Expression of Jun, Fos and ATF-2 proteins in axotomized explanted and cultured adult rat dorsal root ganglia
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[1] S. Waxman,et al. Mechanisms of enhancement of neurite regeneration in vitro following a conditioning sciatic nerve lesion , 1998, The Journal of comparative neurology.
[2] P. Gass,et al. Expression of activating transcription factor-2, serum response factor and cAMP/Ca response element binding protein in the adult rat brain following generalized seizures, nerve fibre lesion and ultraviolet irradiation , 1997, Neuroscience.
[3] S. Dib-Hajj,et al. NGF has opposing effects on Na+ channel III and SNS gene expression in spinal sensory neurons , 1997, Neuroreport.
[4] T. Herdegen,et al. The c-Jun transcription factor – bipotential mediator of neuronal death, survival and regeneration , 1997, Trends in Neurosciences.
[5] X. Montalban,et al. Jun expression is found in neurons located in the vicinity of subacute plaques in patients with multiple sclerosis , 1996, Neuroscience Letters.
[6] R. Sidman,et al. Chondrodysplasia and neurological abnormalities in ATF-2-deficient mice , 1996, Nature.
[7] P. Gass,et al. Neuronal expression of AP-1 proteins in excitotoxic-neurodegenerative disorders and following nerve fiber lesions , 1995, Progress in Neurobiology.
[8] R. Nahin,et al. Comparison of c‐jun, junB, and junD mRNA expression and protein in the rat dorsal root ganglia following sciatic nerve transection , 1995, Journal of neuroscience research.
[9] C. Cotman,et al. Differential Induction of Immediate Early Gene Proteins in Cultured Neurons by β‐Amyloid (Aβ): Association of c‐Jun with Aβ‐Induced Apoptosis , 1995 .
[10] H. Stewart. Expression of c‐Jun, Jun B, Jun D and cAMP Response Element Binding Protein by Schwann Cells and their Precursors In Vivo and In Vitro , 1995, The European journal of neuroscience.
[11] L. Rubin,et al. A c-jun dominant negative mutant protects sympathetic neurons against programmed cell death , 1995, Neuron.
[12] M E Greenberg,et al. Calcium signaling in neurons: molecular mechanisms and cellular consequences. , 1995, Science.
[13] C. Sommer,et al. Selective c‐JUN Expression in CA1 Neurons of the Gerbil Hippocampus during and after Acquisition of an Ischemia‐Tolerant State , 1995, Brain pathology.
[14] G. Yancopoulos,et al. A BDNF autocrine loop in adult sensory neurons prevents cell death , 1995, Nature.
[15] S. Estus,et al. Altered gene expression in neurons during programmed cell death: identification of c-jun as necessary for neuronal apoptosis , 1994, The Journal of cell biology.
[16] M. Bähr,et al. GAP-43 immunoreactivity and axon regeneration in retinal ganglion cells of the rat. , 1994, Journal of neurobiology.
[17] C. de Felipe,et al. The differential control of c-jun expression in regenerating sensory neurons and their associated glial cells , 1994, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[18] M. Yaniv,et al. Mouse JunD negatively regulates fibroblast growth and antagonizes transformation by ras , 1994, Cell.
[19] B. Mayer,et al. Long-lasting expression of JUN and KROX transcription factors and nitric oxide synthase in intrinsic neurons of the rat brain following axotomy , 1993, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[20] B. Gold,et al. Regulation of the transcription factor c-JUN by nerve growth factor in adult sensory neurons , 1993, Neuroscience Letters.
[21] P. Herrlich,et al. Heterodimer formation of cJun and ATF‐2 is responsible for induction of c‐jun by the 243 amino acid adenovirus E1A protein. , 1993, The EMBO journal.
[22] R. Bravo,et al. Existence of different Fos/Jun complexes during the G0-to-G1 transition and during exponential growth in mouse fibroblasts: differential role of Fos proteins , 1992, Molecular and cellular biology.
[23] M. Zimmermann,et al. The transcription factors c-JUN, JUN D and CREB, but not FOS and KROX-24, are differentially regulated in axotomized neurons following transection of rat sciatic nerve. , 1992, Brain research. Molecular brain research.
[24] R. Bravo,et al. Selective expression of Jun proteins following axotomy and axonal transport block in peripheral nerves in the rat: evidence for a role in the regeneration process , 1991, Brain Research.
[25] R. Bravo,et al. Specific temporal and spatial distribution of JUN, FOS, and KROX‐24 proteins in spinal neurons following noxious transsynaptic stimulation , 1991, The Journal of comparative neurology.
[26] S. Hunt,et al. Long-term increase in the levels of c-jun mRNA and jun protein-like immunoreactivity in motor and sensory neurons following axon damage , 1991, Neuroscience Letters.
[27] R. Bravo,et al. Expression of different Jun and Fos proteins during the G0-to-G1 transition in mouse fibroblasts: in vitro and in vivo associations , 1991, Molecular and cellular biology.
[28] P. Roux,et al. Nuclear localization of c-Fos, but not v-Fos proteins, is controlled by extracellular signals , 1990, Cell.
[29] D. Bartel,et al. Growth factors and membrane depolarization activate distinct programs of early response gene expression: dissociation of fos and jun induction. , 1989, Genes & development.
[30] R. Lindsay,et al. Nerve growth factors (NGF, BDNF) enhance axonal regeneration but are not required for survival of adult sensory neurons , 1988, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[31] K. Schlingensiepen,et al. Complementary expression patterns of c-jun and jun B in rat brain and analysis of their function with antisense oligonucleotides , 1995 .
[32] W. Seifert,et al. Opposite functions of jun-B and c-jun in growth regulation and neuronal differentiation. , 1993, Developmental genetics.
[33] T. Curran,et al. Stimulus-transcription coupling in the nervous system: involvement of the inducible proto-oncogenes fos and jun. , 1991, Annual review of neuroscience.
[34] H. Herschman. Primary response genes induced by growth factors and tumor promoters. , 1991, Annual review of biochemistry.