Analysis of amino-terminal variants of amyloid-β peptides by capillary isoelectric focusing immunoassay.
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J. Schuchhardt | J. Wiltfang | H. Zetterberg | E. Portelius | O. Jahn | H. Klafki | Ute Haussmann | H. Knölker | C. Bauer | T. Liepold | Philipp Linning | C. Janßen
[1] J. Wiltfang,et al. Optimisation of BACE1 inhibition of tripartite structures by modification of membrane anchors, spacers and pharmacophores - development of potential agents for the treatment of Alzheimer's disease. , 2012, Organic & biomolecular chemistry.
[2] Kevin Mills,et al. Reference measurement procedures for Alzheimer's disease cerebrospinal fluid biomarkers: definitions and approaches with focus on amyloid β42. , 2012, Biomarkers in medicine.
[3] H. Rosenmann. CSF Biomarkers for Amyloid and Tau Pathology in Alzheimer's Disease , 2012, Journal of Molecular Neuroscience.
[4] Erin E. Chambers,et al. Quantitation of amyloid beta peptides Aβ(1-38), Aβ(1-40), and Aβ(1-42) in human cerebrospinal fluid by ultra-performance liquid chromatography-tandem mass spectrometry. , 2011, Analytical biochemistry.
[5] J. Wiltfang,et al. β-Amyloid Peptide Variants in Brains and Cerebrospinal Fluid from Amyloid Precursor Protein (APP) Transgenic Mice , 2011, The Journal of Biological Chemistry.
[6] J. Wiltfang,et al. Structural design, solid-phase synthesis and activity of membrane-anchored β-secretase inhibitors on Aβ generation from wild-type and Swedish-mutant APP. , 2010, Chemistry.
[7] E. Mandelkow,et al. Aβ Oligomers Cause Localized Ca2+ Elevation, Missorting of Endogenous Tau into Dendrites, Tau Phosphorylation, and Destruction of Microtubules and Spines , 2010, The Journal of Neuroscience.
[8] G. Multhaup,et al. Aberrant Amyloid Precursor Protein (APP) Processing in Hereditary Forms of Alzheimer Disease Caused by APP Familial Alzheimer Disease Mutations Can Be Rescued by Mutations in the APP GxxxG Motif* , 2010, The Journal of Biological Chemistry.
[9] Shaomin Li,et al. Amyloid-β protein dimers isolated directly from Alzheimer's brains impair synaptic plasticity and memory , 2008, Nature Medicine.
[10] J. Schulz,et al. Efficient Inhibition of the Alzheimer's Disease β-Secretase by Membrane Targeting , 2008, Science.
[11] P. Lewczuk,et al. Urea‐based two‐dimensional electrophoresis of beta‐amyloid peptides in human plasma: Evidence for novel Aβ species , 2007, Proteomics.
[12] Bernardo L Sabatini,et al. Natural Oligomers of the Alzheimer Amyloid-β Protein Induce Reversible Synapse Loss by Modulating an NMDA-Type Glutamate Receptor-Dependent Signaling Pathway , 2007, The Journal of Neuroscience.
[13] O. Jahn,et al. Technical innovations for the automated identification of gel-separated proteins by MALDI-TOF mass spectrometry , 2006, Analytical and bioanalytical chemistry.
[14] Henrik Zetterberg,et al. Determination of β-Amyloid Peptide Signatures in Cerebrospinal Fluid Using Immunoprecipitation-Mass Spectrometry , 2006 .
[15] Anders Wallin,et al. Amino-Truncated β-Amyloid42 Peptides in Cerebrospinal Fluid and Prediction of Progression of Mild Cognitive Impairment , 2005 .
[16] P. Lewczuk,et al. Electrophoretic separation of amyloid β peptides in plasma , 2004 .
[17] T. Tabira,et al. Amino‐truncated amyloid β‐peptide (Aβ5‐40/42) produced from caspase‐cleaved amyloid precursor protein is deposited in Alzheimer’s disease brain , 2004 .
[18] A. Delacourte,et al. Truncated beta‐amyloid peptide species in pre‐clinical Alzheimer's disease as new targets for the vaccination approach , 2003, Journal of neurochemistry.
[19] J. Kornhuber,et al. Highly conserved and disease‐specific patterns of carboxyterminally truncated Aβ peptides 1–37/38/39 in addition to 1–40/42 in Alzheimer's disease and in patients with chronic neuroinflammation , 2002, Journal of neurochemistry.
[20] M. Emmerling,et al. Isolation, chemical characterization, and quantitation of A beta 3-pyroglutamyl peptide from neuritic plaques and vascular amyloid deposits. , 1997, Biochemical and biophysical research communications.
[21] D. Mann,et al. Dominant and differential deposition of distinct β-amyloid peptide species, Aβ N3(pE), in senile plaques , 1995, Neuron.
[22] G. Glenner,et al. Alzheimer's disease: Initial report of the purification and characterization of a novel cerebrovascular amyloid protein , 1984 .