Corrigendum: NLRP10 is a NOD-like receptor essential to initiate adaptive immunity by dendritic cells
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E. Elinav | F. Sutterwala | Hailong Meng | R. Flavell | S. Kleinstein | A. Rongvaux | J. Henao-Mejia | Adam Williams | S. Eisenbarth | T. Strowig | S. Joly | C. Thaiss | Lan Xu | L. Zenewicz | Christoph A. Thaiss | O. Colegio | A. Haberman | David G. Gonzalez | Lauren A. Zenewicz | Jorge Henao-Mejia
[1] G. Superti-Furga,et al. Coincidental loss of DOCK8 function in NLRP10-deficient and C3H/HeJ mice results in defective dendritic cell migration , 2015, Proceedings of the National Academy of Sciences.
[2] D. Green,et al. Addendum: Defective Dock2 expression in a subset of ASC-deficient mouse lines , 2012, Nature Immunology.
[3] S. Yokoyama,et al. DOCK8 is a Cdc42 activator critical for interstitial dendritic cell migration during immune responses. , 2012, Blood.
[4] Deepta Bhattacharya,et al. Spontaneous mutation of the Dock2 gene in Irf5−/− mice complicates interpretation of type I interferon production and antibody responses , 2012, Proceedings of the National Academy of Sciences.
[5] D. Green,et al. The inflammasome adaptor ASC regulates adaptive immune cell functions by controlling DOCK2-mediated Rac activation and actin polymerization , 2011, Nature Immunology.
[6] J. Stockman,et al. Combined Immunodeficiency Associated with DOCK8 Mutations , 2011 .
[7] S. Holland,et al. Combined immunodeficiency associated with DOCK8 mutations. , 2009, The New England journal of medicine.
[8] M. Wiles,et al. Know thy mouse. , 2006, Trends in genetics : TIG.