Ubiquitin C-Terminal Hydrolase Is an Immediate-Early Gene Essential for Long-Term Facilitation in Aplysia
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James H. Schwartz | Eric R. Kandel | Kelsey C. Martin | Kaoru Inokuchi | E. Kandel | J. H. Schwartz | K. Martin | M. Ghirardi | A. Casadio | K. Inokuchi | Daniel G. Chain | Ashok N. Hegde | Andrea Casadio | Wanzheng Pei | Mirella Ghirardi | D. Chain | A. Hegde | Wanzheng Pei
[1] E. Kandel,et al. C/EBP is an immediate-early gene required for the consolidation of long-term facilitation in Aplysia , 1994, Cell.
[2] E R Kandel,et al. Synaptic facilitation and behavioral sensitization in Aplysia: possible role of serotonin and cyclic AMP. , 1976, Science.
[3] W N Frost,et al. Monosynaptic connections made by the sensory neurons of the gill- and siphon-withdrawal reflex in Aplysia participate in the storage of long-term memory for sensitization. , 1985, Proceedings of the National Academy of Sciences of the United States of America.
[4] E. Kandel,et al. Roles of PKA and PKC in facilitation of evoked and spontaneous transmitter release at depressed and nondepressed synapses in aplysia sensory neurons , 1992, Neuron.
[5] Philip Goelet,et al. The long and the short of long–term memory—a molecular framework , 1986, Nature.
[6] E R Kandel,et al. Serotonin-mediated endocytosis of apCAM: an early step of learning-related synaptic growth in Aplysia. , 1992, Science.
[7] E. Kandel,et al. Persistent and transcriptionally-dependent increase in protein phosphorylation in long-term facilitation ofAplysia sensory neurons , 1989, Nature.
[8] K. Tanaka,et al. Improved method for preparation of ubiquitin‐ligated lysozyme as substrate of ATP‐dependent proteolysis , 1991, FEBS letters.
[9] E. Kandel,et al. cAMP evokes long-term facilitation in Aplysia sensory neurons that requires new protein synthesis. , 1988, Science.
[10] J. Tobias,et al. Ubiquitin-specific proteases of Saccharomyces cerevisiae. Cloning of UBP2 and UBP3, and functional analysis of the UBP gene family. , 1992, The Journal of biological chemistry.
[11] A. Goldberg,et al. Inhibitors of the proteasome block the degradation of most cell proteins and the generation of peptides presented on MHC class I molecules , 1994, Cell.
[12] Eric R. Kandel,et al. Injection of the cAMP-responsive element into the nucleus of Aplysia sensory neurons blocks long-term facilitation , 1990, Nature.
[13] F. R. Papa,et al. The yeast DOA4 gene encodes a deubiquitinating enzyme related to a product of the human tre-2 oncogene , 1993, Nature.
[14] Mary Chen,et al. Aplysia CREB2 represses long-term facilitation: Relief of repression converts transient facilitation into long-term functional and structural change , 1995, Cell.
[15] S. Schacher,et al. Neurite regeneration by Aplysia neurons in dissociated cell culture: modulation by Aplysia hemolymph and the presence of the initial axonal segment , 1983, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[16] E R Kandel,et al. A critical period for macromolecular synthesis in long-term heterosynaptic facilitation in Aplysia. , 1986, Science.
[17] S. Benzer,et al. bendless, a Drosophila gene affecting neuronal connectivity, encodes a ubiquitin-conjugating enzyme homolog , 1994, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[18] E. Kandel,et al. Serotonin produces long-term changes in the excitability of Aplysia sensory neurons in culture that depend on new protein synthesis , 1987, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[19] R. Baker,et al. Control of Cell Fate by a Deubiquitinating Enzyme Encoded by the fat facets Gene , 1995, Science.
[20] K. Wilkinson. Roles of ubiquitinylation in proteolysis and cellular regulation. , 1995, Annual review of nutrition.
[21] E. Kandel,et al. Serotonin and cyclic AMP close single K+ channels in Aplysia sensory neurones , 1982, Nature.
[22] H. Schmid,et al. The prosomal protein of 27 kDa and a nuclear 38 kDa protein are immunologically related , 1990, FEBS letters.
[23] E. Kandel,et al. Protein synthesis during acquisition of long-term facilitation is needed for the persistent loss of regulatory subunits of the Aplysia cAMP-dependent protein kinase. , 1990, Proceedings of the National Academy of Sciences of the United States of America.
[24] C. Larsen,et al. Substrate binding and catalysis by ubiquitin C-terminal hydrolases: identification of two active site residues. , 1996, Biochemistry.
[25] Marc W. Kirschner,et al. How Proteolysis Drives the Cell Cycle , 1996, Science.
[26] E R Kandel,et al. Presynaptic facilitation revisited: state and time dependence , 1996, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[27] E. Kandel,et al. Structural changes accompanying memory storage. , 1993, Annual review of physiology.
[28] E. Kandel,et al. Facilitatory transmitter causes a selective and prolonged increase in adenosine 3':5'-monophosphate in sensory neurons mediating the gill and siphon withdrawal reflex in Aplysia , 1982, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[29] C. Larsen,et al. Metabolism of the polyubiquitin degradation signal: structure, mechanism, and role of isopeptidase T. , 1995, Biochemistry.
[30] K D Wilkinson,et al. The neuron-specific protein PGP 9.5 is a ubiquitin carboxyl-terminal hydrolase. , 1989, Science.
[31] J. Callis,et al. Utility of polyhistidine-tagged ubiquitin in the purification of ubiquitin-protein conjugates and as an affinity ligand for the purification of ubiquitin-specific hydrolases. , 1993, The Journal of biological chemistry.
[32] R. Hough,et al. Ubiquitin-lysozyme conjugates. Purification and susceptibility to proteolysis. , 1986, The Journal of biological chemistry.
[33] E. Kandel,et al. 5-HT modulates protein synthesis and the expression of specific proteins during long-term facilitation in aplysia sensory neurons , 1989, Neuron.
[34] A. Ciechanover,et al. Persistent activation of cAMP-dependent protein kinase by regulated proteolysis suggests a neuron-specific function of the ubiquitin system in Aplysia , 1995, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[35] E R Kandel,et al. Functioning of identified neurons and synapses in abdominal ganglion of Aplysia in absence of protein synthesis. , 1971, Journal of neurophysiology.
[36] J. H. Schwartz,et al. Regulatory subunits of cAMP-dependent protein kinases are degraded after conjugation to ubiquitin: a molecular mechanism underlying long-term synaptic plasticity. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[37] E M Schwarz,et al. Rel/NF-kappa B/I kappa B family: intimate tales of association and dissociation. , 1995, Genes & development.
[38] K Tanaka,et al. Structure and functions of the 20S and 26S proteasomes. , 1996, Annual review of biochemistry.
[39] James H. Schwartz,et al. A molecular mechanism for long-term sensitization in Aplysia , 1987, Nature.
[40] A. Hershko,et al. A ubiquitin C-terminal isopeptidase that acts on polyubiquitin chains. Role in protein degradation. , 1992, The Journal of biological chemistry.
[41] A. Hershko,et al. Ubiquitin C-terminal hydrolase activity associated with the 26 S protease complex. , 1993, The Journal of biological chemistry.
[42] E. Kandel,et al. Activation of cAMP-Responsive genes by stimuli that produce long-term facilitation in aplysia sensory neurons , 1993, Neuron.
[43] Eric R Kandel,et al. A novel intermediate stage in the transition between short- and long-term facilitation in the sensory to motor neuron synapse of aplysia , 1995, Neuron.
[44] E. Kandel,et al. Long-term heterosynaptic inhibition in Aplysia , 1988, Nature.
[45] J. Byrne,et al. Intracellular injection of cAMP induces a long-term reduction of neuronal K+ currents. , 1988, Science.
[46] T. Préat,et al. Genetic dissection of consolidated memory in Drosophila , 1994, Cell.
[47] W. V. Van Nostrand,et al. Amyloid β-Protein Inhibits Ubiquitin-dependent Protein Degradation in Vitro(*) , 1995, The Journal of Biological Chemistry.
[48] Alcino J. Silva,et al. Deficient long-term memory in mice with a targeted mutation of the cAMP-responsive element-binding protein , 1994, Cell.
[49] M. Carroll,et al. DUB-1, a deubiquitinating enzyme with growth-suppressing activity. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[50] J. Ridgway,et al. Cloning and Expression of a Yeast Ubiquitin-Protein Cleaving Activity in Escherichia Coli , 1989, Bio/Technology.
[51] John B. Thomas,et al. The Drosophila bendless gene encodes a neural protein related to ubiquitin-conjugating enzymes , 1993, Neuron.
[52] A. Goldberg,et al. Red blood cells contain a pathway for the degradation of oxidant-damaged hemoglobin that does not require ATP or ubiquitin. , 1986, The Journal of biological chemistry.
[53] E. Kandel,et al. Molecular biology of learning: modulation of transmitter release. , 1982, Science.
[54] T. Carew,et al. Molecular Enhancement of Memory Formation , 1996, Neuron.