Modulation of ventricular repolarization by a premature stimulus. Role of epicardial dispersion of repolarization kinetics demonstrated by optical mapping of the intact guinea pig heart.
暂无分享,去创建一个
[1] D. Rosenbaum,et al. Unique Properties of Cardiac Action Potentials Recorded with Voltage‐Sensitive Dyes , 1996, Journal of cardiovascular electrophysiology.
[2] D. Rosenbaum,et al. Optical mapping in a new guinea pig model of ventricular tachycardia reveals mechanisms for multiple wavelengths in a single reentrant circuit. , 1996, Circulation.
[3] G. Salama,et al. Optical mapping reveals that repolarization spreads anisotropically and is guided by fiber orientation in guinea pig hearts. , 1995, Circulation research.
[4] Y Rudy,et al. Two components of the delayed rectifier K+ current in ventricular myocytes of the guinea pig type. Theoretical formulation and their role in repolarization. , 1995, Circulation research.
[5] C. Antzelevitch,et al. Characteristics of the delayed rectifier current (IKr and IKs) in canine ventricular epicardial, midmyocardial, and endocardial myocytes. A weaker IKs contributes to the longer action potential of the M cell. , 1995, Circulation research.
[6] W. Schütz,et al. Differential effect of dofetilide on ventricular repolarization during steady state and during restitution in vivo. , 1994, Journal of cardiovascular pharmacology.
[7] G. Salama,et al. Optical Mapping of Repolarization and Refractoriness From Intact Hearts , 1994, Circulation.
[8] J. Davidenko,et al. Effects of diacetyl monoxime on the electrical properties of sheep and guinea pig ventricular muscle. , 1993, Cardiovascular research.
[9] H. Refsum,et al. Effects of temperature on cycle length dependent changes and restitution of action potential duration in guinea pig ventricular muscle. , 1993, Cardiovascular research.
[10] C Antzelevitch,et al. Ionic bases for electrophysiological distinctions among epicardial, midmyocardial, and endocardial myocytes from the free wall of the canine left ventricle. , 1993, Circulation research.
[11] M. Sanguinetti,et al. Rate-dependent prolongation of cardiac action potentials by a methanesulfonanilide class III antiarrhythmic agent. Specific block of rapidly activating delayed rectifier K+ current by dofetilide. , 1993, Circulation research.
[12] W Peters,et al. Cellular Mechanisms of Differential Action Potential Duration Restitution in Canine Ventricular Muscle Cells During Single Versus Double Premature Stimuli , 1992, Circulation.
[13] E. Carmeliet. Voltage- and time-dependent block of the delayed K+ current in cardiac myocytes by dofetilide. , 1992, The Journal of pharmacology and experimental therapeutics.
[14] E. Rowland,et al. Dispersion of monophasic action potential duration: demonstrable in humans after premature ventricular extrastimulation but not in steady state. , 1992, Journal of the American College of Cardiology.
[15] P. Tchou,et al. Induction of Ventricular Fibrillation Versus Monomorphic Ventricular Tachycardia During Programmed Stimulation: Role of Premature Beat Conduction Delay , 1992, Circulation.
[16] R. Myerburg,et al. Potassium rectifier currents differ in myocytes of endocardial and epicardial origin. , 1992, Circulation research.
[17] G. Gintant,et al. Heterogeneity within the ventricular wall. Electrophysiology and pharmacology of epicardial, endocardial, and M cells. , 1991, Circulation research.
[18] W. Giles,et al. Regional variations in action potentials and transient outward current in myocytes isolated from rabbit left ventricle. , 1991, The Journal of physiology.
[19] D. T. Kaplan,et al. Repolarization Inhomogeneities in Ventricular Myocardium Change Dynamically With Abrupt Cycle Length Shortening , 1991, Circulation.
[20] M S Spach,et al. Interaction of Inhomogeneities of Repolarization With Anisotropic Propagation in Dog Atria: Mechanism for Both Preventing and Initiating Reentry , 1989, Circulation research.
[21] J. C. Bailey,et al. Action potential duration alternans in dog Purkinje and ventricular muscle fibers. Further evidence in support of two different mechanisms. , 1989, Circulation.
[22] Michael D. Lesh,et al. Cellular Uncoupling Can Unmask Dispersion of Action Potential Duration in Ventricular Myocardium A Computer Modeling Study , 1989, Circulation research.
[23] C Antzelevitch,et al. Rate dependence of action potential duration and refractoriness in canine ventricular endocardium differs from that of epicardium: role of the transient outward current. , 1989, Journal of the American College of Cardiology.
[24] J A Vassallo,et al. Nonuniform recovery of excitability in the left ventricle. , 1988, Circulation.
[25] M. Franz,et al. Cycle length dependence of human action potential duration in vivo. Effects of single extrastimuli, sudden sustained rate acceleration and deceleration, and different steady-state frequencies. , 1988, The Journal of clinical investigation.
[26] J Toyama,et al. Effects of activation sequence and anisotropic cellular geometry on the repolarization phase of action potential of dog ventricular muscles. , 1987, Circulation.
[27] G. Salama,et al. Maps of optical action potentials and NADH fluorescence in intact working hearts. , 1987, The American journal of physiology.
[28] P M Rautaharju,et al. Ventricular action potentials, ventricular extracellular potentials, and the ECG of guinea pig. , 1985, Circulation research.
[29] B. Surawicz,et al. Dispersion of ventricular repolarization and arrhythmia: study of two consecutive ventricular premature complexes. , 1985, Circulation.
[30] B. Surawicz,et al. Cycle length effect on restitution of action potential duration in dog cardiac fibers. , 1983, The American journal of physiology.
[31] B. Surawicz,et al. Characteristics and Possible Mechanism of Ventricular Arrhythmia Dependent on the Dispersion of Action Potential Durations , 1983, Circulation.
[32] M J Lab,et al. Contraction-excitation feedback in myocardium. Physiological basis and clinical relevance. , 1982, Circulation research.
[33] B. R. Jewell,et al. A study of the factors responsible for rate‐dependent shortening of the action potential in mammalian ventricular muscle. , 1978, The Journal of physiology.
[34] M. Burgess,et al. Electrotonic Interaction during Canine Ventricular Repolarization , 1978, Circulation research.
[35] B. G. Bass. Restitution of the action potential in cat papillary muscle. , 1975, The American journal of physiology.
[36] D. Noble,et al. The dependence of plateau currents in cardiac Purkinje fibres on the interval between action potentials , 1972, The Journal of physiology.
[37] G. Moe,et al. Fibrillation Threshold of Premature Ventricular Responses , 1966, Circulation research.
[38] G. Moe,et al. Nonuniform Recovery of Excitability in Ventricular Muscle , 1964, Circulation research.
[39] Carl J. Wiggers,et al. VENTRICULAR FIBRILLATION DUE TO SINGLE, LOCALIZED INDUCTION AND CONDENSER SHOCKS APPLIED DURING THE VULNERABLE PHASE OF VENTRICULAR SYSTOLE , 1940 .
[40] H. Bazett,et al. AN ANALYSIS OF THE TIME‐RELATIONS OF ELECTROCARDIOGRAMS. , 1997 .