Induction of cardiac dysfunction in developing and adult zebrafish by chronic isoproterenol stimulation.
暂无分享,去创建一个
A. Benz | P. Most | H. Katus | D. Hassel | M. Siragusa | L. Jürgensen | S. Hein | M. Kossack | L. Juergensen | Selina Hein
[1] R. Fink,et al. Essential light chain S195 phosphorylation is required for cardiac adaptation under physical stress. , 2016, Cardiovascular research.
[2] A. Jaźwińska,et al. Acute stress is detrimental to heart regeneration in zebrafish , 2016, Open Biology.
[3] H. Katus,et al. Advanced Echocardiography in Adult Zebrafish Reveals Delayed Recovery of Heart Function after Myocardial Cryoinjury , 2015, PloS one.
[4] M. Caron,et al. Overlapping and Opposing Functions of G Protein-coupled Receptor Kinase 2 (GRK2) and GRK5 during Heart Development* , 2014, The Journal of Biological Chemistry.
[5] S. Royce,et al. Serelaxin Is a More Efficacious Antifibrotic Than Enalapril in an Experimental Model of Heart Disease , 2014, Hypertension.
[6] R. Peterson,et al. The zebrafish as a tool to identify novel therapies for human cardiovascular disease , 2014, Disease Models & Mechanisms.
[7] H. Calkins,et al. Identification of a New Modulator of the Intercalated Disc in a Zebrafish Model of Arrhythmogenic Cardiomyopathy , 2014, Science Translational Medicine.
[8] E. Stanley,et al. CSF-1 receptor signaling in myeloid cells. , 2014, Cold Spring Harbor perspectives in biology.
[9] A. Vickers,et al. Isoproterenol effects evaluated in heart slices of human and rat in comparison to rat heart in vivo. , 2014, Toxicology and applied pharmacology.
[10] Leonard I Zon,et al. Of fish and men: using zebrafish to fight human diseases. , 2013, Trends in cell biology.
[11] M. Luo,et al. Mechanisms of Altered Ca2+ Handling in Heart Failure , 2013, Circulation research.
[12] P. Song,et al. Isoproterenol instigates cardiomyocyte apoptosis and heart failure via AMPK inactivation-mediated endoplasmic reticulum stress , 2013, Apoptosis.
[13] N. Frangogiannis,et al. Fibroblasts in post-infarction inflammation and cardiac repair. , 2013, Biochimica et biophysica acta.
[14] K. Poon,et al. The zebrafish model system in cardiovascular research: A tiny fish with mighty prospects , 2013, Global cardiology science & practice.
[15] J. C. Belmonte,et al. Isolation and in vitro culture of primary cardiomyocytes from adult zebrafish hearts , 2013, Nature Protocols.
[16] J. Molkentin,et al. Signaling effectors underlying pathologic growth and remodeling of the heart. , 2013, The Journal of clinical investigation.
[17] Shuo Lin,et al. Reverse genetic approaches in zebrafish. , 2012, Journal of genetics and genomics = Yi chuan xue bao.
[18] L. Zon,et al. Small molecule screening in zebrafish: swimming in potential drug therapies , 2012, Wiley interdisciplinary reviews. Developmental biology.
[19] F. Pixley,et al. CSF-1 signaling in macrophages: pleiotrophy through phosphotyrosine-based signaling pathways , 2012, Critical reviews in clinical laboratory sciences.
[20] A. Werdich,et al. The regenerative capacity of zebrafish reverses cardiac failure caused by genetic cardiomyocyte depletion , 2011, Development.
[21] B. Blaxall,et al. G Protein Coupled Receptor Kinases as Therapeutic Targets in Cardiovascular Disease , 2011, Circulation research.
[22] Jeroen Bakkers,et al. Zebrafish as a model to study cardiac development and human cardiac disease , 2011, Cardiovascular research.
[23] N. Mercader,et al. Extensive scar formation and regression during heart regeneration after cryoinjury in zebrafish , 2011, Development.
[24] S. Kjeldsen,et al. The role of beta-blockers in the treatment of chronic heart failure. , 2011, Trends in pharmacological sciences.
[25] W. Koch,et al. βARKct: A Therapeutic Approach for Improved Adrenergic Signaling and Function in Heart Disease , 2010, Journal of cardiovascular translational research.
[26] F. Zannad,et al. Extracellular matrix fibrotic markers in heart failure , 2010, Heart Failure Reviews.
[27] Y. Nishimura,et al. Zebrafish beta-adrenergic receptor mRNA expression and control of pigmentation. , 2009, Gene.
[28] C. H. Conrad,et al. Isoproterenol-induced myocardial injury and diastolic dysfunction in mice: structural and functional correlates. , 2009, Comparative medicine.
[29] Lan Ma,et al. Kinase activity-independent regulation of cyclin pathway by GRK2 is essential for zebrafish early development , 2009, Proceedings of the National Academy of Sciences.
[30] K. Poss. Getting to the heart of regeneration in zebrafish. , 2007, Seminars in cell & developmental biology.
[31] K. Urasawa,et al. Chronic beta-adrenergic receptor stimulation enhances the expression of G-Protein coupled receptor kinases, GRK2 and GRK5, in both the heart and peripheral lymphocytes. , 2005, Circulation journal : official journal of the Japanese Circulation Society.
[32] G. Torre-Amione. Immune activation in chronic heart failure. , 2005, The American journal of cardiology.
[33] G. Jaffe,et al. Effect of NF-κB inhibition on TNF-α-induced apoptosis in human RPE cells , 2004 .
[34] Martin J. Lohse,et al. What Is the Role of &bgr;-Adrenergic Signaling in Heart Failure? , 2003, Circulation research.
[35] M. Keating,et al. Heart Regeneration in Zebrafish , 2002, Science.
[36] R. Atkins,et al. Macrophage accumulation at a site of renal inflammation is dependent on the M‐CSF/c‐fms pathway , 2002, Journal of leukocyte biology.
[37] O. Mitrasinovic,et al. Overexpression of Macrophage Colony-stimulating Factor Receptor on Microglial Cells Induces an Inflammatory Response* , 2001, The Journal of Biological Chemistry.
[38] S. Vatner,et al. β-adrenergic cardiac hypertrophy is mediated primarily by the β1-subtype in the rat heart , 2001 .
[39] B. Chandrasekar,et al. Chronic beta-adrenergic stimulation induces myocardial proinflammatory cytokine expression. , 2000, Circulation.
[40] A. Borczuk,et al. β-Adrenergic stimulation causes cardiocyte apoptosis: influence of tachycardia and hypertrophy. , 1998, American journal of physiology. Heart and circulatory physiology.
[41] C. Nüsslein-Volhard,et al. Large scale genetics in a small vertebrate, the zebrafish. , 1996, The International journal of developmental biology.
[42] E. Lakatta,et al. Isoproterenol infusion induces alterations in expression of hypertrophy-associated genes in rat heart. , 1995, The American journal of physiology.
[43] M. Böhm,et al. Expression of beta-arrestins and beta-adrenergic receptor kinases in the failing human heart. , 1994, Circulation research.
[44] M. Böhm,et al. Altered expression of beta-adrenergic receptor kinase and beta 1-adrenergic receptors in the failing human heart. , 1993, Circulation.
[45] I. Benjamin,et al. Isoproterenol-Induced Myocardial Fibrosis in Relation to Myocyte Necrosis , 1989, Circulation research.
[46] C. Malbon,et al. Down-regulation of beta-adrenergic receptors: agonist-induced reduction in receptor mRNA levels. , 1988, Proceedings of the National Academy of Sciences of the United States of America.
[47] D C Harrison,et al. Decreased catecholamine sensitivity and beta-adrenergic-receptor density in failing human hearts. , 1982, The New England journal of medicine.
[48] M. Ekker,et al. Interactive effects of development and hypoxia on catecholamine synthesis and cardiac function in zebrafish (Danio rerio) , 2010, Journal of Comparative Physiology B.
[49] O. Brodde. β-Adrenergic receptors in failing human myocardium , 2004, Basic Research in Cardiology.
[50] M. Entman,et al. The inflammatory response in myocardial infarction. , 2002, Cardiovascular research.
[51] H. Schunkert,et al. Development of heart failure following isoproterenol administration in the rat: role of the renin-angiotensin system. , 1998, Cardiovascular research.
[52] M. Böhm,et al. Analysis of beta-adrenergic receptor mRNA levels in human ventricular biopsy specimens by quantitative polymerase chain reactions: progressive reduction of beta 1-adrenergic receptor mRNA in heart failure. , 1996, Journal of the American College of Cardiology.
[53] J. Szabó,et al. Experimental cardiac hypertrophy induced by isoproterenol in the rat. , 1975, Acta physiologica Academiae Scientiarum Hungaricae.