Characterization of defective nucleotide excision repair in XPC mutant mice.

[1]  R. Hammer,et al.  Synergistic interactions between XPC and p53 mutations in double-mutant mice: neural tube abnormalities and accelerated UV radiation-induced skin cancer , 1996, Current Biology.

[2]  T. Ishikawa,et al.  High incidence of ultraviolet-B-or chemical-carcinogen-induced skin tumours in mice lacking the xeroderma pigmentosum group A gene , 1995, Nature.

[3]  F. Gruijl,et al.  Increased susceptibility to ultraviolet-B and carcinogens of mice lacking the DNA excision repair gene XPA , 1995, Nature.

[4]  A. Bradley,et al.  High susceptibility to ultraviolet-induced carcinogenesis in mice lacking XPC , 1995, Nature.

[5]  L. Mullenders,et al.  Transcription‐coupled repair removes both cyclobutane pyrimidine dimers and 6‐4 photoproducts with equal efficiency and in a sequential way from transcribed DNA in xeroderma pigmentosum group C fibroblasts. , 1995, The EMBO journal.

[6]  L. Mullenders,et al.  Strand-specific removal of cyclobutane pyrimidine dimers from the p53 gene in the epidermis of UVB-irradiated hairless mice. , 1994, Oncogene.

[7]  K. Tanaka,et al.  Purification and cloning of a nucleotide excision repair complex involving the xeroderma pigmentosum group C protein and a human homologue of yeast RAD23. , 1994, The EMBO journal.

[8]  J. Mcwhir,et al.  Mice with DNA repair gene (ERCC-1) deficiency have elevated levels of p53, liver nuclear abnormalities and die before weaning , 1993, Nature Genetics.

[9]  L. Mullenders,et al.  Xeroderma pigmentosum complementation group C cells remove pyrimidine dimers selectively from the transcribed strand of active genes , 1991, Molecular and cellular biology.

[10]  E. Jung,et al.  Clinical symptoms and DNA repair characteristics of xeroderma pigmentosum patients from Germany. , 1991, Cancer research.

[11]  K. Kraemer,et al.  Xeroderma Pigmentosum: Cutaneous, Ocular, and Neurologic Abnormalities in 830 Published Cases , 1987 .

[12]  O. Nikaido,et al.  Excision repair of mouse and human fibroblast cells, and a factor affecting the amount of UV-induced unscheduled DNA synthesis. , 1984, Mutation research.

[13]  D. Kaden,et al.  Mutation of xeroderma pigmentosum lymphoblasts by far-ultraviolet light. , 1984, Mutation research.

[14]  K. Kraemer,et al.  DNA repair protects against cutaneous and internal neoplasia: evidence from xeroderma pigmentosum. , 1984, Carcinogenesis.

[15]  J. Mccormick,et al.  XERODERMA PIGMENTOSUM FIBROBLASTS INCLUDING CELLS FROM XP VARIANTS ARE ABNORMALLY SENSITIVE TO THE MUTAGENIC AND CYTOTOXIC ACTION OF BROAD SPECTRUM SIMULATED SUNLIGHT , 1984, Photochemistry and photobiology.

[16]  T. Yagi DNA repair ability of cultured cells derived from mouse embryos in comparison with human cells. , 1982, Mutation research.

[17]  Charles R.scriver The Metabolic basis of inherited disease , 1989 .