Effect of glatiramer acetate on conversion to clinically definite multiple sclerosis in patients with clinically isolated syndrome (PreCISe study): a randomised, double-blind, placebo-controlled trial
暂无分享,去创建一个
F Fazekas | M Filippi | G Comi | M. Filippi | X. Montalban | F. Fazekas | V. Martinelli | G. Comi | I. Elovaara | P. Rieckmann | L. Moiola | C. Young | K. Myhr | S. Komoly | J. Hillert | K. Myhr | A. Carrá | C. Lubetzki | J. King | H. Hartung | O. Bajenaru | X Montalban | V Martinelli | M Rodegher | L Moiola | O Bajenaru | A Carra | I Elovaara | HP Hartung | J Hillert | J King | S Komoly | C Lubetzki | KM Myhr | M Ravnborg | P Rieckmann | D Wynn | C Young | C. Young | M. Ravnborg | D. Wynn | M. Rodegher | J. King
[1] Hans Lassmann,et al. Cortical demyelination and diffuse white matter injury in multiple sclerosis. , 2005, Brain : a journal of neurology.
[2] Marco Rovaris,et al. Effect of early interferon treatment on conversion to definite multiple sclerosis: a randomised study , 2001, The Lancet.
[3] Michael Wall,et al. High- and low-risk profiles for the development of multiple sclerosis within 10 years after optic neuritis: experience of the optic neuritis treatment trial. , 2003, Archives of ophthalmology.
[4] H. Lassmann,et al. The neuroprotective effect of inflammation: implications for the therapy of multiple sclerosis , 2000, Journal of Neuroimmunology.
[5] Massimo Filippi,et al. Clinically isolated syndromes suggestive of multiple sclerosis, part 2: non-conventional MRI, recovery processes, and management , 2005, The Lancet Neurology.
[6] F. Barkhof,et al. Evaluation of patients treated with natalizumab for progressive multifocal leukoencephalopathy. , 2006, The New England journal of medicine.
[7] J. De Keyser,et al. Treatment with interferon beta-1b delays conversion to clinically definite and McDonald MS in patients with clinically isolated syndromes , 2006, Neurology.
[8] R. Rudick,et al. Axonal transection in the lesions of multiple sclerosis. , 1998, The New England journal of medicine.
[9] Massimo Filippi,et al. European/Canadian multicenter, double‐blind, randomized, placebo‐controlled study of the effects of glatiramer acetate on magnetic resonance imaging–measured disease activity and burden in patients with relapsing multiple sclerosis , 2001, Annals of neurology.
[10] J H Simon,et al. Intramuscular interferon beta-1a therapy initiated during a first demyelinating event in multiple sclerosis. CHAMPS Study Group. , 2000, The New England journal of medicine.
[11] A. Thompson,et al. Magnetic resonance imaging in monitoring the treatment of multiple sclerosis: concerted action guidelines. , 1991, Journal of neurology, neurosurgery, and psychiatry.
[12] A. Thompson,et al. Clinically isolated syndromes suggestive of multiple sclerosis, part I: natural history, pathogenesis, diagnosis, and prognosis , 2005, The Lancet Neurology.
[13] P M Matthews,et al. Evidence of axonal damage in the early stages of multiple sclerosis and its relevance to disability. , 2001, Archives of neurology.
[14] K. Selmaj,et al. Alemtuzumab vs. interferon beta-1a in early multiple sclerosis. , 2008, The New England journal of medicine.
[15] S. Cole,et al. Interferon β‐1a for early multiple sclerosis: CHAMPS trial subgroup analyses , 2002 .
[16] M. Esiri,et al. The contribution of demyelination to axonal loss in multiple sclerosis. , 2006, Brain : a journal of neurology.
[17] D. Paty,et al. Interferon beta-1b in secondary progressive MS , 2004, Neurology.
[18] D. Bates. Future therapies in multiple sclerosis , 2008 .
[19] J S Wolinsky,et al. Extended use of glatiramer acetate (Copaxone) is well tolerated and maintains its clinical effect on multiple sclerosis relapse rate and degree of disability , 1998, Neurology.
[20] B. Thrower. Clinically isolated syndromes , 2007, Neurology.
[21] P. Narayana,et al. Glatiramer acetate in primary progressive multiple sclerosis: Results of a multinational, multicenter, double‐blind, placebo‐controlled trial , 2007, Annals of neurology.
[22] M Rovaris,et al. Improving interobserver variation in reporting gadolinium-enhanced MRI lesions in multiple sclerosis , 1997, Neurology.
[23] M. Boggild,et al. Therapy-related acute leukaemia with Mitoxantrone: what is the risk and can we minimise it? , 2009, Multiple sclerosis.
[24] F. Barkhof,et al. Comparison of subcutaneous interferon beta-1a with glatiramer acetate in patients with relapsing multiple sclerosis (the REbif vs Glatiramer Acetate in Relapsing MS Disease [REGARD] study): a multicentre, randomised, parallel, open-label trial , 2008, The Lancet Neurology.
[25] S. Wassertheil-Smoller,et al. A pilot trial of Cop 1 in exacerbating-remitting multiple sclerosis. , 1987, The New England journal of medicine.
[26] M. Filippi,et al. Simple and complex movement‐associated functional MRI changes in patients at presentation with clinically isolated syndromes suggestive of multiple sclerosis , 2004, Human brain mapping.
[27] M. Tintoré. Rationale for early intervention with immunomodulatory treatments , 2008, Journal of Neurology.
[28] M Rovaris,et al. Measurement error of two different techniques for brain atrophy assessment in multiple sclerosis , 2004, Neurology.
[29] C. Pozzilli,et al. The Relationship between inflammation and atrophy in clinically isolated syndromes suggestive of multiple sclerosis , 2004, Journal of Neurology.
[30] D. Silberberg,et al. New diagnostic criteria for multiple sclerosis: Guidelines for research protocols , 1983, Annals of neurology.
[31] G. Comi. Clinically isolated syndrome: the rationale for early treatment , 2008, Nature Clinical Practice Neurology.
[32] Wolfgang Brück,et al. Acute axonal damage in multiple sclerosis is most extensive in early disease stages and decreases over time. , 2002, Brain : a journal of neurology.
[33] S. Reingold,et al. Diagnostic criteria for multiple sclerosis: 2005 revisions to the “McDonald Criteria” , 2005, Annals of neurology.
[34] J Grimaud,et al. Effect of training and different measurement strategies on the reproducibility of brain MRI lesion load measurements in multiple sclerosis , 1998, Neurology.
[35] A. Thompson,et al. Disability and T2 MRI lesions: a 20-year follow-up of patients with relapse onset of multiple sclerosis. , 2008, Brain : a journal of neurology.