The neurobiology of depression—revisiting the serotonin hypothesis. I. Cellular and molecular mechanisms
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[1] P. Lavori,et al. Multiple recurrences of major depressive disorder. , 2000, The American journal of psychiatry.
[2] Norito Kawakami,et al. Lifetime prevalence and age-of-onset distributions of mental disorders in the World Health Organization's World Mental Health Survey Initiative. , 2007, World psychiatry : official journal of the World Psychiatric Association.
[3] Janet B W Williams,et al. Diagnostic and Statistical Manual of Mental Disorders , 2013 .
[4] A. Cantor,et al. The relationship between age and the fatty acid composition of cerebral cortex and erythrocytes in human subjects , 2001, Brain Research Bulletin.
[5] Meena Vythilingam,et al. The psychobiology of depression and resilience to stress: implications for prevention and treatment. , 2005, Annual review of clinical psychology.
[6] E. Evers,et al. Effects of Acute Tryptophan Depletion on Mood and Facial Emotion Perception Related Brain Activation and Performance in Healthy Women with and without a Family History of Depression , 2007, Neuropsychopharmacology.
[7] P. Gaspar,et al. Probing the diversity of serotonin neurons , 2012, Philosophical Transactions of the Royal Society B: Biological Sciences.
[8] M. Caron,et al. The 5-HT deficiency theory of depression: perspectives from a naturalistic 5-HT deficiency model, the tryptophan hydroxylase 2Arg439His knockin mouse , 2012, Philosophical Transactions of the Royal Society B: Biological Sciences.
[9] Keri Martinowich,et al. Interaction between BDNF and Serotonin: Role in Mood Disorders , 2008, Neuropsychopharmacology.
[10] C. Benkelfat,et al. Mood-lowering effect of tryptophan depletion. Enhanced susceptibility in young men at genetic risk for major affective disorders. , 1994, Archives of general psychiatry.
[11] K. Lesch,et al. Targeting brain serotonin synthesis: insights into neurodevelopmental disorders with long-term outcomes related to negative emotionality, aggression and antisocial behaviour , 2012, Philosophical Transactions of the Royal Society B: Biological Sciences.
[12] L. Descarries,et al. Effects of the antidepressant fluoxetine on the subcellular localization of 5-HT1A receptors and SERT , 2012, Philosophical Transactions of the Royal Society B: Biological Sciences.
[13] W. Coryell,et al. Recurrence after recovery from major depressive disorder during 15 years of observational follow-up. , 1999, The American journal of psychiatry.
[14] R. Belmaker,et al. Major depressive disorder. , 2008, The New England journal of medicine.
[15] René Hen,et al. Serotonin receptor expression along the dorsal–ventral axis of mouse hippocampus , 2012, Philosophical Transactions of the Royal Society B: Biological Sciences.
[16] J. Schildkraut,et al. The catecholamine hypothesis of affective disorders. A review of supporting evidence. , 1967, International journal of psychiatry.
[17] Alan D. Lopez,et al. Evidence-Based Health Policy--Lessons from the Global Burden of Disease Study , 1996, Science.
[18] M. Egan,et al. Serotonin Transporter Genetic Variation and the Response of the Human Amygdala , 2002, Science.
[19] A. Coppen. The Biochemistry of Affective Disorders , 1967, British Journal of Psychiatry.
[20] C. Mathers,et al. Projections of Global Mortality and Burden of Disease from 2002 to 2030 , 2006, PLoS medicine.
[21] A. Caspi,et al. Influence of Life Stress on Depression: Moderation by a Polymorphism in the 5-HTT Gene , 2003, Science.
[22] M. Meaney,et al. Maternal licking regulates hippocampal glucocorticoid receptor transcription through a thyroid hormone–serotonin–NGFI-A signalling cascade , 2012, Philosophical Transactions of the Royal Society B: Biological Sciences.
[23] E. Walker,et al. Diagnostic and Statistical Manual of Mental Disorders , 2013 .
[24] A. Meyer-Lindenberg,et al. 5-HTTLPR polymorphism impacts human cingulate-amygdala interactions: a genetic susceptibility mechanism for depression , 2005, Nature Neuroscience.
[25] S. Kennedy,et al. Differential pharmacological treatment response in high angry hostile and low angry hostile depressed patients: a retrospective analysis. , 1997, Journal of affective disorders.
[26] Nanxin Li,et al. A neurotrophic hypothesis of depression: role of synaptogenesis in the actions of NMDA receptor antagonists , 2012, Philosophical Transactions of the Royal Society B: Biological Sciences.
[27] David Julius,et al. Cellular and Molecular Mechanisms of Pain , 2009, Cell.
[28] P. Albert. Transcriptional regulation of the 5-HT1A receptor: implications for mental illness , 2012, Philosophical Transactions of the Royal Society B: Biological Sciences.
[29] J. Beaulieu,et al. Selective deletion of forebrain glycogen synthase kinase 3β reveals a central role in serotonin-sensitive anxiety and social behaviour , 2012, Philosophical Transactions of the Royal Society B: Biological Sciences.
[30] K. Lesch,et al. Association of Anxiety-Related Traits with a Polymorphism in the Serotonin Transporter Gene Regulatory Region , 1996, Science.
[31] R. Hen,et al. Early-Life Blockade of the 5-HT Transporter Alters Emotional Behavior in Adult Mice , 2004, Science.
[32] L. Lanfumey,et al. Beyond the monoaminergic hypothesis: neuroplasticity and epigenetic changes in a transgenic mouse model of depression , 2012, Philosophical Transactions of the Royal Society B: Biological Sciences.
[33] Patricia M. Whitaker-Azmitia,et al. Serotonin and brain development: role in human developmental diseases , 2001, Brain Research Bulletin.
[34] P A Sargent,et al. Persistent reduction in brain serotonin1A receptor binding in recovered depressed men measured by positron emission tomography with [11C]WAY-100635 , 2004, Molecular Psychiatry.