withdrawalof superoxide in apoptosis induced by growth factor

[1]  W. Lieberthal,et al.  Lysophosphatidic acid: a novel growth and survival factor for renal proximal tubular cells. , 1997, American journal of physiology. Renal physiology.

[2]  P. Krammer,et al.  Interleukin 1β-converting Enzyme Related Proteases/Caspases Are Involved in TRAIL-induced Apoptosis of Myeloma and Leukemia Cells , 1997, The Journal of cell biology.

[3]  J S Beckman,et al.  Nitric oxide, superoxide, and peroxynitrite: the good, the bad, and ugly. , 1996, The American journal of physiology.

[4]  Junying Yuan,et al.  Human ICE/CED-3 Protease Nomenclature , 1996, Cell.

[5]  W. Lieberthal,et al.  Mechanisms of apoptosis and its potential role in renal tubular epithelial cell injury. , 1996, The American journal of physiology.

[6]  M. Walsh,et al.  Anti-phospholipid autoantibodies bind to apoptotic, but not viable, thymocytes in a beta 2-glycoprotein I-dependent manner. , 1996, Journal of immunology.

[7]  G. Salvesen,et al.  Molecular Ordering of Apoptotic Mammalian CED-3/ICE-like Proteases* , 1996, The Journal of Biological Chemistry.

[8]  M. Whyte,et al.  ICE/CED-3 proteasesin apoptosis. , 1996, Trends in cell biology.

[9]  G. Evan,et al.  A License to Kill , 1996, Cell.

[10]  Matthias Mann,et al.  FLICE, A Novel FADD-Homologous ICE/CED-3–like Protease, Is Recruited to the CD95 (Fas/APO-1) Death-Inducing Signaling Complex , 1996, Cell.

[11]  M. Raff,et al.  Role of Ced-3/ICE-family proteases in staurosporine-induced programmed cell death , 1996, The Journal of cell biology.

[12]  W. Lieberthal,et al.  Mechanisms of death induced by cisplatin in proximal tubular epithelial cells: apoptosis vs. necrosis. , 1996, The American journal of physiology.

[13]  G. Gores,et al.  The role of proteases during apoptosis , 1996, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[14]  A. Chinnaiyan,et al.  Molecular Ordering of the Cell Death Pathway , 1996, The Journal of Biological Chemistry.

[15]  M. Jacobson.  Reactive oxygen species and programmed cell death. , 1996, Trends in biochemical sciences.

[16]  H. Manev,et al.  Removal of serum from primary cultures of cerebellar granule neurons induces oxidative stress and DNA fragmentation: Protection with antioxidants and glutamate receptor antagonists , 1996, Journal of neuroscience research.

[17]  P. Amiri,et al.  Requirement of an ICE-Like Protease for Induction of Apoptosis and Ceramide Generation by REAPER , 1996, Science.

[18]  L. Ghibelli,et al.  Different basal NAD levels determine opposite effects of poly(ADP-ribosyl)polymerase inhibitors on H2O2-induced apoptosis. , 1995, Experimental cell research.

[19]  G. Cohen,et al.  An interleukin‐1β‐converting enzyme‐like protease is a common mediator of apoptosis in thymocytes , 1995, FEBS letters.

[20]  R. DuBois,et al.  Alterations in cellular adhesion and apoptosis in epithelial cells overexpressing prostaglandin endoperoxide synthase 2 , 1995, Cell.

[21]  F. Shibasaki,et al.  Calcineurin functions in Ca(2+)-activated cell death in mammalian cells , 1995, The Journal of cell biology.

[22]  J. Mankovich,et al.  Inhibition of ICE family proteases by baculovirus antiapoptotic protein p35. , 1995, Science.

[23]  M. Su,et al.  Cleavage of Poly(ADP-ribose) Polymerase by Interleukin-1β Converting Enzyme and Its Homologs TX and Nedd-2 (*) , 1995, The Journal of Biological Chemistry.

[24]  Seamus J. Martin,et al.  Protease activation during apoptosis: Death by a thousand cuts? , 1995, Cell.

[25]  Patrick R. Griffin,et al.  Identification and inhibition of the ICE/CED-3 protease necessary for mammalian apoptosis , 1995, Nature.

[26]  Muneesh Tewari,et al.  Yama/CPP32β, a mammalian homolog of CED-3, is a CrmA-inhibitable protease that cleaves the death substrate poly(ADP-ribose) polymerase , 1995, Cell.

[27]  A. Hoffman,et al.  Renal growth hormone--insulin-like growth factor-I system in acute renal failure. , 1995, Kidney international.

[28]  P. Gallop,et al.  An NADPH oxidase superoxide-generating system in the rabbit aorta. , 1995, The American journal of physiology.

[29]  M. Raff,et al.  Programmed cell death and Bcl-2 protection in very low oxygen , 1995, Nature.

[30]  S. Miller,et al.  Therapeutic use of growth factors in renal failure. , 1994, Journal of the American Society of Nephrology : JASN.

[31]  R. Ashman,et al.  Decreased membrane phospholipid packing and decreased cell size precede DNA cleavage in mature mouse B cell apoptosis. , 1994, Journal of immunology.

[32]  R. Zucker,et al.  Comparison of cellular and nuclear flow cytometric techniques for discriminating apoptotic subpopulations. , 1994, Experimental cell research.

[33]  A. Tolkovsky,et al.  Characterization of apoptosis in cultured rat sympathetic neurons after nerve growth factor withdrawal , 1994, The Journal of cell biology.

[34]  Z. Oltvai,et al.  Bcl-2 functions in an antioxidant pathway to prevent apoptosis , 1993, Cell.

[35]  S. Masino,et al.  Renal mouse proximal tubular cells are more susceptible than MDCK cells to chemical anoxia. , 1993, The American journal of physiology.

[36]  X. M. Sun,et al.  Increased membrane permeability of apoptotic thymocytes: a flow cytometric study. , 1993, Cytometry.

[37]  X. M. Sun,et al.  A flow-cytometric method for the separation and quantitation of normal and apoptotic thymocytes. , 1992, Analytical biochemistry.

[38]  M. Raff,et al.  Social controls on cell survival and cell death , 1992, Nature.

[39]  W. Swat,et al.  Detection of apoptosis of immature CD4+8+ thymocytes by flow cytometry. , 1991, Journal of immunological methods.

[40]  J. Messana,et al.  Epidermal growth factor enhances renal tubule cell regeneration and repair and accelerates the recovery of renal function in postischemic acute renal failure. , 1989, The Journal of clinical investigation.

[41]  H. Gyllenhammar,et al.  Lucigenin chemiluminescence in the assessment of neutrophil superoxide production. , 1987, Journal of immunological methods.

[42]  A. Wyllie,et al.  Apoptosis: A Basic Biological Phenomenon with Wide-ranging Implications in Tissue Kinetics , 1972, British Journal of Cancer.

[43]  D. Wheeler,et al.  Lipid peroxidation contributes to hydrogen peroxide induced cytotoxicity in renal epithelial cells. , 1996, Kidney international.

[44]  J. Tilly,et al.  Inhibitors of oxidative stress mimic the ability of follicle-stimulating hormone to suppress apoptosis in cultured rat ovarian follicles. , 1995, Endocrinology.

[45]  W. Lieberthal,et al.  Functional and cytoskeletal changes induced by sublethal injury in proximal tubular epithelial cells. , 1994, The American journal of physiology.

[46]  D. Martin,et al.  Hepatocyte growth factor accelerates recovery from acute ischemic renal injury in rats. , 1994, The American journal of physiology.

[47]  T. Buttke,et al.  Oxidative stress as a mediator of apoptosis. , 1994, Immunology today.

[48]  F. Toback,et al.  Regeneration after acute tubular necrosis. , 1992, Kidney international.