Alleviating effects and mechanisms of action of large-leaf yellow tea drinking on diabetes and diabetic nephropathy in mice
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Qiuyan Ban | Chung S. Yang | Guangshan Zhao | Zhe Ren | Hanlin Pu | Ruixia Dong | Jianyuan Teng | Lian Yang | K. Du | Yifei Wang
[1] Ruiyue Yang,et al. Naringin Attenuates High Fat Diet Induced Non-alcoholic Fatty Liver Disease and Gut Bacterial Dysbiosis in Mice , 2020, Frontiers in Microbiology.
[2] Chung S. Yang,et al. Tea Drinking Alleviates Diabetic Symptoms via Upregulating Renal Water Reabsorption Proteins and Downregulating Renal Gluconeogenic Enzymes in db/db Mice. , 2020, Molecular nutrition & food research.
[3] H. Herrema,et al. Intestinal microbial metabolites in human metabolism and type 2 diabetes , 2020, Diabetologia.
[4] F. Ren,et al. Age- and diet-specific effects of chronic exposure to chlorpyrifos on hormones, inflammation and gut microbiota in rats. , 2019, Pesticide biochemistry and physiology.
[5] H. Qin,et al. Probiotics improve gut microbiota dysbiosis in obese mice fed a high-fat or high-sucrose diet. , 2019, Nutrition.
[6] Fitra Rianto,et al. Role of PKC and AMPK in hypertonicity-stimulated water reabsorption in rat inner medullary collecting ducts. , 2019, American journal of physiology. Renal physiology.
[7] Shuangjiang Liu,et al. Parabacteroides distasonis Alleviates Obesity and Metabolic Dysfunctions via Production of Succinate and Secondary Bile Acids. , 2019, Cell reports.
[8] X. Yao,et al. Modulation of the Gut Microbiota in Rats by Hugan Qingzhi Tablets during the Treatment of High-Fat-Diet-Induced Nonalcoholic Fatty Liver Disease , 2018, Oxidative medicine and cellular longevity.
[9] R. Reiter,et al. Melatonin reprogramming of gut microbiota improves lipid dysmetabolism in high‐fat diet‐fed mice , 2018, Journal of pineal research.
[10] Chi-Tang Ho,et al. Roasting improves the hypoglycemic effects of a large-leaf yellow tea infusion by enhancing the levels of epimerized catechins that inhibit α-glucosidase. , 2018, Food & function.
[11] I. Park,et al. Effect of Kombucha on gut-microbiota in mouse having non-alcoholic fatty liver disease , 2018, Food Science and Biotechnology.
[12] V. Tremaroli,et al. Differential metabolic effects of oral butyrate treatment in lean versus metabolic syndrome subjects , 2018, Clinical and Translational Gastroenterology.
[13] M. Murphy,et al. Ketogenic diet enhances neurovascular function with altered gut microbiome in young healthy mice , 2018, Scientific Reports.
[14] T. Igumenova,et al. Structural Basis of Protein Kinase Cα Regulation by the C-Terminal Tail. , 2018, Biophysical journal.
[15] Zhongwen Xie,et al. Large Yellow Tea Attenuates Macrophage-Related Chronic Inflammation and Metabolic Syndrome in High-Fat Diet Treated Mice. , 2018, Journal of agricultural and food chemistry.
[16] N. Vaziri,et al. Altered microbiome in chronic kidney disease: systemic effects of gut-derived uremic toxins. , 2018, Clinical science.
[17] S. Uchida,et al. Activation of AQP2 water channels without vasopressin: therapeutic strategies for congenital nephrogenic diabetes insipidus , 2018, Clinical and Experimental Nephrology.
[18] Baoxue Yang,et al. Dapagliflozin Aggravates Renal Injury via Promoting Gluconeogenesis in db/db Mice , 2018, Cellular Physiology and Biochemistry.
[19] T. Ando,et al. Increased sugar intake as a form of compensatory hyperphagia in patients with type 2 diabetes under dapagliflozin treatment. , 2018, Diabetes research and clinical practice.
[20] Ji-Hoon Lee,et al. Genes and Gut Bacteria Involved in Luminal Butyrate Reduction Caused by Diet and Loperamide , 2017, Genes.
[21] Thanka Johnson,et al. Impact of EGCG Supplementation on the Progression of Diabetic Nephropathy in Rats: An Insight into Fibrosis and Apoptosis. , 2017, Journal of agricultural and food chemistry.
[22] X. Wan,et al. Corrigendum: Safety and anti-hyperglycemic efficacy of various tea types in mice , 2016, Scientific Reports.
[23] J. Holst,et al. Distal, not proximal, colonic acetate infusions promote fat oxidation and improve metabolic markers in overweight/obese men. , 2016, Clinical science.
[24] Li Li Guo,et al. Epigallocatechin-3-gallate Attenuates Renal Damage by Suppressing Oxidative Stress in Diabetic db/db Mice , 2016, Oxidative medicine and cellular longevity.
[25] J. B. Lopes de Faria,et al. The use of green tea polyphenols for treating residual albuminuria in diabetic nephropathy: A double-blind randomised clinical trial , 2016, Scientific Reports.
[26] F. Bäckhed,et al. From Dietary Fiber to Host Physiology: Short-Chain Fatty Acids as Key Bacterial Metabolites , 2016, Cell.
[27] K. Burns,et al. PGE2 receptor EP3 inhibits water reabsorption and contributes to polyuria and kidney injury in a streptozotocin-induced mouse model of diabetes , 2016, Diabetologia.
[28] Yijun Wang,et al. Mechanisms of body weight reduction and metabolic syndrome alleviation by tea. , 2016, Molecular nutrition & food research.
[29] E. Ferrannini,et al. Energy Balance After Sodium–Glucose Cotransporter 2 Inhibition , 2015, Diabetes Care.
[30] W. D. de Vos,et al. Inulin-type fructans modulate intestinal Bifidobacterium species populations and decrease fecal short-chain fatty acids in obese women. , 2015, Clinical nutrition.
[31] M. Wheeler,et al. FFAR out new targets for diabetes. , 2015, Cell metabolism.
[32] Stefan Offermanns,et al. Loss of FFA2 and FFA3 increases insulin secretion and improves glucose tolerance in type 2 diabetes , 2015, Nature Medicine.
[33] Jimmy D Bell,et al. Effects of targeted delivery of propionate to the human colon on appetite regulation, body weight maintenance and adiposity in overweight adults , 2014, Gut.
[34] Barbara M. Bakker,et al. Gut-derived short-chain fatty acids are vividly assimilated into host carbohydrates and lipids. , 2013, American journal of physiology. Gastrointestinal and liver physiology.
[35] C. Lelliott,et al. Adaptive Changes of the Insig1/SREBP1/SCD1 Set Point Help Adipose Tissue to Cope With Increased Storage Demands of Obesity , 2013, Diabetes.
[36] Chung S. Yang,et al. Prevention of chronic diseases by tea: possible mechanisms and human relevance. , 2013, Annual review of nutrition.
[37] T. Ulven. Short-chain free fatty acid receptors FFA2/GPR43 and FFA3/GPR41 as new potential therapeutic targets , 2012, Front. Endocrin..
[38] J. Sands,et al. Lack of protein kinase C-α leads to impaired urine concentrating ability and decreased aquaporin-2 in angiotensin II-induced hypertension. , 2012, American journal of physiology. Renal physiology.
[39] S. Bagnasco. Protein kinase C-α comes to the rescue of aquaporin-2. , 2012, American journal of physiology. Renal physiology.
[40] Jun Sheng,et al. Inhibition of advanced glycation end product formation by Pu-erh tea ameliorates progression of experimental diabetic nephropathy. , 2012, Journal of agricultural and food chemistry.
[41] A. M. Habib,et al. Short-Chain Fatty Acids Stimulate Glucagon-Like Peptide-1 Secretion via the G-Protein–Coupled Receptor FFAR2 , 2012, Diabetes.
[42] Hong Zhao,et al. PKCα regulates vasopressin-induced aquaporin-2 trafficking in mouse kidney collecting duct cells in vitro via altering microtubule assembly , 2012, Acta Pharmacologica Sinica.
[43] A. Mitrakou. Kidney: its impact on glucose homeostasis and hormonal regulation. , 2011, Diabetes research and clinical practice.
[44] R. Fenton,et al. Vasopressin-independent targeting of aquaporin-2 by selective E-prostanoid receptor agonists alleviates nephrogenic diabetes insipidus , 2011, Proceedings of the National Academy of Sciences.
[45] E. Chao. A paradigm shift in diabetes therapy--dapagliflozin and other SGLT2 inhibitors. , 2011, Discovery medicine.
[46] R. Henry,et al. SGLT2 inhibition — a novel strategy for diabetes treatment , 2010, Nature Reviews Drug Discovery.
[47] A. Schwiertz,et al. Microbiota and SCFA in Lean and Overweight Healthy Subjects , 2010, Obesity.
[48] O. Marsenic. Glucose control by the kidney: an emerging target in diabetes. , 2009, American journal of kidney diseases : the official journal of the National Kidney Foundation.
[49] G. Tamma,et al. Regulation of aquaporin-2 trafficking. , 2009, Handbook of experimental pharmacology.
[50] Enrique Morales,et al. Sodium-Glucose Cotransport Inhibition With Dapagliflozin in Type 2 Diabetes , 2008, Diabetes Care.
[51] Leigh A. Stoddart,et al. International Union of Pharmacology. LXXI. Free Fatty Acid Receptors FFA1, -2, and -3: Pharmacology and Pathophysiological Functions , 2008, Pharmacological Reviews.
[52] S. Abdeen,et al. Effect of green tea on kidney tubules of diabetic rats , 2008, British Journal of Nutrition.
[53] Shinong Wang,et al. Osmotic polyuria: an overlooked mechanism in diabetic nephropathy. , 2008, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.
[54] G. King,et al. The role of protein kinase C activation in diabetic nephropathy. , 2007, Kidney international. Supplement.
[55] G. King,et al. Reduction of Diabetes-Induced Oxidative Stress, Fibrotic Cytokine Expression, and Renal Dysfunction in Protein Kinase Cβ–Null Mice , 2006, Diabetes.
[56] M. Delorenzi,et al. Transcript Profiling Suggests That Differential Metabolic Adaptation of Mice to a High Fat Diet Is Associated with Changes in Liver to Muscle Lipid Fluxes* , 2004, Journal of Biological Chemistry.
[57] H. Haller,et al. Diminished loss of proteoglycans and lack of albuminuria in protein kinase C-alpha-deficient diabetic mice. , 2004, Diabetes.
[58] V. Vallon,et al. Evidence for a role of protein kinase C-alpha in urine concentration. , 2004, American journal of physiology. Renal physiology.
[59] B. W. van Balkom,et al. Glycosylation Is Important for Cell Surface Expression of the Water Channel Aquaporin-2 but Is Not Essential for Tetramerization in the Endoplasmic Reticulum* , 2004, Journal of Biological Chemistry.
[60] K. Tuttle,et al. A novel potential therapy for diabetic nephropathy and vascular complications: protein kinase C beta inhibition. , 2003, American journal of kidney diseases : the official journal of the National Kidney Foundation.
[61] M. Krempf,et al. Production rates and metabolism of short-chain fatty acids in the colon and whole body using stable isotopes. , 2003, The Proceedings of the Nutrition Society.
[62] J. Shaw,et al. Global and societal implications of the diabetes epidemic , 2001, Nature.
[63] M. Stumvoll,et al. Renal gluconeogenesis: its importance in human glucose homeostasis. , 2001, Diabetes care.
[64] R. DeFronzo. Pharmacologic Therapy for Type 2 Diabetes Mellitus , 1999, Annals of Internal Medicine.
[65] Lawrence A Leiter,et al. Time of day and glucose tolerance status affect serum short-chain fatty acid concentrations in humans. , 1997, Metabolism: clinical and experimental.
[66] G. King,et al. Characterization of protein kinase C beta isoform activation on the gene expression of transforming growth factor-beta, extracellular matrix components, and prostanoids in the glomeruli of diabetic rats. , 1997, The Journal of clinical investigation.
[67] M. Stumvoll,et al. Renal glucose production and utilization: new aspects in humans , 1997, Diabetologia.
[68] G. Mithieux,et al. Glucose-6-Phosphatase mRNA and Activity Are Increased to the Same Extent in Kidney and Liver of Diabetic Rats , 1996, Diabetes.
[69] R. Klein. Hyperglycemie and Microvascular and Macrovascular Disease in Diabetes , 1995, Diabetes Care.
[70] S. Teutsch,et al. The problem of diabetic renal failure in the United States: an overview. , 1989, American journal of kidney diseases : the official journal of the National Kidney Foundation.
[71] P. Fournel,et al. Renal enzymes during experimental diabetes mellitus in the rat. Role of insulin, carbohydrate metabolism, and ketoacidosis. , 1984, Canadian journal of physiology and pharmacology.
[72] G. W. Sanderson,et al. The biochemistry and technology of tea manufacture. , 1980, Critical reviews in food science and nutrition.
[73] D. Williamson,et al. Origins of blood acetate in the rat. , 1977, Biochemical Journal.
[74] N. Stamm,et al. Regulation of gluconeogenesis and glycolysis: studies of mechanisms controlling enzyme activity. , 1967, Advances in enzyme regulation.