Human mesothelial cells are unusually susceptible to simian virus 40-mediated transformation and asbestos cocarcinogenicity.
暂无分享,去创建一个
H. Pass | J. Testa | M. Carbone | M. Bocchetta | A. Powers | P. Rizzo | J R Testa | H I Pass | M Carbone | R Fresco | P Rizzo | I Di Resta | A Powers | M Bocchetta | A Tosolini | A. Tosolini | I. Di Resta | R. Fresco | Ilaria Di Resta | Paola Rizzo
[1] E. Appella,et al. Human wild-type p53 adopts a unique conformational and phosphorylation state in vivo during growth arrest of glioblastoma cells. , 1992, Oncogene.
[2] D. Lane,et al. p53 and DNA polymerase α compete for binding to SV40 T antigen , 1987, Nature.
[3] R. Fisher,et al. Simian virus 40 is present in most United States human mesotheliomas, but it is rarely present in non-Hodgkin's lymphoma. , 1999, Chest.
[4] H. Pass,et al. The retinoblastoma gene family pRb/p105, p107, pRb2/p130 and simian virus-40 large T-antigen in human mesotheliomas , 1997, Nature Medicine.
[5] I. Wistuba,et al. Presence of simian virus 40 sequences in malignant mesotheliomas and mesothelial cell proliferations , 2000, Journal of cellular biochemistry.
[6] J. Butel,et al. Response: More About: Cell and Molecular Biology of Simian Virus 40: Implications for Human Infections and Disease. , 2000, Journal of the National Cancer Institute.
[7] D. Schrump,et al. Antisense to SV40 early gene region induces growth arrest and apoptosis in T-antigen-positive human pleural mesothelioma cells. , 1999, Cancer research.
[8] R. Reddel,et al. SV40-induced immortalization of human cells. , 1994, Critical reviews in oncogenesis.
[9] J. Milner. A conformation hypothesis for the suppressor and promoter functions of p53 in cell growth control and in cancer , 1991, Proceedings of the Royal Society of London. Series B: Biological Sciences.
[10] Lena Sun,et al. Regulation of Myocardial β-Adrenergic Receptor Function in Adult and Neonatal Rabbits , 1999, Neonatology.
[11] H. Pass,et al. New molecular and epidemiological issues in mesothelioma: Role of SV40 , 1999, Journal of cellular physiology.
[12] H. Zhen,et al. Expression of the simian virus 40 large tumor antigen (Tag) and formation of Tag‐p53 and Tag‐pRb complexes in human brain tumors , 1999, Cancer.
[13] N. Heintz,et al. Persistent induction of c-fos and c-jun expression by asbestos. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[14] É. Khandjian,et al. Physiological and cytogenetic characterization of immortalized human endometriotic cells containing episomal simian virus 40 DNA. , 1999, The American journal of pathology.
[15] S. Steinberg,et al. Simian virus-40 large-T antigen binds p53 in human mesotheliomas , 1997, Nature Medicine.
[16] N. Bouck,et al. A novel simian virus 40 early-region domain mediates transactivation of the cyclin A promoter by small-t antigen and is required for transformation in small-t antigen-dependent assays , 1996, Journal of virology.
[17] C. Cicala,et al. SV40 induces mesotheliomas in hamsters. , 1993, The American journal of pathology.
[18] G. Jacobson,et al. Mutations of p53 gene and SV40 sequences in asbestos associated and non-asbestos-associated mesotheliomas. , 1999, Journal of clinical pathology.
[19] G. Rosenthal,et al. Asbestos Toxicity: An Immunologic Perspective , 1999, Reviews on environmental health.
[20] R. Robledo,et al. Cellular and molecular mechanisms of asbestos‐induced fibrosis , 1999, Journal of cellular physiology.
[21] L. Norkin,et al. Human glioblastoma cells persistently infected with simian virus 40 carry nondefective episomal viral DNA and acquire the transformed phenotype and numerous chromosomal abnormalities , 1985, Journal of virology.
[22] J. Jenkins,et al. Mouse p53 inhibits SV40 origin-dependent DNA replication , 1987, Nature.
[23] E. Yamasaki,et al. Maintenance of episomal SV40 genomes in GM637 human fibroblasts. , 1999, Virology.
[24] K. Dixon,et al. Enhanced metastasis of tumours induced by a SV40 small T deletion mutant , 1982, Nature.