Unaltered influenza disease outcomes in swine prophylactically treated with α-galactosylceramide.

[1]  S. Blome,et al.  Porcine Invariant Natural Killer T Cells: Functional Profiling and Dynamics in Steady State and Viral Infections , 2019, Front. Immunol..

[2]  A. Holian,et al.  Mapping of Dynamic Transcriptome Changes Associated With Silica-Triggered Autoimmune Pathogenesis in the Lupus-Prone NZBWF1 Mouse , 2019, Front. Immunol..

[3]  R. Sachidanandam,et al.  Next Generation Sequencing of the Pig αβ TCR Repertoire Identifies the Porcine Invariant NKT Cell Receptor , 2019, The Journal of Immunology.

[4]  Weng-Lang Yang,et al.  Activation of Invariant Natural Killer T Cells Redirects the Inflammatory Response in Neonatal Sepsis , 2018, Front. Immunol..

[5]  B. Artiaga,et al.  Characterizing porcine invariant natural killer T cells: A comparative study with NK cells and T cells , 2017, Developmental and comparative immunology.

[6]  L. Tedeschi,et al.  Investigation of the effect of pegbovigrastim on some periparturient immune disorders and performance in Mexican dairy herds. , 2017, Journal of dairy science.

[7]  S. Salek-Ardakani,et al.  Rapid control of pandemic H1N1 influenza by targeting NKT-cells , 2016, Scientific Reports.

[8]  Yasuhiro Yamasaki,et al.  Corrigendum: A metabolic profile in Ruditapes philippinarum associated with growth-promoting effects of alginate hydrolysates , 2016, Scientific Reports.

[9]  S. Salek-Ardakani,et al.  α-Galactosylceramide protects swine against influenza infection when administered as a vaccine adjuvant , 2016, Scientific Reports.

[10]  Morten H. H. Nørholm,et al.  Generation of mutation hotspots in ageing bacterial colonies , 2016, Scientific Reports.

[11]  Yan Zhou,et al.  Influence of maternally-derived antibodies on live attenuated influenza vaccine efficacy in pigs. , 2015, Vaccine.

[12]  A. Vincent,et al.  Swine as a model for influenza A virus infection and immunity. , 2015, ILAR journal.

[13]  S. Salek-Ardakani,et al.  Targeted disruption of CD1d prevents NKT cell development in pigs , 2015, Mammalian Genome.

[14]  M. Lang,et al.  CD1d-dependent expansion of NKT follicular helper cells in vivo and in vitro is a product of cellular proliferation and differentiation. , 2015, International immunology.

[15]  J. Roth,et al.  Optimal Use of Vaccines for Control of Influenza A Virus in Swine , 2015, Vaccines.

[16]  B. Artiaga,et al.  Adjuvant effects of therapeutic glycolipids administered to a cohort of NKT cell-diverse pigs. , 2014, Veterinary immunology and immunopathology.

[17]  B. Janke Influenza A Virus Infections in Swine , 2014, Veterinary pathology.

[18]  A. Herbelin,et al.  Identification of invariant natural killer T cells in porcine peripheral blood. , 2012, Veterinary immunology and immunopathology.

[19]  Olivier White,et al.  Active Collisions in Altered Gravity Reveal Eye-Hand Coordination Strategies , 2012, PloS one.

[20]  J. Sirard,et al.  Key role for respiratory CD103(+) dendritic cells, IFN-γ, and IL-17 in protection against Streptococcus pneumoniae infection in response to α-galactosylceramide. , 2012, The Journal of infectious diseases.

[21]  智典 伊豫田,et al.  樹状細胞を介する invariant natural killer T 細胞の寛容誘導 , 2012 .

[22]  S. Gubbins,et al.  Immune Responses in Pigs Vaccinated with Adjuvanted and Non-Adjuvanted A(H1N1)pdm/09 Influenza Vaccines Used in Human Immunization Programmes , 2012, PloS one.

[23]  A. McMichael,et al.  Pivotal Advance: Invariant NKT cells reduce accumulation of inflammatory monocytes in the lungs and decrease immune‐pathology during severe influenza A virus infection , 2012, Journal of leukocyte biology.

[24]  J. Renauld,et al.  Interleukin-22 Is Produced by Invariant Natural Killer T Lymphocytes during Influenza A Virus Infection , 2012, The Journal of Biological Chemistry.

[25]  Dapeng Zhou,et al.  Intranasal but not intravenous delivery of the adjuvant α‐galactosylceramide permits repeated stimulation of natural killer T cells in the lung , 2011, European journal of immunology.

[26]  G. Besra,et al.  Innate and cytokine-driven signals, rather than microbial antigens, dominate in natural killer T cell activation during microbial infection , 2011, The Journal of experimental medicine.

[27]  Xiangming Li,et al.  Functional Invariant NKT Cells in Pig Lungs Regulate the Airway Hyperreactivity: A Potential Animal Model , 2011, Journal of Clinical Immunology.

[28]  M. Smyth,et al.  Presumed guilty: natural killer T cell defects and human disease , 2011, Nature Reviews Immunology.

[29]  Tetsuya Matsumoto,et al.  IFN-γ production downstream of NKT cell activation in mice infected with influenza virus enhances the cytolytic activities of both NK cells and viral antigen-specific CD8+ T cells. , 2010, Virology.

[30]  Chi‐Huey Wong,et al.  In Vivo Protection Provided by a Synthetic New Alpha-Galactosyl Ceramide Analog against Bacterial and Viral Infections in Murine Models , 2010, Antimicrobial Agents and Chemotherapy.

[31]  J. Driver,et al.  Invariant Natural Killer T-Cell Control of Type 1 Diabetes: A Dendritic Cell Genetic Decision of a Silver Bullet or Russian Roulette , 2009, Diabetes.

[32]  W. Langdon,et al.  Mechanisms of NKT cell anergy induction involve Cbl-b-promoted monoubiquitination of CARMA1 , 2009, Proceedings of the National Academy of Sciences.

[33]  R. Rabadán,et al.  Reassortment Patterns in Swine Influenza Viruses , 2009, PloS one.

[34]  Anneliese O. Speak,et al.  Invariant NKT cells reduce the immunosuppressive activity of influenza A virus-induced myeloid-derived suppressor cells in mice and humans. , 2008, The Journal of clinical investigation.

[35]  N. Nagarajan,et al.  Cutting Edge: The Mechanism of Invariant NKT Cell Responses to Viral Danger Signals1 , 2008, The Journal of Immunology.

[36]  A. McMichael,et al.  Activation of invariant NKT cells enhances the innate immune response and improves the disease course in influenza A virus infection , 2008, European journal of immunology.

[37]  Vrajesh V. Parekh,et al.  Glycolipid antigen induces long-term natural killer T cell anergy in mice. , 2005, The Journal of clinical investigation.

[38]  R. Webby,et al.  Mutations in the NS1 Protein of Swine Influenza Virus Impair Anti-Interferon Activity and Confer Attenuation in Pigs , 2005, Journal of Virology.

[39]  M. Smyth,et al.  Antigen-induced tolerance by intrathymic modulation of self-recognizing inhibitory receptors , 2004, Nature Immunology.

[40]  Toshihiro Ito,et al.  Down-regulation of the invariant Valpha14 antigen receptor in NKT cells upon activation. , 2004, International immunology.

[41]  M. Taniguchi,et al.  The NKT cell system: bridging innate and acquired immunity , 2003, Nature Immunology.

[42]  M. Smyth,et al.  Glycolipid Antigen Drives Rapid Expansion and Sustained Cytokine Production by NK T Cells 1 , 2003, The Journal of Immunology.

[43]  Michael T. Wilson,et al.  The response of natural killer T cells to glycolipid antigens is characterized by surface receptor down-modulation and expansion , 2003, Proceedings of the National Academy of Sciences of the United States of America.

[44]  G. Giaccone,et al.  A phase I study of the natural killer T-cell ligand alpha-galactosylceramide (KRN7000) in patients with solid tumors. , 2002, Clinical cancer research : an official journal of the American Association for Cancer Research.

[45]  L. Reed,et al.  A SIMPLE METHOD OF ESTIMATING FIFTY PER CENT ENDPOINTS , 1938 .

[46]  L. Neumann Veterinary Immunology and Immunopathology , 2010 .

[47]  J. Lenstra,et al.  Functional CD1d and/or NKT cell invariant chain transcript in horse, pig, African elephant and guinea pig, but not in ruminants , 2009, Molecular immunology.

[48]  Uenza Manu WHO Manual on Animal Influenza Diagnosis and Surveillance , 2002 .