Interleukin‐1β (IL‐1β) increases pain behavior and the blood glucose level: Possible involvement of sympathetic nervous system
暂无分享,去创建一个
H. Suh | Soo-Hyun Park | Jun-Sub Jung | Y. Sim | O. Ryu | M. Choi | Yu-jung Kang | Yu-Jung Kang
[1] H. Suh,et al. Antidepressant-like effect of chlorogenic acid isolated from Artemisia capillaris Thunb. , 2010 .
[2] B. McEwen,et al. The role of corticosteroids and stress in chronic pain conditions. , 2010, Metabolism: clinical and experimental.
[3] R. Seeley,et al. Targeting the CNS to treat type 2 diabetes , 2009, Nature Reviews Drug Discovery.
[4] L. Niskanen,et al. Chronic pain, impaired glucose tolerance and diabetes: A community-based study , 2008, PAIN®.
[5] R. Albertini,et al. Low Level Laser Therapy (LLLT) Decreases Pulmonary Microvascular Leakage, Neutrophil Influx and IL-1β Levels in Airway and Lung from Rat Subjected to LPS-Induced Inflammation , 2008, Inflammation.
[6] C. Coe,et al. Altered cytokine levels in the blood and cerebrospinal fluid of chronic pain patients , 2008, Journal of Neuroimmunology.
[7] H. Suh,et al. Changes in pain behavior induced by formalin, substance P, glutamate and pro-inflammatory cytokines in immobilization-induced stress mouse model , 2006, Brain Research Bulletin.
[8] H. Suh,et al. Effect of Aspirin and Acetaminophen on Proinflammatory Cytokine-Induced Pain Behavior in Mice , 2005, Pharmacology.
[9] H. Suh,et al. Differential Modulatory Effects of Cholera Toxin and Pertussis Toxin on Pain Behavior Induced by TNF-a, lnterleukin-1β and Interferon- Injected Intrathecally , 2005 .
[10] A. Gürol,et al. Losartan may prevent the elevation of plasma glucose, corticosterone and catecholamine levels induced by chronic stress , 2004, Journal of the renin-angiotensin-aldosterone system : JRAAS.
[11] J. Delgado-García,et al. Analgesia Induced by Dietary Restriction Is Mediated by the κ-Opioid System , 2003, The Journal of Neuroscience.
[12] H. Suh,et al. Antinociceptive mechanisms of orally administered decursinol in the mouse. , 2003, Life sciences.
[13] G. Blackburn-Munro,et al. Pain in the brain: are hormones to blame? , 2003, Trends in Endocrinology & Metabolism.
[14] C. Tsigos,et al. Hypothalamic-pituitary-adrenal axis, neuroendocrine factors and stress. , 2002, Journal of psychosomatic research.
[15] T. van der Poll,et al. Role of interleukin-1 in the pulmonary immune response during Pseudomonas aeruginosa pneumonia. , 2002, American journal of physiology. Lung cellular and molecular physiology.
[16] Adam Sapirstein,et al. Interleukin-1β-mediated induction of Cox-2 in the CNS contributes to inflammatory pain hypersensitivity , 2001, Nature.
[17] Bruce S. McEwen,et al. The neurobiology of stress: from serendipity to clinical relevance. , 2000, Brain research.
[18] A. Reeve,et al. Intrathecally administered endotoxin or cytokines produce allodynia, hyperalgesia and changes in spinal cord neuronal responses to nociceptive stimuli in the rat , 2000, European journal of pain.
[19] H. Suh,et al. Cycloheximide increases proenkephalin and tyrosine hydroxylase gene expression in rat adrenal medulla. , 2000, Molecular pharmacology.
[20] W. Young,et al. Role of Glycemia in Acute Spinal Cord Injury: Data from a Rat Experimental Model and Clinical Experience , 1999, Annals of the New York Academy of Sciences.
[21] K. Tan-No,et al. Induction of nociceptive responses by intrathecal injection of interleukin-1 in mice. , 1999, Life sciences.
[22] J. Deleo,et al. Acute peripheral inflammation induces moderate glial activation and spinal IL-1β expression that correlates with pain behavior in the rat 1 Published on the World Wide Web on 17 March 1999. 1 , 1999, Brain Research.
[23] H. Suh,et al. Effects of histamine receptor antagonists injected intrathecally on antinociception induced by opioids administered intracerebroventricularly in the mouse , 1999, Neuropeptides.
[24] M. Herkenham,et al. Induction of pro-inflammatory cytokine mRNAs in the brain after peripheral injection of subseptic doses of lipopolysaccharide in the rat , 1999, Journal of Neuroimmunology.
[25] H. Suh,et al. Differential effects of adenosine receptor antagonists injected intrathecally on antinociception induced by morphine and β-endorphin administered intracerebroventricularly in the mouse , 1997, Neuropeptides.
[26] J. Wrathall,et al. Increase of interleukin-1β mRNA and protein in the spinal cord following experimental traumatic injury in the rat , 1997, Brain Research.
[27] H. Suh,et al. Antinociceptive mechanisms of dipsacus saponin C administered intracerebroventricularly in the mouse. , 1996, General pharmacology.
[28] S. Maier,et al. Characterization of cytokine-induced hyperalgesia , 1994, Brain Research.
[29] S. Satoh,et al. Glucoregulatory hormones in the immobilization stress-induced increase of plasma glucose in fasted and fed rats. , 1993, Endocrinology.
[30] M. Irwin,et al. Sympathetic nervous system mediates central corticotropin-releasing factor induced suppression of natural killer cytotoxicity. , 1990, The Journal of pharmacology and experimental therapeutics.
[31] P. Chomczyński,et al. Single-step method of RNA isolation by acid guanidinium thiocyanate-phenol-chloroform extraction. , 1987, Analytical biochemistry.
[32] B. McEwen,et al. The neuroendocrinology of stress and aging: the glucocorticoid cascade hypothesis. , 1986, Endocrine reviews.
[33] G. Smythe,et al. Relationships between brain noradrenergic activity and blood glucose , 1984, Nature.
[34] C. A. Benzo. The hypothalamus and blood glucose regulation , 1983 .
[35] G. Wilcox,et al. Intrathecal substance P elicits a caudally-directed biting and scratching behavior in mice , 1981, Brain Research.
[36] J. Exton,et al. Studies on the alpha-adrenergic activation of hepatic glucose output. I. Studies on the alpha-adrenergic activation of phosphorylase and gluconeogenesis and inactivation of glycogen synthase in isolated rat liver parenchymal cells. , 1976, The Journal of biological chemistry.
[37] T. O'donnell,et al. Energy Metabolism in Sepsis: Treatment Based on Different Patterns in Shock and High Output Stage , 1974, Annals of surgery.
[38] D. Porte,et al. A mechanism for regulation of insulin release in hypoxia. , 1972, The American journal of physiology.
[39] D. Dillard,et al. Metabolic aspects of deep surgical hypothermia in infancy. , 1968, Pediatrics.
[40] H. Suh,et al. Differential modulatory effects of cholera toxin and pertussis toxin on pain behavior induced by TNF-alpha, interleukin-1beta and interferon-gamma injected intrathecally. , 2005, Archives of pharmacal research.
[41] J. Venter,et al. Studies on the a-Adrenergic Activation of Hepatic Glucose Output , 2001 .
[42] David A Hildeman,et al. Immunopathologic weight loss in intracranial LCMV infection initiated by the anorexigenic effects of IL-1beta. , 2000, Viral immunology.