Disruption of the Sarcoglycan–Sarcospan Complex in Vascular Smooth Muscle A Novel Mechanism for Cardiomyopathy and Muscular Dystrophy

[1]  J. Sanes,et al.  ε-Sarcoglycan Replaces α-Sarcoglycan in Smooth Muscle to Form a Unique Dystrophin-Glycoprotein Complex* , 1999, The Journal of Biological Chemistry.

[2]  J. Sanes,et al.  Membrane Targeting and Stabilization of Sarcospan Is Mediated by the Sarcoglycan Subcomplex , 1999, The Journal of cell biology.

[3]  E. Hoffman,et al.  Heart involvement in muscular dystrophies due to sarcoglycan gene mutations , 1999, Muscle & nerve.

[4]  A. Sakamoto,et al.  Delineation of genomic deletion in cardiomyopathic hamster , 1999, FEBS letters.

[5]  F. Muntoni,et al.  Mutations in the gene encoding lamin A/C cause autosomal dominant Emery-Dreifuss muscular dystrophy , 1999, Nature Genetics.

[6]  M. Martone,et al.  Enteroviral protease 2A cleaves dystrophin: Evidence of cytoskeletal disruption in an acquired cardiomyopathy , 1999, Nature Medicine.

[7]  M. Passos-Bueno,et al.  A first missense mutation in the delta sarcoglycan gene associated with a severe phenotype and frequency of limb-girdle muscular dystrophy type 2F (LGMD2F) in Brazilian sarcoglycanopathies. , 1998, Journal of medical genetics.

[8]  K. Campbell,et al.  The sarcoglycan complex in limb-girdle muscular dystrophy. , 1998, Current opinion in neurology.

[9]  John A. Faulkner,et al.  Progressive Muscular Dystrophy in α-Sarcoglycan–deficient Mice , 1998, The Journal of cell biology.

[10]  R. Wollmann,et al.  γ-Sarcoglycan Deficiency Leads to Muscle Membrane Defects and Apoptosis Independent of Dystrophin , 1998, The Journal of cell biology.

[11]  J. Towbin,et al.  The role of cytoskeletal proteins in cardiomyopathies. , 1998, Current opinion in cell biology.

[12]  K. Campbell,et al.  Sarcospan, the 25-kDa Transmembrane Component of the Dystrophin-Glycoprotein Complex* , 1997, The Journal of Biological Chemistry.

[13]  J. Fewell,et al.  A treadmill exercise regimen for identifying cardiovascular phenotypes in transgenic mice. , 1997, The American journal of physiology.

[14]  K. Campbell,et al.  Muscular dystrophies and the dystrophin-glycoprotein complex. , 1997, Current opinion in neurology.

[15]  Minoru Hongo,et al.  MLP-Deficient Mice Exhibit a Disruption of Cardiac Cytoarchitectural Organization, Dilated Cardiomyopathy, and Heart Failure , 1997, Cell.

[16]  M. Passos-Bueno,et al.  Autosomal recessive limbgirdle muscular dystrophy, LGMD2F, is caused by a mutation in the δ–sarcoglycan gene , 1996, Nature Genetics.

[17]  Y. Capetanaki,et al.  Disruption of muscle architecture and myocardial degeneration in mice lacking desmin , 1996, The Journal of cell biology.

[18]  L. Kunkel,et al.  Mutations in the Dystrophin-Associated Protein γ-Sarcoglycan in Chromosome 13 Muscular Dystrophy , 1995, Science.

[19]  J. Beckmann,et al.  Missense mutations in the adhalin gene linked to autosomal recessive muscular dystrophy , 1994, Cell.

[20]  J. Ervasti,et al.  A role for the dystrophin-glycoprotein complex as a transmembrane linker between laminin and actin , 1993, The Journal of cell biology.

[21]  H. Sweeney,et al.  Dystrophin protects the sarcolemma from stresses developed during muscle contraction. , 1993, Proceedings of the National Academy of Sciences of the United States of America.

[22]  E. Sonnenblick,et al.  Microvascular Spasm in the Cardiomyopathic Syrian Hamster: A Preventable Cause of Focal Myocardial Necrosis , 1982, Circulation.

[23]  G. Pöch,et al.  Molecular Mechanism of Action of Nicorandil , 1992, Journal of cardiovascular pharmacology.