And finally, genes for human obesity

[1]  S. O’Rahilly,et al.  Obesity and impaired prohormone processing associated with mutations in the human prohormone convertase 1 gene , 1997, Nature Genetics.

[2]  M. Nicolson,et al.  Phenotype of the Obese Koletsky (f) Rat Due to Tyr763Stop Mutation in the Extracellular Domain of the Leptin Receptor (Lepr): Evidence for Deficient Plasma-to-CSF Transport of Leptin in Both the Zucker and Koletsky Obese Rat , 1997, Diabetes.

[3]  L. Pannell,et al.  Carboxypeptidase E Is a Regulated Secretory Pathway Sorting Receptor: Genetic Obliteration Leads to Endocrine Disorders in Cpefat Mice , 1997, Cell.

[4]  K. Nakao,et al.  Nonsense mutation of leptin receptor in the obese spontaneously hypertensive Koletsky rat , 1996, Nature Genetics.

[5]  W. Chung,et al.  Quantitative variation in obesity-related traits and insulin precursors linked to the OB gene region on human chromosome 7. , 1996, American journal of human genetics.

[6]  T. Murakami,et al.  Phenotype-linked amino acid alteration in leptin receptor cDNA from Zucker fatty (fa/fa) rat. , 1996, Biochemical and biophysical research communications.

[7]  E. Ravussin,et al.  Absence of Mutations in the Human OB Gene in Obese/Diabetic Subjects , 1996, Diabetes.

[8]  M. Phillips,et al.  Leptin receptor missense mutation in the fatty Zucker rat , 1996, Nature Genetics.

[9]  E. Green,et al.  Extreme Obesity May Be Linked to Markers Flanking the Human OB Gene , 1996, Diabetes.

[10]  C. Ebeling,et al.  Identification and Characterization of the Mouse Obesity Gene tubby: A Member of a Novel Gene Family , 1996, Cell.

[11]  M. North,et al.  A candidate gene for the mouse mutation tubby , 1996, Nature.

[12]  L. Tartaglia,et al.  Phenotypes of Mouse diabetes and Rat fatty Due to Mutations in the OB (Leptin) Receptor , 1996, Science.

[13]  Rene Devos,et al.  Identification and expression cloning of a leptin receptor, OB-R , 1995, Cell.

[14]  S. O’Rahilly,et al.  Brief report: impaired processing of prohormones associated with abnormalities of glucose homeostasis and adrenal function. , 1995, The New England journal of medicine.

[15]  N. Seidah,et al.  Expression of candidate pro-GnRH processing enzymes in rat hypothalamus and an immortalized hypothalamic neuronal cell line. , 1995, Neuroendocrinology.

[16]  J. Naggert,et al.  Hyperproinsulinaemia in obese fat/fat mice associated with a carboxypeptidase E mutation which reduces enzyme activity , 1995, Nature Genetics.

[17]  R. Considine,et al.  Evidence against either a premature stop codon or the absence of obese gene mRNA in human obesity. , 1995, The Journal of clinical investigation.

[18]  M. Maffei,et al.  Positional cloning of the mouse obese gene and its human homologue , 1994, Nature.

[19]  L. Pérusse,et al.  Genetics of obesity. , 1993, Annual review of nutrition.

[20]  R. Woychik,et al.  Molecular characterization of the mouse agouti locus , 1992, Cell.

[21]  H. Davidson,et al.  Proteolytic conversion of proinsulin into insulin. Identification of a Ca2+-dependent acidic endopeptidase in isolated insulin-secretory granules. , 1987, The Biochemical journal.

[22]  G. Bray,et al.  Hypothalamic and genetic obesity in experimental animals: an autonomic and endocrine hypothesis. , 1979, Physiological reviews.

[23]  D. Coleman Obesity genes: beneficial effects in heterozygous mice. , 1979, Science.

[24]  L. J. Cole,et al.  BLACKHEAD, A COCCIDIAL DISEASE OF TURKEYS. , 1908, Science.