Epidermal cytokines in murine cutaneous irritant responses
暂无分享,去创建一个
H I Maibach | I. Effendy | H Löffler | I Effendy | H. Löffler | H. Maibach
[1] L. Owen-Schaub,et al. Effect of IL-12 on immune suppression and suppressor cell induction by ultraviolet radiation. , 1995, Journal of immunology.
[2] J. Saurat,et al. Nickel and skin irritants up-regulate tumor necrosis factor-alpha mRNA in keratinocytes by different but potentially synergistic mechanisms. , 1995, International immunology.
[3] K. Rajewsky,et al. Interleukin 10 but not interleukin 4 is a natural suppressant of cutaneous inflammatory responses , 1995, The Journal of experimental medicine.
[4] H. Pinkus,et al. Examination of the epidermis by the strip method of removing horny layers. I. Observations on thickness of the horny layer, and on mitotic activity after stripping. , 1951, The Journal of investigative dermatology.
[5] V. Dixit,et al. Monocyte chemotaxis and activating factor production by keratinocytes in response to IFN-gamma. , 1991, Journal of immunology.
[6] S. Grabbe,et al. Immunoregulatory mechanisms involved in elicitation of allergic contact hypersensitivity. , 1996, American journal of contact dermatitis : official journal of the American Contact Dermatitis Society.
[7] I. Kimber,et al. Cytokine endpoints for the local lymph node assay: consideration of interferon‐γ and interleukin 12 , 1999, Journal of applied toxicology : JAT.
[8] T. Luger,et al. Effect of UV irradiation on epidermal cell cytokine production. , 1989, Journal of photochemistry and photobiology. B, Biology.
[9] T. Luger,et al. The effect of in vitro and in vivo UV irradiation on the production of ETAF activity by human and murine keratinocytes. , 1983, The Journal of investigative dermatology.
[10] H. Asada,et al. Cytokine gene expression during the elicitation phase of contact sensitivity: regulation by endogenous IL-4. , 1997, The Journal of investigative dermatology.
[11] J. Knop,et al. Is cytokine expression responsible for differences between allergens and irritants? , 1996, American journal of contact dermatitis : official journal of the American Contact Dermatitis Society.
[12] A. Takashima,et al. Langerhans cell responses to ultraviolet B radiation. , 1999, Journal of dermatological science.
[13] P. Elias,et al. Barrier function coordinately regulates epidermal IL‐1 and IL‐1 receptor antagonist mRNA levels , 1994, Experimental dermatology.
[14] P. Elias,et al. Cutaneous barrier perturbation stimulates cytokine production in the epidermis of mice. , 1992, The Journal of clinical investigation.
[15] A. Enk,et al. An essential role for Langerhans cell-derived IL-1 beta in the initiation of primary immune responses in skin. , 1993, Journal of immunology.
[16] J. Kanno,et al. Cytokine induction in human epidermal keratinocytes exposed to contact irritants and its relation to chemical-induced inflammation in mouse skin. , 1994, The Journal of investigative dermatology.
[17] S. Kunkel,et al. Modulation of leucocyte adhesion molecules, a T‐cell chemotaxin (IL‐8) and a regulatory cytokine (TNF‐α) in allergic contact dermatitis (rhus dermatitis) , 1991, The British journal of dermatology.
[18] A. Enk,et al. Induction of hapten-specific tolerance by interleukin 10 in vivo , 1994, The Journal of experimental medicine.
[19] A. Shaw,et al. Resistance to fever induction and impaired acute-phase response in interleukin-1 beta-deficient mice. , 1995, Immunity.
[20] T. Luger,et al. Removal of the majority of epidermal Langerhans cells by topical or systemic steroid application enhances the effector phase of murine contact hypersensitivity. , 1995, Journal of immunology.
[21] A. Enk,et al. Enhanced expression of IL‐8 in normal human keratinocytes and human keratinocyte cell line HaCaT in vitro after stimulation with contact sensitizers., tolerogens and irritants , 1994, Experimental dermatology.
[22] M. Kripke,et al. Activation of keratinocytes with psoralen plus UVA radiation induces the release of soluble factors that suppress delayed and contact hypersensitivity. , 1991, The Journal of investigative dermatology.
[23] C. Bunker,et al. Interleukin‐4 and the interleukin‐4 receptor in allergic contact dermatitis , 1998, Contact dermatitis.
[24] I. Kimber,et al. Cytokines and regulation of allergic sensitization to chemicals. , 1994, Toxicology.
[25] P. Askenase,et al. Molecular mechanisms of CD8+ T cell-mediated delayed hypersensitivity: implications for allergies, asthma, and autoimmunity. , 1999, The Journal of allergy and clinical immunology.
[26] A. Enk,et al. Early molecular events in the induction phase of contact sensitivity. , 1992, Proceedings of the National Academy of Sciences of the United States of America.
[27] A. Rao,et al. Chromatin-based regulatory mechanisms governing cytokine gene transcription. , 1999, The Journal of allergy and clinical immunology.
[28] B. Kong,et al. Surfactant-Induced Alteration of Arachidonic Acid Metabolism of Mammalian Cells in Culture , 1987, Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine.
[29] J. Krutmann,et al. Interleukin-10 production by cultured human keratinocytes: regulation by ultraviolet B and ultraviolet A1 radiation. , 1995, The Journal of investigative dermatology.
[30] K. Elgjo,et al. UVB-induced epidermal hyperproliferation is modified by a single, topical treatment with a mitosis inhibitory epidermal pentapeptide. , 1990, The Journal of investigative dermatology.
[31] T. Luger,et al. Cytokine modulation of keratinocyte cytokines. , 1990, The Journal of investigative dermatology.
[32] Ian Kimber,et al. Interleukin 6 (IL-6) Production by Lymph Node Cells: An Alternative Endpoint for the Murine Local Lymph Node Assay , 1993 .
[33] T. Griffith,et al. Regulation of contact hypersensitivity by interleukin 10 , 1994, The Journal of experimental medicine.
[34] P. Elias,et al. Occlusion lowers cytokine mRNA levels in essential fatty acid-deficient and normal mouse epidermis, but not after acute barrier disruption. , 1994, The Journal of investigative dermatology.
[35] R. Maizels,et al. Th1-Th2: reliable paradigm or dangerous dogma? , 1997, Immunology today.
[36] D. Sauder,et al. The role of keratinocyte cytokines in inflammation and immunity. , 1990, The Journal of investigative dermatology.
[37] Y. Aragane,et al. In vivo effects of interleukin-10 on contact hypersensitivity and delayed-type hypersensitivity reactions. , 1994, The Journal of investigative dermatology.
[38] E. Claassen,et al. In vivo cytokine profiles in allergic and irritant contact dermatitis , 1995, Contact dermatitis.
[39] B. Nickoloff,et al. Perturbation of epidermal barrier function correlates with initiation of cytokine cascade in human skin. , 1994, Journal of the American Academy of Dermatology.
[40] N. Yawalkar,et al. IL–1β protein in human skin lymph does not discriminate allergic from irritant contact dermatitis , 1996 .
[41] Baohui Xu,et al. RT-PCR Analysis of in Vivo Cytokine Profiles in Murine Allergic Contact Dermatitis to DNCB , 1996 .
[42] C. Mackay,et al. Chemokines and chemokine receptors in T-cell priming and Th1/Th2-mediated responses. , 1998, Immunology today.
[43] M. Middleton,et al. Ultraviolet radiation can either suppress or induce expression of intercellular adhesion molecule 1 (ICAM-1) on the surface of cultured human keratinocytes. , 1990, The Journal of investigative dermatology.
[44] M. Mohamadzadeh,et al. Ultraviolet B radiation up-regulates the expression of IL-15 in human skin. , 1995, Journal of immunology.
[45] H. Maibach,et al. Recent investigations of mechanisms of chemically induced skin irritation in laboratory mice. , 1987, The Journal of investigative dermatology.
[46] K. Katsuoka,et al. The expression of Th1 and Th2 type cytokines in a lesion of allergic contact dermatitis , 1996, Contact dermatitis.
[47] V. Dixit,et al. Keratinocytes as initiators of inflammation , 1991, The Lancet.
[48] T. Luger,et al. Regulation of epidermal cell interleukin-6 production by UV light and corticosteroids. , 1991, The Journal of investigative dermatology.
[49] P. Cruz,et al. Langerhans Cells Are the Major Source of mRNA for IL-1β and MIP-1α Among Unstimulated Mouse Epidermal Cells , 1992 .
[50] R. Coffman,et al. TH1 and TH2 cells: different patterns of lymphokine secretion lead to different functional properties. , 1989, Annual review of immunology.
[51] C. Willis,et al. Differential effects of structurally unrelated chemical irritants on the density of proliferating keratinocytes in 48 h patch test reactions. , 1992, The Journal of investigative dermatology.
[52] D. Griswold,et al. Intralesional cytokines in chronic oxazolone-induced contact sensitivity suggest roles for tumor necrosis factor alpha and interleukin-4. , 1998, The Journal of investigative dermatology.
[53] H. Renz,et al. Analysis of the phenotype and cytokine pattern of blood- and skin-derived nickel specific T cells in allergic contact dermatitis. , 1997, International archives of allergy and immunology.
[54] P. Bergstresser,et al. Impact of UVB Radiation on the Epidermal Cytokine Network , 1996, Photochemistry and photobiology.
[55] G. Trinchieri,et al. Neutralization of IL-12 in vivo prevents induction of contact hypersensitivity and induces hapten-specific tolerance. , 1996, Journal of immunology.
[56] J. Haas,et al. Induction of inflammatory cytokines in murine keratinocytes upon in vivo stimulation with contact sensitizers and tolerizing analogues , 1992, Experimental dermatology.
[57] J. Lee,et al. Activation of the IL-1 gene in UV-irradiated mouse skin: association with inflammatory sequelae and pharmacologic intervention. , 1991, The Journal of investigative dermatology.
[58] T. Kupper,et al. Hydrocortisone reduces both constitutive and UV-elicited release of epidermal thymocyte activating factor (ETAF) by cultured keratinocytes. , 1986, The Journal of investigative dermatology.
[59] S. Kondo,et al. Interleukin-10 inhibits the elicitation phase of allergic contact hypersensitivity. , 1994, The Journal of investigative dermatology.