Antiapoptotic effects of nicotine in its protection against salsolinol-induced cytotoxicity

[1]  K. Manaye,et al.  Nicotine blocks ethanol-induced apoptosis in primary cultures of rat cerebral cortical and cerebellar granule cells , 2009, Neurotoxicity Research.

[2]  Yasmine M. Kanaan,et al.  Neuroprotective effects of nicotine against salsolinol-induced cytotoxicity: Implications for parkinson’s disease , 2005, Neurotoxicity Research.

[3]  M. Toborek,et al.  ERK 1/2 signaling pathway is involved in nicotine‐mediated neuroprotection in spinal cord neurons , 2007, Journal of cellular biochemistry.

[4]  J. Padmanabhan,et al.  Rb function in the apoptosis and senescence of non-neuronal and neuronal cells: role in oncogenesis. , 2006, Current molecular medicine.

[5]  Y. Ahn,et al.  Membrane depolarization stimulates the proliferation of SH-SY5Y human neuroblastoma cells by increasing retinoblastoma protein (RB) phosphorylation through the activation of cyclin-dependent kinase 2 (Cdk2) , 2006, Neuroscience Letters.

[6]  Brij B. Singh,et al.  TRPC1 protects human SH-SY5Y cells against salsolinol-induced cytotoxicity by inhibiting apoptosis , 2006, Brain Research.

[7]  Y. Akao,et al.  Type A monoamine oxidase is the target of an endogenous dopaminergic neurotoxin, N‐methyl(R)salsolinol, leading to apoptosis in SH‐SY5Y cells , 2006, Journal of neurochemistry.

[8]  J. Pinski,et al.  Discovery and preclinical evaluation of a novel class of small-molecule compounds in hormone-dependent and -independent cancer cell lines , 2005, Molecular Cancer Therapeutics.

[9]  M. Naoi,et al.  Neuroprotective function of R-(-)-1-(benzofuran-2-yl)-2-propylaminopentane, [R-(-)-BPAP], against apoptosis induced by N-methyl(R)salsolinol, an endogenous dopaminergic neurotoxin, in human dopaminergic neuroblastoma SH-SY5Y cells. , 2004, Life sciences.

[10]  L. Forno,et al.  Loss of α‐conotoxinMII‐ and A85380‐sensitive nicotinic receptors in Parkinson's disease striatum , 2004 .

[11]  M. Naoi,et al.  Dopamine-derived salsolinol derivatives as endogenous monoamine oxidase inhibitors: occurrence, metabolism and function in human brains. , 2004, Neurotoxicology.

[12]  Jean Y. J. Wang,et al.  Coordinated regulation of life and death by RB , 2003, Nature Reviews Cancer.

[13]  Johannes Schwarz,et al.  Selective dopaminergic neurotoxicity of isoquinoline derivatives related to Parkinson's disease: studies using heterologous expression systems of the dopamine transporter. , 2002, Biochemical pharmacology.

[14]  M. Naoi,et al.  Future of neuroprotection in Parkinson's disease. , 2001, Parkinsonism & related disorders.

[15]  F. Zunino,et al.  Cell cycle checkpoint efficiency and cellular response to paclitaxel in prostate cancer cells , 2001, The Prostate.

[16]  N. Tatton.  Increased Caspase 3 and Bax Immunoreactivity Accompany Nuclear GAPDH Translocation and Neuronal Apoptosis in Parkinson's Disease , 2000, Experimental Neurology.

[17]  R. Weinberg,et al.  The retinoblastoma protein and cell cycle control , 1995, Cell.

[18]  L. Benítez-Bribiesca.  [Apoptosis in the pathogenesis and treatment of disease]. , 1995, Gaceta medica de Mexico.

[19]  R. Lukas,et al.  Characterization of Nicotinic Acetylcholine Receptors Expressed by Cells of the SH-SY5Y Human Neuroblastoma Clonal Line , 1993, Molecular and Cellular Neuroscience.