Lack of association association between the presence of dupA and babA2 genes in Helicobacter pylori and gastroduodenal disorders

[1]  R. Mansour,et al.  High frequency of Helicobacter pylori DNA in drinking water in Kermanshah, Iran, during June-November 2012. , 2014, Journal of water and health.

[2]  M. Pereira,et al.  Role of Helicobacter pylori in gastric mucosa-associated lymphoid tissue lymphomas. , 2014, World journal of gastroenterology.

[3]  Y. Yamaoka,et al.  Diversity of Helicobacter pylori genotypes in Iranian patients with different gastroduodenal disorders. , 2013, World journal of gastroenterology.

[4]  A. Talebi Bezmin Abadi,et al.  High correlation of babA2-positive strains of Helicobacter pylori with the presence of gastric cancer , 2013, Internal and Emergency Medicine.

[5]  H. Abdollahi,et al.  THE PREVALENCE OF HELICOBACTER PYLORI BABA2, ICEA1 AND ICEA2 GENES AND THEIR ASSOCIATION WITH CLINICAL OUTCOMES IN PATIENTS WITH CHRONIC GASTRITIS, ULCERATIVE DISEASES AND NON-ULCER DYSPEPSIA IN SOUTH EAST OF IRAN , 2013 .

[6]  Carina M. Schlebusch,et al.  Age of the Association between Helicobacter pylori and Man , 2012, PLoS pathogens.

[7]  F. Poursina,et al.  Helicobacter pylori in Iran: A systematic review on the association of genotypes and gastroduodenal diseases , 2012, Journal of research in medical sciences : the official journal of Isfahan University of Medical Sciences.

[8]  Y. Yamaoka.  Mechanisms of disease: Helicobacter pylori virulence factors , 2010, Nature Reviews Gastroenterology &Hepatology.

[9]  Mario Plebani,et al.  Pathogenesis of Helicobacter pylori Infection , 2010, Helicobacter.

[10]  Yi-Chun Yeh,et al.  Matrix metalloproteinase-3 promoter polymorphisms but not dupA-H. pylori correlate to duodenal ulcers in H. pylori-infected females , 2010, BMC Microbiology.

[11]  K. Murakami,et al.  Helicobacter pylori dupA gene is not associated with clinical outcomes in the Japanese population. , 2010, Clinical microbiology and infection : the official publication of the European Society of Clinical Microbiology and Infectious Diseases.

[12]  F. Navabakbar,et al.  Relation of bab A2 genotype of Helicobacter pylori infection with chronic active gastritis, duodenal ulcer and non-cardia active gastritis in Alzahra hospital Isfahan, Iran , 2010 .

[13]  R. Peek,et al.  Role of innate immunity in Helicobacter pylori-induced gastric malignancy. , 2010, Physiological reviews.

[14]  S. Suerbaum,et al.  Helicobacter pylori infection , 2013, Nature Reviews Disease Primers.

[15]  S. Moss,et al.  Helicobacter pylori virulence factors in gastric carcinogenesis. , 2009, Cancer letters.

[16]  J. L. Cortés,et al.  Annals of Clinical Microbiology and Antimicrobials Frequency of Vaca, Caga and Baba2 Virulence Markers in Helicobacter Pylori Strains Isolated from Mexican Patients with Chronic Gastritis , 2022 .

[17]  D. Forman,et al.  The prevalence of the duodenal ulcer promoting gene (dupA) in Helicobacter pylori isolates varies by ethnic group and is not universally associated with disease development: a case-control study , 2009, Gut pathogens.

[18]  Hong Lu,et al.  The Helicobacter pylori duodenal ulcer promoting gene, dupA in China , 2008, BMC gastroenterology.

[19]  J. Gould.  Prevalence of Helicobacter pylori Infection Among Patients Undergoing Bariatric Surgery: A Preliminary Study , 2008, World Journal of Surgery.

[20]  M. Mohagheghi,et al.  dupA as a risk determinant in Helicobacter pylori infection. , 2008, Journal of medical microbiology.

[21]  Y. Yamaoka.  Roles of the plasticity regions of Helicobacter pylori in gastroduodenal pathogenesis. , 2008, Journal of medical microbiology.

[22]  C. A. Oliveira,et al.  Lack of association between Helicobacter pylori infection with dupA-positive strains and gastroduodenal diseases in Brazilian patients. , 2008, International journal of medical microbiology : IJMM.

[23]  B. Lal,et al.  Prevalence of Duodenal Ulcer‐Promoting Gene (dupA) of Helicobacter pylori in Patients with Duodenal Ulcer in North Indian Population , 2007, Helicobacter.

[24]  V. Y. Miendje Deyi,et al.  The presence of dupA in Helicobacter pylori is not significantly associated with duodenal ulceration in Belgium, South Africa, China, or North America. , 2007, Clinical infectious diseases : an official publication of the Infectious Diseases Society of America.

[25]  R. B. de Oliveira,et al.  Prevalence of Helicobacter pylori cagA, iceA and babA2 alleles in Brazilian patients with upper gastrointestinal diseases. , 2006, Acta tropica.

[26]  Y. Erzin,et al.  Prevalence of Helicobacter pylori vacA, cagA, cagE, iceA, babA2 Genotypes and Correlation with Clinical Outcome in Turkish Patients with Dyspepsia , 2006, Helicobacter.

[27]  T. Cover,et al.  Multiple Chromosomal Loci for the babA Gene in Helicobacter pylori , 2006, Infection and Immunity.

[28]  R. Mattar,et al.  No Correlation of babA2 with vacA and cagA Genotypes of Helicobacter pylori and Grading of Gastritis from Peptic Ulcer Disease Patients in Brazil , 2005, Helicobacter.

[29]  C. Prinz,et al.  Correlation of the Helicobacter pylori adherence factor BabA with duodenal ulcer disease in four European countries. , 2005, FEMS immunology and medical microbiology.

[30]  D. Graham,et al.  Duodenal ulcer promoting gene of Helicobacter pylori. , 2005, Gastroenterology.

[31]  Wen Zhong Liu,et al.  Clinical relevance of iceA and babA2 genotypes of Helicobacter pylori in a Shanghai population. , 2004, Chinese journal of digestive diseases.

[32]  R. Malekzadeh,et al.  Prevalence of gastric precancerous lesions in Ardabil, a high incidence province for gastric adenocarcinoma in the northwest of Iran , 2003, Journal of clinical pathology.

[33]  P. Hohenberger,et al.  Helicobacter pylori virulence factors—one part of a big picture , 2003, The Lancet.

[34]  C. A. Oliveira,et al.  babA2- and cagA-Positive Helicobacter pylori Strains Are Associated with Duodenal Ulcer and Gastric Carcinoma in Brazil , 2003, Journal of Clinical Microbiology.

[35]  B. Sheu,et al.  Host gastric Lewis expression determines the bacterial density of Helicobacter pylori in babA2 genopositive infection , 2003, Gut.

[36]  M. Rugge,et al.  Helicobacter pylori babA2, cagA, and s1 vacA genes work synergistically in causing intestinal metaplasia , 2003, Journal of clinical pathology.

[37]  Wen-Ching Wang,et al.  High Prevalence of cagA- and babA2-Positive Helicobacter pylori Clinical Isolates in Taiwan , 2002, Journal of Clinical Microbiology.

[38]  C. Prinz,et al.  Pathogenesis of Helicobacter pylori infection , 2002, Helicobacter.

[39]  C. Prinz,et al.  The Helicobacter pylori Blood Group Antigen-Binding Adhesin Facilitates Bacterial Colonization and Augments a Nonspecific Immune Response1 , 2002, The Journal of Immunology.

[40]  M. Blaser,et al.  Theoretical and experimental approaches for studying factors defining the Helicobacter pylori-host relationship. , 2000, Trends in microbiology.

[41]  M. Carrington,et al.  Interleukin-1 polymorphisms associated with increased risk of gastric cancer , 2000, Nature.

[42]  C. Prinz,et al.  Clinical relevance of the Helicobacter pylori gene for blood-group antigen-binding adhesin. , 1999, Proceedings of the National Academy of Sciences of the United States of America.

[43]  M. Blaser,et al.  Dynamics of Helicobacter pylori colonization in relation to the host response. , 1999, Proceedings of the National Academy of Sciences of the United States of America.

[44]  F. Galeotti,et al.  Analysis of Helicobacter pylori vacA andcagA genotypes and serum antibody profile in benign and malignant gastroduodenal diseases , 1998, Gut.

[45]  J. Parsonnet.  Helicobacter pylori: the size of the problem , 1998, Gut.

[46]  L. Engstrand,et al.  Helicobacter pylori adhesin binding fucosylated histo-blood group antigens revealed by retagging. , 1998, Science.

[47]  J. G. Kusters,et al.  Infection with Helicobacter pylori strains lacking dupA is associated with an increased risk of gastric ulcer and gastric cancer development. , 2012, Journal of medical microbiology.

[48]  C. Chomvarin,et al.  Prevalence of Helicobacter pylori vacA, cagA, cagE, iceA and babA2 genotypes in Thai dyspeptic patients. , 2008, International journal of infectious diseases : IJID : official publication of the International Society for Infectious Diseases.

[49]  D. Graham,et al.  Helicobacter pylori BabA expression, gastric mucosal injury, and clinical outcome. , 2007, Clinical gastroenterology and hepatology : the official clinical practice journal of the American Gastroenterological Association.

[50]  R. Stein.  Duodenal Ulcer Promoting Gene of Helicobacter pylori , 2006 .

[51]  M. Blaser,et al.  Helicobacter pylori and gastrointestinal tract adenocarcinomas , 2002, Nature Reviews Cancer.