Endoplasmic Reticulum Selective Autophagy Alleviates Anthracycline-Induced Cardiotoxicity

[1]  P. M. Hwang,et al.  ATVB IN FOCUS: Cardiovascular Effects of Anticancer Treatments/Agents , 2021 .

[2]  Jianping Liu,et al.  Phosphorylation regulates the binding of autophagy receptors to FIP200 Claw domain for selective autophagy initiation , 2021, Nature Communications.

[3]  I. Perrotta.  ER-phagy in human atherosclerosis: an exploratory ultrastructural study , 2020, Ultrastructural pathology.

[4]  Yanbin Dong,et al.  Globular adiponectin alleviates chronic intermittent hypoxia-induced H9C2 cardiomyocytes apoptosis via ER-phagy induction , 2020, Cell cycle.

[5]  J. Olzmann,et al.  A Genome-wide ER-phagy Screen Highlights Key Roles of Mitochondrial Metabolism and ER-Resident UFMylation , 2020, Cell.

[6]  Y. Maejima.  The critical roles of protein quality control systems in the pathogenesis of heart failure. , 2019, Journal of cardiology.

[7]  D. Klionsky,et al.  TEX264 is a major receptor for mammalian reticulophagy , 2019, Autophagy.

[8]  I. Tetko,et al.  Prokineticin Receptor-1 Signaling Inhibits Dose- and Time-Dependent Anthracycline-Induced Cardiovascular Toxicity Via Myocardial and Vascular Protection , 2019, JACC. CardioOncology.

[9]  T. Natsume,et al.  Intrinsically Disordered Protein TEX264 Mediates ER-phagy. , 2019, Molecular cell.

[10]  Vladimir Denic,et al.  TEX264 Is an Endoplasmic Reticulum-Resident ATG8-Interacting Protein Critical for ER Remodeling during Nutrient Stress. , 2019, Molecular cell.

[11]  J. Teng,et al.  ATL3 Is a Tubular ER-Phagy Receptor for GABARAP-Mediated Selective Autophagy , 2019, Current Biology.

[12]  D. Klionsky,et al.  Cargo recognition and degradation by selective autophagy , 2018, Nature Cell Biology.

[13]  Matthew D. Smith,et al.  CCPG1 Is a Non-canonical Autophagy Cargo Receptor Essential for ER-Phagy and Pancreatic ER Proteostasis , 2017, Developmental cell.

[14]  N. Mizushima,et al.  An Autophagic Flux Probe that Releases an Internal Control. , 2016, Molecular cell.

[15]  M. Peter,et al.  Translocon component Sec62 acts in endoplasmic reticulum turnover during stress recovery , 2016, Nature Cell Biology.

[16]  B. A. French,et al.  Chemical Endoplasmic Reticulum Chaperone Alleviates Doxorubicin-Induced Cardiac Dysfunction. , 2016, Circulation research.

[17]  Randal J. Kaufman,et al.  Protein misfolding in the endoplasmic reticulum as a conduit to human disease , 2016, Nature.

[18]  I. Katona,et al.  Regulation of endoplasmic reticulum turnover by selective autophagy , 2015, Nature.

[19]  S. Kaneko,et al.  Muscle-Specific RING Finger 1 Negatively Regulates Pathological Cardiac Hypertrophy Through Downregulation of Calcineurin A , 2014, Circulation. Heart failure.

[20]  M. Isobe,et al.  Mst1 inhibits autophagy by promoting Beclin1-Bcl-2 interaction , 2013, Nature medicine.

[21]  Xiaoxue Zhang,et al.  Parkin Protein Deficiency Exacerbates Cardiac Injury and Reduces Survival following Myocardial Infarction*♦ , 2012, The Journal of Biological Chemistry.

[22]  G. Barber,et al.  Endoplasmic Reticulum Stress Regulates the Innate Immunity Critical Transcription Factor IRF3 , 2012, The Journal of Immunology.

[23]  Masaaki Komatsu,et al.  Autophagy: Renovation of Cells and Tissues , 2011, Cell.

[24]  D. Glass,et al.  Endogenous Muscle Atrophy F-Box Mediates Pressure Overload–Induced Cardiac Hypertrophy Through Regulation of Nuclear Factor-&kgr;B , 2011, Circulation research.

[25]  K. Hirao,et al.  Induction of premature senescence in cardiomyocytes by doxorubicin as a novel mechanism of myocardial damage , 2008, Aging cell.

[26]  Yasushi Matsumura,et al.  The role of autophagy in cardiomyocytes in the basal state and in response to hemodynamic stress , 2007, Nature Medicine.

[27]  Peter Walter,et al.  Autophagy Counterbalances Endoplasmic Reticulum Expansion during the Unfolded Protein Response , 2006, PLoS biology.

[28]  T. Nishino,et al.  Dilated Cardiomyopathy Caused by Aberrant Endoplasmic Reticulum Quality Control in Mutant KDEL Receptor Transgenic Mice , 2004, Molecular and Cellular Biology.

[29]  M. Hori,et al.  Prolonged Endoplasmic Reticulum Stress in Hypertrophic and Failing Heart After Aortic Constriction: Possible Contribution of Endoplasmic Reticulum Stress to Cardiac Myocyte Apoptosis , 2004, Circulation.

[30]  T. Maniatis,et al.  IKKε and TBK1 are essential components of the IRF3 signaling pathway , 2003, Nature Immunology.

[31]  G. Schultz,et al.  Morphological, biochemical, and electrophysiological characterization of a clonal cell (H9c2) line from rat heart. , 1991, Circulation research.

[32]  M. Isobe,et al.  Nitric oxide inhibits myocardial apoptosis by preventing caspase-3 activity via S-nitrosylation. , 2005, Journal of molecular and cellular cardiology.