The risk of transmitting prion disease by blood or plasma products.
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[1] J Mackenzie,et al. Possible transmission of variant Creutzfeldt-Jakob disease by blood transfusion , 2004, The Lancet.
[2] E. Rodriguez-Merchan,et al. Radioactive synoviorthesis for the treatment of haemophilic synovitis , 2007, Haemophilia : the official journal of the World Federation of Hemophilia.
[3] R. Will,et al. Creutzfeldt–Jakob disease and blood transfusion: results of the UK Transfusion Medicine Epidemiological Review study , 2006, BMJ.
[4] P. Brown. Transmissible spongiform encephalopathy in the 21st century , 2008, Neurology.
[5] J. Ironside,et al. Prevalence of disease related prion protein in anonymous tonsil specimens in Britain: cross sectional opportunistic survey , 2009, BMJ : British Medical Journal.
[6] Andrew F. Hill,et al. Molecular analysis of prion strain variation and the aetiology of 'new variant' CJD , 1996, Nature.
[7] P. Brown. Creutzfeldt‐Jakob disease: reflections on the risk from blood product therapy , 2007, Haemophilia : the official journal of the World Federation of Hemophilia.
[8] A. Molesworth,et al. Variant Creutzfeldt‐Jakob disease and exposure to fractionated plasma products , 2009, Vox sanguinis.
[9] J. Ironside,et al. Predicting susceptibility and incubation time of human-to-human transmission of vCJD , 2005, The Lancet Neurology.
[10] Peter Rudge,et al. Variant CJD in an individual heterozygous for PRNP codon 129 , 2009, The Lancet.
[11] Paul Clarke,et al. Projections of the future course of the primary vCJD epidemic in the UK: inclusion of subclinical infection and the possibility of wider genetic susceptibility , 2005, Journal of The Royal Society Interface.
[12] R. Knight. The Relationship between New Variant Creutzfeldt–Jakob Disease and Bovine Spongiform Encephalopathy , 1999, Vox Sanguinis.
[13] Mark Penney,et al. Prevalence of lymphoreticular prion protein accumulation in UK tissue samples , 2004, The Journal of pathology.
[14] S. Prusiner,et al. Prion biology and diseases. , 1999, Harvey lectures.
[15] O. Simell,et al. The normal population distribution of PRNP codon 129 polymorphism , 2003, Acta neurologica Scandinavica.
[16] N. Hunter,et al. Transmission of BSE by blood transfusion in sheep , 2000, The Lancet.
[17] M. Turner,et al. Managing the risk of transmission of variant Creutzfeldt Jakob disease by blood products , 2006, British journal of haematology.
[18] M. Head,et al. Preclinical vCJD after blood transfusion in a PRNP codon 129 heterozygous patient , 2004, The Lancet.
[19] N. Hunter,et al. Prion diseases are efficiently transmitted by blood transfusion in sheep. , 2008, Blood.
[20] L. McShane,et al. Further studies of blood infectivity in an experimental model of transmissible spongiform encephalopathy, with an explanation of why blood components do not transmit Creutzfeldt‐Jakob disease in humans , 1999, Transfusion.
[21] R. Carbonell,et al. Reduction in infectivity of endogenous transmissible spongiform encephalopathies present in blood by adsorption to selective affinity resins , 2006, The Lancet.
[22] S. Cousens,et al. Transmissions to mice indicate that ‘new variant’ CJD is caused by the BSE agent , 1997, Nature.
[23] N. Hunter,et al. Transmission of prion diseases by blood transfusion. , 2002, The Journal of general virology.
[24] S. Love,et al. Variant CJD infection in the spleen of a neurologically asymptomatic UK adult patient with haemophilia , 2010, Haemophilia : the official journal of the World Federation of Hemophilia.
[25] Antonio Giulivi,et al. Effectiveness of leucoreduction for removal of infectivity of transmissible spongiform encephalopathies from blood , 2004, The Lancet.
[26] Suvankar Pal,et al. Clinical presentation and pre-mortem diagnosis of variant Creutzfeldt-Jakob disease associated with blood transfusion: a case report , 2006, The Lancet.
[27] P. Brown,et al. The distribution of infectivity in blood components and plasma derivatives in experimental models of transmissible spongiform encephalopathy , 1998, Transfusion.