Oxidative Stress and Obesity: Their Impact on Metabolic Syndrome
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[1] M. Matsuda,et al. Human catalase gene is regulated by peroxisome proliferator activated receptor-gamma through a response element distinct from that of mouse. , 2010, Endocrine journal.
[2] S. Grundy,et al. Harmonizing the metabolic syndrome: a joint interim statement of the International Diabetes Federation Task Force on Epidemiology and Prevention; National Heart, Lung, and Blood Institute; American Heart Association; World Heart Federation; International Atherosclerosis Society; and International As , 2009, Circulation.
[3] M. Matsuda,et al. Dysregulated glutathione metabolism links to impaired insulin action in adipocytes. , 2009, American journal of physiology. Endocrinology and metabolism.
[4] T. Fujita,et al. Sympathoexcitation by Oxidative Stress in the Brain Mediates Arterial Pressure Elevation in Obesity-Induced Hypertension , 2009, Circulation.
[5] M. Matsuda,et al. Adipose expression of catalase is regulated via a novel remote PPARgamma-responsive region. , 2008, Biochemical and Biophysical Research Communications - BBRC.
[6] S. Kihara,et al. Blockade of Angiotensin II type-1 receptor reduces oxidative stress in adipose tissue and ameliorates adipocytokine dysregulation. , 2006, Kidney international.
[7] T. Funahashi,et al. Systemic oxidative stress is associated with visceral fat accumulation and the metabolic syndrome. , 2006, Circulation journal : official journal of the Japanese Circulation Society.
[8] E. Lander,et al. Reactive oxygen species have a causal role in multiple forms of insulin resistance , 2006, Nature.
[9] Paul Zimmet,et al. The metabolic syndrome—a new worldwide definition , 2005, The Lancet.
[10] Michael Brownlee,et al. The pathobiology of diabetic complications: a unifying mechanism. , 2005, Diabetes.
[11] Jun Ren,et al. High dietary fat induces NADPH oxidase-associated oxidative stress and inflammation in rat cerebral cortex , 2005, Experimental Neurology.
[12] Morihiro Matsuda,et al. Increased oxidative stress in obesity and its impact on metabolic syndrome. , 2004, The Journal of clinical investigation.
[13] A. Quyyumi,et al. Renin-Angiotensin System and Angiotensin Receptor Blockers in the Metabolic Syndrome , 2004, Circulation.
[14] M. Desai,et al. Obesity is associated with macrophage accumulation in adipose tissue. , 2003, The Journal of clinical investigation.
[15] L. Tartaglia,et al. Chronic inflammation in fat plays a crucial role in the development of obesity-related insulin resistance. , 2003, The Journal of clinical investigation.
[16] D. Loskutoff,et al. Monocyte chemoattractant protein 1 in obesity and insulin resistance , 2003, Proceedings of the National Academy of Sciences of the United States of America.
[17] E. Benjamin,et al. Obesity and Systemic Oxidative Stress: Clinical Correlates of Oxidative Stress in The Framingham Study , 2003, Arteriosclerosis, thrombosis, and vascular biology.
[18] E. Blaak,et al. Possible involvement of the adipose tissue renin‐angiotensin system in the pathophysiology of obesity and obesity‐related disorders , 2003, Obesity reviews : an official journal of the International Association for the Study of Obesity.
[19] S. Kihara,et al. Adiponectin Reduces Atherosclerosis in Apolipoprotein E-Deficient Mice , 2002, Circulation.
[20] M. Matsuda,et al. Role of Adiponectin in Preventing Vascular Stenosis , 2002, The Journal of Biological Chemistry.
[21] M. Matsuda,et al. Diet-induced insulin resistance in mice lacking adiponectin/ACRP30 , 2002, Nature Medicine.
[22] P. Scherer,et al. The adipocyte-secreted protein Acrp30 enhances hepatic insulin action , 2001, Nature Medicine.
[23] Y. Terauchi,et al. The fat-derived hormone adiponectin reverses insulin resistance associated with both lipoatrophy and obesity , 2001, Nature Medicine.
[24] Bruce M. Spiegelman,et al. Obesity and the Regulation of Energy Balance , 2001, Cell.
[25] J. Lawrence,et al. Protection against oxidative stress-induced insulin resistance in rat L6 muscle cells by mircomolar concentrations of alpha-lipoic acid. , 2001, Diabetes.
[26] J. Flier,et al. Obesity and insulin resistance. , 2000, The Journal of clinical investigation.
[27] D. Sorescu,et al. NAD(P)H oxidase: role in cardiovascular biology and disease. , 2000, Circulation research.
[28] T. Funahashi,et al. Molecular Mechanism of Metabolic Syndrome X: Contribution of Adipocytokines · Adipocyte‐derived Bioactive Substances , 1999, Annals of the New York Academy of Sciences.
[29] A. Rudich,et al. Oxidative Stress Disrupts Insulin-induced Cellular Redistribution of Insulin Receptor Substrate-1 and Phosphatidylinositol 3-Kinase in 3T3-L1 Adipocytes , 1999, The Journal of Biological Chemistry.
[30] T Nakamura,et al. Paradoxical decrease of an adipose-specific protein, adiponectin, in obesity. , 1999, Biochemical and biophysical research communications.
[31] J. Halaas,et al. Leptin and the regulation of body weight in mammals , 1998, Nature.
[32] A. Rudich,et al. Prolonged oxidative stress impairs insulin-induced GLUT4 translocation in 3T3-L1 adipocytes. , 1998, Diabetes.
[33] P. Zimmet,et al. Definition, diagnosis and classification of diabetes mellitus and its complications. Part 1: diagnosis and classification of diabetes mellitus. Provisional report of a WHO Consultation , 1998, Diabetic medicine : a journal of the British Diabetic Association.
[34] A. Greenberg,et al. Journal of Clinical Endocrinology and Metabolism Printed in U.S.A. Copyright © 1998 by The Endocrine Society Omental and Subcutaneous Adipose Tissues of Obese Subjects Release Interleukin-6: Depot Difference and Regulation by Glucocorticoid* , 1997 .
[35] D. Harrison,et al. Angiotensin II-mediated hypertension in the rat increases vascular superoxide production via membrane NADH/NADPH oxidase activation. Contribution to alterations of vasomotor tone. , 1996, The Journal of clinical investigation.
[36] D. Harrison,et al. Hypercholesterolemia increases endothelial superoxide anion production. , 1993, The Journal of clinical investigation.
[37] J. Flier,et al. Tissue-specific nutritional regulation of angiotensinogen in adipose tissue. , 1992, Hypertension.
[38] H. Krieger-Brauer,et al. Human fat cells possess a plasma membrane-bound H2O2-generating system that is activated by insulin via a mechanism bypassing the receptor kinase. , 1992, The Journal of clinical investigation.
[39] J. Sasaki,et al. Does superoxide underlie the pathogenesis of hypertension? , 1991, Proceedings of the National Academy of Sciences of the United States of America.
[40] T. Funahashi,et al. The concept of metabolic syndrome: contribution of visceral fat accumulation and its molecular mechanism. , 2011, Journal of atherosclerosis and thrombosis.
[41] H. Kaneto,et al. Glycation-dependent, reactive oxygen species-mediated suppression of the insulin gene promoter activity in HIT cells. , 1997, The Journal of clinical investigation.
[42] B. Spiegelman,et al. Adipose expression of tumor necrosis factor-alpha: direct role in obesity-linked insulin resistance. , 1993, Science.
[43] G. Cecchini,et al. Possible role of aldehydic lipid peroxidation products as chemoattractants. , 1987, International journal of tissue reactions.