Open or closed
暂无分享,去创建一个
[1] N. Weiss,et al. PERICONCEPTIONAL MULTIVITAMIN USE IN RELATION TO THE RISK OF CONGENITAL URINARY TRACT ANOMALIES , 1995, Epidemiology.
[2] V. Vigneaud. A trail of research in sulfur chemistry and metabolism, and related fields , 1952 .
[3] Prevention of neural tube defects: Results of the Medical Research Council vitamin study , 1991 .
[4] A. Czeizel,et al. Prevention of the First Occurrence of Neural-Tube Defects by Periconceptional Vitamin Supplementation , 1992 .
[5] O. Heby. DNA methylation and polyamines in embryonic development and cancer. , 1995, The International journal of developmental biology.
[6] G. Shaw,et al. Maternal periconceptional use of multivitamins and reduced risk for conotruncal heart defects and limb deficiencies among offspring. , 1995, American journal of medical genetics.
[7] R. Clarke,et al. Hyperhomocysteinemia: an independent risk factor for vascular disease. , 1991, The New England journal of medicine.
[8] G. Boers,et al. Neural-tube defects and derangement of homocysteine metabolism. , 1991, The New England journal of medicine.
[9] H. Blom,et al. Hyperhomocysteinemia: a risk factor for placental abruption or infarction. , 1996, European journal of obstetrics, gynecology, and reproductive biology.
[10] G. Shaw,et al. Risks of orofacial clefts in children born to women using multivitamins containing folic acid periconceptionally , 1995, The Lancet.
[11] H. Blom,et al. Maternal hyperhomocysteinemia: a risk factor for neural-tube defects? , 1994, Metabolism: clinical and experimental.
[12] B. Hibbard. THE ROLE OF FOLIC ACID IN PREGNANCY * , 1964, The Journal of obstetrics and gynaecology of the British Commonwealth.
[13] J. Finkelstein,et al. Methionine metabolism in mammals. , 1990, The Journal of nutritional biochemistry.
[14] A. Czeizel. Reduction of urinary tract and cardiovascular defects by periconceptional multivitamin supplementation. , 1996, American journal of medical genetics.
[15] J. Finkelstein,et al. Homocystinuria: An Enzymatic Defect , 1964, Science.
[16] J. Selhub,et al. Homocysteine induces congenital defects of the heart and neural tube: effect of folic acid. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[17] H. McNulty,et al. Effect of increasing dietary folate on red-cell folate: implications for prevention of neural tube defects , 1996, The Lancet.
[18] H. Blom,et al. Hyperhomocysteinemia: a risk factor in women with unexplained recurrent early pregnancy loss. , 1993, Fertility and sterility.
[19] J. Os. Schizophrenia After Prenatal Famine , 1997 .
[20] A. Read,et al. POSSIBLE PREVENTION OF NEURAL-TUBE DEFECTS BY PERICONCEPTIONAL VITAMIN SUPPLEMENTATION , 1980, The Lancet.
[21] M. Norusis,et al. Thermolabile methylenetetrahydrofolate reductase: an inherited risk factor for coronary artery disease. , 1991, American journal of human genetics.
[22] A. Czeizel,et al. Can terathanasia explain the protective effect of folic-acid supplementation on birth defects? , 1997, The Lancet.
[23] W. Gehring. The homeo box: A key to the understanding of development? , 1985, Cell.
[24] H. Blom,et al. Is the common 677C-->T mutation in the methylenetetrahydrofolate reductase gene a risk factor for neural tube defects? A meta-analysis. , 1997, QJM : monthly journal of the Association of Physicians.
[25] G. Boers,et al. Heterozygosity for homocystinuria in premature peripheral and cerebral occlusive arterial disease. , 1985, The New England journal of medicine.
[26] G. Oakley. Folic acid-preventable spina bifida and anencephaly. , 1993, JAMA.
[27] A. Herbst,et al. Adenocarcinoma of the vagina. Association of maternal stilbestrol therapy with tumor appearance in young women. , 1971, The New England journal of medicine.
[28] M. Susser,et al. Famine and Human Development: The Dutch Hunger Winter of 1944-1945 , 1975 .
[29] M. den Heyer,et al. Mutated methylenetetrahydrofolate reductase as a risk factor for spina bifida , 1995, The Lancet.
[30] D C Shields,et al. A genetic defect in 5,10 methylenetetrahydrofolate reductase in neural tube defects. , 1995, QJM : monthly journal of the Association of Physicians.
[31] J. Noordhoek,et al. Prevention of neural tube defects by and toxicity of L-homocysteine in cultured postimplantation rat embryos. , 1994, Teratology.
[32] R. Hirasing,et al. Prevalentie, klinisch beeld en prognose van neuralebuisdefecten in Nederland. , 1996 .
[33] R. Steegers-Theunissen,et al. Periconceptional vitamin profiles are not suitable for identifying women at risk for neural tube defects. , 1993, The Journal of nutrition.
[34] E. Mariman,et al. Decreased methylene tetrahydrofolate reductase activity due to the 677C→T mutation in families with spina bifida offspring , 1996, Journal of Molecular Medicine.
[35] R. Smithells,et al. FOLIC ACID METABOLISM AND HUMAN EMBRYOPATHY , 1965 .
[36] A. Kuijpers-Jagtman,et al. Nonsyndromic orofacial clefts: association with maternal hyperhomocysteinemia. , 1999, Teratology.
[37] E. Susser,et al. Schizophrenia after prenatal famine. Further evidence. , 1996, Archives of general psychiatry.
[38] H. Blom,et al. Hormone replacement therapy may reduce high serum homocysteine in postmenopausal women , 1994, European journal of clinical investigation.
[39] H. Blom,et al. Genetic risk factor for unexplained recurrent early pregnancy loss , 1997, The Lancet.
[40] H. Blom,et al. Thermolabile 5,10-methylenetetrahydrofolate reductase as a cause of mild hyperhomocysteinemia. , 1995, American journal of human genetics.