Involvement of oxidative species in cyclosporine-mediated cholestasis
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[1] Doyoung Kwon,et al. Taurine Ameliorates Tunicamycin-Induced Liver Injury by Disrupting the Vicious Cycle between Oxidative Stress and Endoplasmic Reticulum Stress , 2022, Life.
[2] S. Cantoni,et al. Efficacy and safety of cyclosporine A treatment in autoimmune cytopenias: the experience of two Italian reference centers , 2021, Therapeutic advances in hematology.
[3] Michael S. Cohen,et al. Mitochondrial Permeability Transition Causes Mitochondrial Reactive Oxygen Species- and Caspase 3-Dependent Atrophy of Single Adult Mouse Skeletal Muscle Fibers , 2021, Cells.
[4] Yu Deng,et al. Role of Calcium/Calcineurin Signalling in Regulating Intracellular Reactive Oxygen Species Homeostasis in Saccharomyces cerevisiae , 2021, Genes.
[5] K. Yalçın,et al. Analyzing the clinical outcomes of switching from cyclosporine to tacrolimus in pediatric hematopoietic stem cell transplantation , 2021, Clinical transplantation.
[6] Stephen Adeniyi Adefegha,et al. Evaluation of different almond (Terminalia catappa) extracts against oxidative stress induced by cyclosporine in brain and liver of rats , 2021, Journal of complementary & integrative medicine.
[7] Shelly C. Lu,et al. Boosting mitochondria activity by silencing MCJ overcomes cholestasis-induced liver injury , 2021, JHEP reports : innovation in hepatology.
[8] Yun Bai,et al. Calcineurin A-alpha suppression drives Nuclear Factor κB-mediated NADPH oxidase-2 upregulation. , 2021, American journal of physiology. Renal physiology.
[9] G. Mironova,et al. Mitochondrial Cyclosporine A-Independent Palmitate/Ca2+-Induced Permeability Transition Pore (PA-mPT Pore) and Its Role in Mitochondrial Function and Protection against Calcium Overload and Glutamate Toxicity , 2021, Cells.
[10] Yizhuo Wang,et al. Mitochondrial ROS promote mitochondrial dysfunction and inflammation in ischemic acute kidney injury by disrupting TFAM-mediated mtDNA maintenance , 2021, Theranostics.
[11] D. Biswas,et al. Targeting Reactive Oxygen Species Homeostasis and Metabolism in Cancer Stem Cells , 2021, Handbook of Oxidative Stress in Cancer: Mechanistic Aspects.
[12] R. Heidari,et al. Mitigation of cholestasis-associated hepatic and renal injury by edaravone treatment: Evaluation of its effects on oxidative stress and mitochondrial function , 2020 .
[13] K. Kuča,et al. Cyclosporine A: Chemistry and Toxicity - A Review. , 2020, Current medicinal chemistry.
[14] Yanmin Zhang,et al. Mitochondrial reactive oxygen species cause major oxidative mitochondrial DNA damages and repair pathways , 2020, Journal of Biosciences.
[15] Pei-Hsuan Chen,et al. Mutual interaction between endoplasmic reticulum and mitochondria in nonalcoholic fatty liver disease , 2020, Lipids in Health and Disease.
[16] T. Vanhaecke,et al. Robustness testing and optimization of an adverse outcome pathway on cholestatic liver injury , 2020, Archives of Toxicology.
[17] Y. Qiu,et al. Li-Dan-He-Ji Improves Infantile Cholestasis Hepatopathy Through Inhibiting Calcium-Sensing Receptor-Mediated Hepatocyte Apoptosis , 2020, Frontiers in Pharmacology.
[18] M. Tanokura,et al. Identification of novel interacting regions involving calcineurin and nuclear factor of activated T cells , 2020, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[19] J. Loscalzo,et al. Metabolic Responses to Reductive Stress , 2020, Antioxidants & redox signaling.
[20] B. Fromenty. Alteration of mitochondrial DNA homeostasis in drug-induced liver injury. , 2020, Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association.
[21] Ke Chen,et al. Protective Role of mTOR in Liver Ischemia/Reperfusion Injury: Involvement of Inflammation and Autophagy , 2019, Oxidative medicine and cellular longevity.
[22] Xiuwen Wu,et al. Mitochondrial DNA in liver inflammation and oxidative stress. , 2019, Life sciences.
[23] R. Wills,et al. Effects of cyclosporine and dexamethasone on canine T cell expression of interleukin-2 and interferon-gamma. , 2019, Veterinary immunology and immunopathology.
[24] M. Uysal,et al. The protective effect of resveratrol against cyclosporine A-induced oxidative stress and hepatotoxicity , 2019, Archives of physiology and biochemistry.
[25] J. Klawitter,et al. CYCLOPHILIN D KNOCKOUT PROTECTS THE MOUSE KIDNEY AGAINST CYCLOSPORIN A-INDUCED OXIDATIVE STRESS. , 2019, American journal of physiology. Renal physiology.
[26] Esin Akbay,et al. Effects of N-acetyl cysteine, vitamin E and vitamin C on liver glutathione levels following amiodarone treatment in rats , 2019, Kardiochirurgia i torakochirurgia polska = Polish journal of cardio-thoracic surgery.
[27] Ruiqi Liu,et al. Mitochondrial DNA-Induced Inflammatory Responses and Lung Injury in Thermal Injury Murine Model: Protective Effect of Cyclosporine-A. , 2019, Journal of burn care & research : official publication of the American Burn Association.
[28] V. Nincevic,et al. Mechanisms of Hepatic Cholestatic Drug Injury , 2019, Journal of clinical and translational hepatology.
[29] F. He,et al. Neutrophils promote the development of reparative macrophages mediated by ROS to orchestrate liver repair , 2019, Nature Communications.
[30] R. Heidari,et al. The Role and Study of Mitochondrial Impairment and Oxidative Stress in Cholestasis. , 2019, Methods in molecular biology.
[31] J. Fernandez-Checa,et al. Mitochondrial Oxidative Stress and Antioxidants Balance in Fatty Liver Disease , 2018, Hepatology communications.
[32] W. Meng,et al. Attenuation of Lipopolysaccharide-Induced Acute Lung Injury by Cyclosporine-A via Suppression of Mitochondrial DNA , 2018, Medical science monitor : international medical journal of experimental and clinical research.
[33] Sabine U. Vorrink,et al. Calcium Signaling in Liver Injury and Regeneration , 2018, Front. Med..
[34] G. Labbe,et al. Drug-Induced Alterations of Mitochondrial DNA Homeostasis in Steatotic and Nonsteatotic HepaRG Cells , 2018, The Journal of Pharmacology and Experimental Therapeutics.
[35] N. Samra,et al. Evaluation of vitamin D status bone mineral density and dental health in children with cholestasis. , 2018, Clinics and research in hepatology and gastroenterology.
[36] C. Moraes,et al. Mitochondrial DNA damage and reactive oxygen species in neurodegenerative disease , 2018, FEBS letters.
[37] A. Guillouzo,et al. Endoplasmic reticulum stress precedes oxidative stress in antibiotic‐induced cholestasis and cytotoxicity in human hepatocytes , 2018, Free radical biology & medicine.
[38] G. Kullak-Ublick,et al. Drug-induced bile duct injury. , 2017, Biochimica et biophysica acta. Molecular basis of disease.
[39] C. Culmsee,et al. Glucose-regulated protein 75 determines ER–mitochondrial coupling and sensitivity to oxidative stress in neuronal cells , 2017, Cell Death Discovery.
[40] Hanmei Wei,et al. Attenuation of cyclosporine A induced nephrotoxicity by schisandrin B through suppression of oxidative stress, apoptosis and autophagy , 2017, International immunopharmacology.
[41] J. Kleinjans,et al. Nuclear and Mitochondrial DNA Methylation Patterns Induced by Valproic Acid in Human Hepatocytes , 2017, Chemical research in toxicology.
[42] V. Parra,et al. Calcium Transport and Signaling in Mitochondria. , 2017, Comprehensive Physiology.
[43] A. Vickers,et al. Liver Effects of Clinical Drugs Differentiated in Human Liver Slices , 2017, International journal of molecular sciences.
[44] A. Ramos,et al. Calcineurin inhibitors cyclosporine A and tacrolimus induce vascular inflammation and endothelial activation through TLR4 signaling , 2016, Scientific Reports.
[45] G. Czechowska,et al. Oxidative Stress and Liver Morphology in Experimental Cyclosporine A-Induced Hepatotoxicity , 2016, BioMed research international.
[46] Hyung-Ryong Kim,et al. Endoplasmic Reticulum Stress and Associated ROS , 2016, International journal of molecular sciences.
[47] Longkun Li,et al. Hydrophobic bile acids relax rat detrusor contraction via inhibiting the opening of the Na+/Ca2+ exchanger , 2016, Scientific Reports.
[48] A. Michelucci,et al. Antioxidants Protect Calsequestrin-1 Knockout Mice from Halothane- and Heat-induced Sudden Death , 2015, Anesthesiology.
[49] Mathieu Vinken,et al. Adverse Outcome Pathways and Drug-Induced Liver Injury Testing. , 2015, Chemical research in toxicology.
[50] Shaoyuan Cui,et al. The mitochondrial Na+/Ca2+ exchanger may reduce high glucose-induced oxidative stress and nucleotide-binding oligomerization domain receptor 3 inflammasome activation in endothelial cells , 2015, Journal of geriatric cardiology : JGC.
[51] A. Michelucci,et al. Oxidative stress, mitochondrial damage, and cores in muscle from calsequestrin-1 knockout mice , 2015, Skeletal Muscle.
[52] Ao-lin He,et al. Ginsenoside Rg1 protects chronic cyclosporin a nephropathy from tubular cell apoptosis by inhibiting endoplasmic reticulum stress in rats. , 2015, Transplantation proceedings.
[53] Güneş Parlakgül,et al. Chronic enrichment of hepatic endoplasmic reticulum–mitochondria contact leads to mitochondrial dysfunction in obesity , 2014, Nature Medicine.
[54] J. Banales,et al. Post-translational Regulation of the Type III Inositol 1,4,5-Trisphosphate Receptor by miRNA-506* , 2014, The Journal of Biological Chemistry.
[55] A. Guillouzo,et al. Different dose-dependent mechanisms are involved in early cyclosporine a-induced cholestatic effects in hepaRG cells. , 2014, Toxicological sciences : an official journal of the Society of Toxicology.
[56] V. Sundaram,et al. Atypical causes of cholestasis. , 2014, World journal of gastroenterology.
[57] S. Ryeom,et al. Cyclosporin A Promotes Tumor Angiogenesis in a Calcineurin-Independent Manner by Increasing Mitochondrial Reactive Oxygen Species , 2014, Molecular Cancer Research.
[58] H. Cichoż-Lach,et al. Oxidative stress as a crucial factor in liver diseases. , 2014, World journal of gastroenterology.
[59] Yasunori Sato,et al. A possible involvement of endoplasmic reticulum stress in biliary epithelial autophagy and senescence in primary biliary cirrhosis , 2014, Journal of Gastroenterology.
[60] A. Boorsma,et al. Detection of the mechanism of immunotoxicity of cyclosporine A in murine in vitro and in vivo models , 2014, Archives of Toxicology.
[61] M. Nathanson,et al. Calcium signaling in the liver. , 2013, Comprehensive Physiology.
[62] P. Bie,et al. Damage to mtDNA in liver injury of patients with extrahepatic cholestasis: the protective effects of mitochondrial transcription factor A. , 2012, Free radical biology & medicine.
[63] J. Sastre,et al. Mitochondrial dysfunction in cholestatic liver diseases. , 2012, Frontiers in bioscience.
[64] Y. Aniya,et al. The role of a membrane-bound glutathione transferase in the peroxynitrite-induced mitochondrial permeability transition pore: formation of a disulfide-linked protein complex. , 2011, Archives of biochemistry and biophysics.
[65] G. Giaever,et al. Inhibition of mitochondrial translation as a therapeutic strategy for human acute myeloid leukemia. , 2011, Cancer cell.
[66] H. Jaeschke,et al. Reactive oxygen species in the normal and acutely injured liver. , 2011, Journal of hepatology.
[67] H. Erdjument-Bromage,et al. TLR signaling augments macrophage bactericidal activity through mitochondrial ROS , 2011, Nature.
[68] B. Aggarwal,et al. Oxidative stress, inflammation, and cancer: how are they linked? , 2010, Free radical biology & medicine.
[69] T. Peng,et al. Oxidative stress caused by mitochondrial calcium overload , 2010, Annals of the New York Academy of Sciences.
[70] H. Jaeschke,et al. Oxidative Stress and the Pathogenesis of Cholestasis , 2010, Seminars in liver disease.
[71] Tsu-Kung Lin,et al. Early transcriptional deregulation of hepatic mitochondrial biogenesis and its consequent effects on murine cholestatic liver injury , 2009, Apoptosis.
[72] Hongqiao Zhang,et al. Glutathione: overview of its protective roles, measurement, and biosynthesis. , 2009, Molecular aspects of medicine.
[73] K. Bushby,et al. Cyclosporine A treatment for Ullrich congenital muscular dystrophy: a cellular study of mitochondrial dysfunction and its rescue. , 2009, Brain : a journal of neurology.
[74] Hasibur Rehman,et al. NIM811 (N-Methyl-4-isoleucine Cyclosporine), a Mitochondrial Permeability Transition Inhibitor, Attenuates Cholestatic Liver Injury but Not Fibrosis in Mice , 2008, Journal of Pharmacology and Experimental Therapeutics.
[75] P. Beaune,et al. Cyclosporine‐Induced Endoplasmic Reticulum Stress Triggers Tubular Phenotypic Changes and Death , 2008, American journal of transplantation : official journal of the American Society of Transplantation and the American Society of Transplant Surgeons.
[76] Y. Mizuguchi,et al. Hepatotoxicity caused by both tacrolimus and cyclosporine after living donor liver transplantation. , 2008, Journal of Nippon Medical School = Nippon Ika Daigaku zasshi.
[77] D. Pessayre,et al. Tamoxifen Inhibits Topoisomerases, Depletes Mitochondrial DNA, and Triggers Steatosis in Mouse Liver , 2007, Journal of Pharmacology and Experimental Therapeutics.
[78] C. Sabin,et al. Incidence and risk factors for the development of prolonged and severe intrahepatic cholestasis after liver transplantation , 2006, Liver transplantation : official publication of the American Association for the Study of Liver Diseases and the International Liver Transplantation Society.
[79] A. Miyawaki,et al. Identification of Mitochondrial DNA Polymorphisms That Alter Mitochondrial Matrix pH and Intracellular Calcium Dynamics , 2006, PLoS genetics.
[80] J. Duszyński,et al. Calcium Signals Are Affected by Ciprofloxacin as a Consequence of Reduction of Mitochondrial DNA Content in Jurkat Cells , 2006, Antimicrobial Agents and Chemotherapy.
[81] L. Van Haute,et al. Linezolid-induced inhibition of mitochondrial protein synthesis. , 2006, Clinical infectious diseases : an official publication of the Infectious Diseases Society of America.
[82] R. Sokol,et al. Human Hepatic Mitochondria Generate Reactive Oxygen Species and Undergo the Permeability Transition in Response to Hydrophobic Bile Acids , 2005, Journal of pediatric gastroenterology and nutrition.
[83] T. D. Pugh,et al. Mitochondrial DNA Mutations, Oxidative Stress, and Apoptosis in Mammalian Aging , 2005, Science.
[84] Jeffrey Robbins,et al. Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death , 2005, Nature.
[85] H. Ke,et al. Crystal structures of cyclophilin and its partners. , 2004, Frontiers in bioscience : a journal and virtual library.
[86] R. Rezzani,et al. Cyclosporine A and adverse effects on organs: histochemical studies. , 2004, Progress in histochemistry and cytochemistry.
[87] T. Seidler,et al. Na(+)-Ca(2+) exchanger overexpression predisposes to reactive oxygen species-induced injury. , 2003, Cardiovascular research.
[88] M. Nathanson,et al. Loss of inositol 1,4,5-trisphosphate receptors from bile duct epithelia is a common event in cholestasis. , 2003, Gastroenterology.
[89] D. Pessayre,et al. Tacrine inhibits topoisomerases and DNA synthesis to cause mitochondrial DNA depletion and apoptosis in mouse liver , 2003, Hepatology.
[90] Y. Shinohara,et al. Silver ion induces a cyclosporine a-insensitive permeability transition in rat liver mitochondria and release of apoptogenic cytochrome C. , 2003, Journal of biochemistry.
[91] E. Bertini,et al. Analysis of glutathione: implication in redox and detoxification. , 2003, Clinica chimica acta; international journal of clinical chemistry.
[92] J. Prieto,et al. Liver failure caused by herpes simplex virus thymidine kinase plus ganciclovir therapy is associated with mitochondrial dysfunction and mitochondrial DNA depletion. , 2003, Human gene therapy.
[93] Shelly C. Lu,et al. Functional proteomics of nonalcoholic steatohepatitis: Mitochondrial proteins as targets of S-adenosylmethionine , 2003, Proceedings of the National Academy of Sciences of the United States of America.
[94] D. Pessayre,et al. Impaired adaptive resynthesis and prolonged depletion of hepatic mitochondrial DNA after repeated alcohol binges in mice. , 2002, Gastroenterology.
[95] M. Cascales,et al. Novel mechanism of Vitamin E protection against cyclosporine A cytotoxicity in cultured rat hepatocytes. , 2002, Biochemical pharmacology.
[96] D. Bartelt,et al. Oxidative stress-induced calcium signalling in Aspergillus nidulans. , 2002, Cellular signalling.
[97] N. Tygstrup,et al. Cholestasis and regulation of genes related to drug metabolism and biliary transport in rat liver following treatment with cyclosporine A and sirolimus (Rapamycin). , 2001, Pharmacology & toxicology.
[98] J. González‐Gallego,et al. Effects of aging on the susceptibility to the toxic effects of cyclosporin A in rats. Changes in liver glutathione and antioxidant enzymes. , 2001, Free radical biology & medicine.
[99] R. Sokol,et al. Bile acid‐induced rat hepatocyte apoptosis is inhibited by antioxidants and blockers of the mitochondrial permeability transition , 2001, Hepatology.
[100] J. Palomero,et al. Glutathione Metabolism In Cyclosporine A‐Treated Rats: Dose‐ And Time‐Related Changes In Liver And Kidney , 2000, Clinical and experimental pharmacology & physiology.
[101] P. Oliveira,et al. Bile acids affect liver mitochondrial bioenergetics: possible relevance for cholestasis therapy. , 2000, Toxicological sciences : an official journal of the Society of Toxicology.
[102] J. Parks,et al. Mitochondrial DNA-depleted neuroblastoma (Rho‡) cells exhibit altered calcium signaling , 2000 .
[103] C. Canter,et al. Increased morbidity and high variability of cyclosporine levels in pediatric heart transplant recipients. , 2000, The Journal of heart and lung transplantation : the official publication of the International Society for Heart Transplantation.
[104] D. Turnbull,et al. Reanalysis and revision of the Cambridge reference sequence for human mitochondrial DNA , 1999, Nature Genetics.
[105] G. Gores,et al. Induction of the mitochondrial permeability transition as a mechanism of liver injury during cholestasis: a potential role for mitochondrial proteases. , 1998, Biochimica et biophysica acta.
[106] A. I. Galán,et al. Inhibition of biliary glutathione secretion by cyclosporine A in the rat: possible mechanisms and role in the cholestasis induced by the drug. , 1998, Journal of hepatology.
[107] M. Deters,et al. Reevaluation of cyclosporine induced hepatotoxicity in the isolated perfused rat liver. , 1997, Toxicology.
[108] H. C. Lee,et al. Mutation and oxidative damage of mitochondrial DNA and defective turnover of mitochondria in human aging. , 1997, Journal of the Formosan Medical Association = Taiwan yi zhi.
[109] E. Shaffer,et al. Cholestatic effects of cyclosporine in the rat. , 1997, Transplantation.
[110] W. Trommer,et al. Cyclosporine A-induced oxidative stress in rat hepatocytes. , 1997, The Journal of pharmacology and experimental therapeutics.
[111] E. Levine,et al. Fialuridine and its metabolites inhibit DNA polymerase gamma at sites of multiple adjacent analog incorporation, decrease mtDNA abundance, and cause mitochondrial structural defects in cultured hepatoblasts. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[112] P. Carrupt,et al. Trimetazidine reverses calcium accumulation and impairment of phosphorylation induced by cyclosporine A in isolated rat liver mitochondria. , 1996, The Journal of pharmacology and experimental therapeutics.
[113] M. Soresi,et al. [Effects of cyclosporin A on various indices of cholestasis in kidney transplant recipients]. , 1995, Minerva urologica e nefrologica = The Italian journal of urology and nephrology.
[114] B. Herman,et al. Mitochondrial and glycolytic dysfunction in lethal injury to hepatocytes by t-butylhydroperoxide: protection by fructose, cyclosporin A and trifluoperazine. , 1993, The Journal of pharmacology and experimental therapeutics.
[115] A. Neims,et al. 4‐Quinolones cause a selective loss of mitochondrial DNA from mouse L1210 leukemia cells , 1993, Journal of cellular biochemistry.
[116] D. J. Reed,et al. Cyclosporin A protects hepatocytes subjected to high Ca2+ and oxidative stress , 1992, FEBS letters.
[117] R. Branch,et al. Cholestasis in transplant patients—what is the role of cyclosporin? , 1992, Alimentary pharmacology & therapeutics.
[118] U. Di Tondo,et al. [Cholestasis in liver transplantation: incidence and diagnostic significance]. , 1990, Pathologica.
[119] S. Orrenius,et al. Release of Ca2+ from the endoplasmic reticulum is not the mechanism for bile acid-induced cholestasis and hepatotoxicity in the intact rat liver. , 1990, The Journal of clinical investigation.
[120] M. Dempsey,et al. Cyclosporin A is a potent inhibitor of the inner membrane permeability transition in liver mitochondria. , 1989, The Journal of biological chemistry.
[121] S. Orrenius,et al. Correlation between cytosolic Ca2+ concentration and cytotoxicity in hepatocytes exposed to oxidative stress. , 1988, Toxicology.
[122] A. Crevât,et al. Action of cyclosporine on mitochondrial calcium fluxes , 1987, Journal of bioenergetics and biomembranes.
[123] F. Berr,et al. Characterization of calcium deprivation-induced cholestasis in the perfused rat liver. , 1985, American Journal of Physiology.
[124] K. Jung,et al. Influence of cyclosporin A on the respiration of isolated rat kidney mitochondria , 1985, FEBS letters.
[125] G. Santacana,et al. The Na+/Ca2+ exchange system of the liver cell. , 1985, Research communications in chemical pathology and pharmacology.
[126] I. Rosenberg,et al. Intestinal Calcium Absorption and Vitamin D Status in Chronic Cholestatic Liver Disease , 1984, Hepatology.