American College of Cardiology.

[11]  R. Kloner Does reperfusion injury exist in humans? , 1993, Journal of the American College of Cardiology.

[12]  Y. Shimono,et al.  Is ST segment re-elevation associated with reperfusion an indicator of marked myocardial damage after thrombolysis? , 1993, Journal of the American College of Cardiology.

[13]  K. Messmer,et al.  Microvascular ischemia-reperfusion injury in striated muscle: significance of "reflow paradox". , 1992, The American journal of physiology.

[14]  K. Messmer,et al.  Microvascular ischemia-reperfusion injury in striated muscle: significance of "no reflow". , 1992, The American journal of physiology.

[15]  A. Kitabatake,et al.  Lack of Myocardial Perfusion Immediately After Successful Thrombolysis: A Predictor of Poor Recovery of Left Ventricular Function in Anterior Myocardial Infarction , 1992, Circulation.

[16]  L. Becker,et al.  Progressive impairment of regional myocardial perfusion after initial restoration of postischemic blood flow. , 1989, Circulation.

[17]  J. Covell,et al.  Granulocytes cause reperfusion ventricular dysfunction after 15-minute ischemia in the dog. , 1987, Circulation research.

[18]  P. Serruys,et al.  Which patients benefit most from early thrombolytic therapy with intracoronary streptokinase? , 1986, Circulation.

[19]  G. Schmid-Schönbein,et al.  Accumulation of polymorphonuclear leukocytes during 3-h experimental myocardial ischemia. , 1986, The American journal of physiology.

[20]  A prospective trial of intravenous streptokinase in acute myocardial infarction (I.S.A.M.). Mortality, morbidity, and infarct size at 21 days. , 1986, The New England journal of medicine.

[21]  W. Ganz,et al.  The thrombolysis in myocardial infarction (TIMI) trial. , 1985, The New England journal of medicine.

[22]  J. Schofer,et al.  Scintigraphic evidence of the "no reflow" phenomenon in human beings after coronary thrombolysis. , 1985, Journal of the American College of Cardiology.

[23]  G. Schmid-Schönbein,et al.  Leukocyte capillary plugging in myocardial ischemia and reperfusion in the dog. , 1983, The American journal of pathology.

[24]  E. Braunwald,et al.  Ultrastructural Evidence of Microvascular Damage and Myocardial Cell Injury After Coronary Artery Occlusion: Which Comes First? , 1980, Circulation.

[25]  S. Glantz,et al.  Quantitative Left Ventricular Wall Motion Analysis: A Comparison of Area, Chord Radial Methods , 1979, Circulation.

[26]  M. P. Judkins,et al.  Particulate myocardial perfusion scintigraphy: its clinical usefulness in evaluation of coronary artery disease. , 1977, Seminars in nuclear medicine.

[27]  J. Ritchie,et al.  Myocardial Imaging with Radionuclide-Labeled Particles , 1976 .

[28]  J. Murray,et al.  Myocardial perfusion imaging with 99mTc or 113mIn macroaggregated albumin: Correlation of the perfusion image with clinical, angiographic, surgical, and histologic findings , 1975 .

[29]  J. Murray,et al.  Myocardial perfusion imaging with 99m-Tc or 113m-In macroaggregated albumin: correlation of the perfusion image with clinical, angiographic, surgical, and histologic findings. , 1975, American Heart Journal.

[30]  R. Kloner,et al.  The "no-reflow" phenomenon after temporary coronary occlusion in the dog. , 1974, The Journal of clinical investigation.

[31]  A. L. Simon,et al.  Myocardial Perfusion Imaging with Radioactive‐Labeled Particles Injected Directly into the Coronary Circulation of Patients with Coronary Artery Disease , 1971, Circulation.

[32]  S. Konno,et al.  The direct diagnosis of human myocardial ischemia using 131I-MAA via the selective coronary catheter. Preliminary report. , 1970, American heart journal.

[33]  S. Rosalki An improved procedure for serum creatine phosphokinase determination. , 1967, The Journal of laboratory and clinical medicine.