Hotspot Mutation Panel Testing Reveals Clonal Evolution in a Study of 265 Paired Primary and Metastatic Tumors
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F. Meric-Bernstam | A. Lazar | R. Broaddus | R. Goswami | M. Routbort | K. Patel | L. Medeiros | R. Luthra | S. Roy-Chowdhuri | V. Subbiah | Rajesh R. Singh | E. S. Kopetz | R. Alvarez | K. Jabbar | M. Davies | Rashmi S. Goswami | E. Kopetz | L. J. Medeiros | Rajesh R. Singh | Alexander J. Lazar | Rashmi S. Goswami | Ricardo H. Alvarez | Michael A. Davies | Russell Broaddus
[1] G. Cathomas. PIK3CA in Colorectal Cancer , 2014, Front. Oncol..
[2] D. Kerr,et al. Evaluation of PIK3CA mutation as a predictor of benefit from nonsteroidal anti-inflammatory drug therapy in colorectal cancer. , 2013, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[3] Benjamin J. Raphael,et al. Advances for studying clonal evolution in cancer. , 2013, Cancer letters.
[4] Rashmi Kanagal-Shamanna,et al. Clinical validation of a next-generation sequencing screen for mutational hotspots in 46 cancer-related genes. , 2013, The Journal of molecular diagnostics : JMD.
[5] S. Jeffrey,et al. Colorectal cancer diagnostics: biomarkers, cell-free DNA, circulating tumor cells and defining heterogeneous populations by single-cell analysis , 2013, Expert review of molecular diagnostics.
[6] Elisabeth Brambilla,et al. Next-generation sequencing reveals high concordance of recurrent somatic alterations between primary tumor and metastases from patients with non-small-cell lung cancer. , 2013, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[7] K. Vousden,et al. p53 mutations in cancer , 2013, Nature Cell Biology.
[8] F. André,et al. Discrepancies between primary tumor and metastasis: a literature review on clinically established biomarkers. , 2012, Critical reviews in oncology/hematology.
[9] Reiko Nishihara,et al. Aspirin use, tumor PIK3CA mutation, and colorectal-cancer survival. , 2012, The New England journal of medicine.
[10] A. Viale,et al. Comparative genomic analysis of primary versus metastatic colorectal carcinomas. , 2012, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[11] Irmtraud M. Meyer,et al. The clonal and mutational evolution spectrum of primary triple-negative breast cancers , 2012, Nature.
[12] Christine A Iacobuzio-Donahue,et al. A new branch on the tree: next-generation sequencing in the study of cancer evolution. , 2012, Seminars in cell & developmental biology.
[13] Rebecca A Betensky,et al. Mosaic amplification of multiple receptor tyrosine kinase genes in glioblastoma. , 2011, Cancer cell.
[14] Bernard P. Puc,et al. An integrated semiconductor device enabling non-optical genome sequencing , 2011, Nature.
[15] M. Nowak,et al. Distant Metastasis Occurs Late during the Genetic Evolution of Pancreatic Cancer , 2010, Nature.
[16] M. Ringnér,et al. Multiple metastases from cutaneous malignant melanoma patients may display heterogeneous genomic and epigenomic patterns , 2010, Melanoma research.
[17] Yong-jig Cho,et al. Antimetastatic role of Smad4 signaling in colorectal cancer. , 2010, Gastroenterology.
[18] R. Lothe,et al. Tumor Spreading to the Contralateral Ovary in Bilateral Ovarian Carcinoma Is a Late Event in Clonal Evolution , 2009, Journal of oncology.
[19] Massimo Libra,et al. PIK3CA mutations in human solid tumors: Role in sensitivity to various therapeutic approaches , 2009, Cell cycle.
[20] Martin A. Nowak,et al. Comparative lesion sequencing provides insights into tumor evolution , 2008, Proceedings of the National Academy of Sciences.
[21] Liang Cheng,et al. Clonal origin of lymph node metastases in bladder carcinoma , 2005, Cancer.
[22] Chi-Hung Lin,et al. Genetic changes and clonality relationship between primary colorectal cancers and their pulmonary metastases—An analysis by comparative genomic hybridization , 2005, Genes, chromosomes & cancer.
[23] Lorena Losi,et al. Evolution of intratumoral genetic heterogeneity during colorectal cancer progression. , 2005, Carcinogenesis.
[24] Weichung J Shih,et al. Loss of Smad Signaling in Human Colorectal Cancer Is Associated with Advanced Disease and Poor Prognosis , 2003, Cancer journal.
[25] R. Eils,et al. From latent disseminated cells to overt metastasis: Genetic analysis of systemic breast cancer progression , 2003, Proceedings of the National Academy of Sciences of the United States of America.
[26] G. Capellá,et al. Genetic Evolution in the Metastatic Progression of Human Pancreatic Cancer Studied by CGH , 2001, Laboratory Investigation.
[27] B. Vogelstein,et al. A genetic model for colorectal tumorigenesis , 1990, Cell.
[28] G Poste,et al. Evolution of tumor cell heterogeneity during progressive growth of individual lung metastases. , 1982, Proceedings of the National Academy of Sciences of the United States of America.
[29] J E Talmadge,et al. Evidence for the clonal origin of spontaneous metastases. , 1982, Science.
[30] P. Nowell. The clonal evolution of tumor cell populations. , 1976, Science.
[31] R. Arceci. Intratumor Heterogeneity and Branched Evolution Revealed by Multiregion Sequencing , 2012 .