Pathology and Pathogenesis of Severe Acute Respiratory Syndrome
暂无分享,去创建一个
[1] Junya Fukuoka,et al. Lung pathology of severe acute respiratory syndrome (SARS): a study of 8 autopsy cases from Singapore , 2003, Human Pathology.
[2] Qingling Zhang,et al. Organ distribution of severe acute respiratory syndrome (SARS) associated coronavirus (SARS‐CoV) in SARS patients: implications for pathogenesis and virus transmission pathways , 2004, The Journal of pathology.
[3] M. Carrington,et al. A Dendritic Cell–Specific Intercellular Adhesion Molecule 3–Grabbing Nonintegrin (Dc-Sign)–Related Protein Is Highly Expressed on Human Liver Sinusoidal Endothelial Cells and Promotes HIV-1 Infection , 2001, The Journal of experimental medicine.
[4] Y. Chan,et al. Short term outcome and risk factors for adverse clinical outcomes in adults with severe acute respiratory syndrome (SARS) , 2003, Thorax.
[5] W. Hong,et al. Overexpression of 7a, a Protein Specifically Encoded by the Severe Acute Respiratory Syndrome Coronavirus, Induces Apoptosis via a Caspase-Dependent Pathway , 2004, Journal of Virology.
[6] Yi Guan,et al. Lung pathology of fatal severe acute respiratory syndrome , 2003, The Lancet.
[7] S. Harrison,et al. SARS Coronavirus, but Not Human Coronavirus NL63, Utilizes Cathepsin L to Infect ACE2-expressing Cells , 2006, Journal of Biological Chemistry.
[8] Kuender D Yang,et al. Altered p38 Mitogen-Activated Protein Kinase Expression in Different Leukocytes with Increment of Immunosuppressive Mediators in Patients with Severe Acute Respiratory Syndrome1 , 2004, The Journal of Immunology.
[9] W. Leung,et al. Coronaviral hypothetical and structural proteins were found in the intestinal surface enterocytes and pneumocytes of severe acute respiratory syndrome (SARS) , 2005, Modern Pathology.
[10] F. Pei,et al. Severe Acute Respiratory Syndrome Associated Coronavirus Is Detected in Intestinal Tissues of Fatal Cases , 2005, The American Journal of Gastroenterology.
[11] J. Loo,et al. Association of HLA class I with severe acute respiratory syndrome coronavirus infection , 2003, BMC Medical Genetics.
[12] S. Diamond,et al. Inhibitors of cathepsin L prevent severe acute respiratory syndrome coronavirus entry. , 2005, Proceedings of the National Academy of Sciences of the United States of America.
[13] Bo Zhang,et al. Multiple organ infection and the pathogenesis of SARS , 2005, The Journal of experimental medicine.
[14] Christophe Fraser,et al. The Epidemiology of Severe Acute Respiratory Syndrome in the 2003 Hong Kong Epidemic: An Analysis of All 1755 Patients , 2004, Annals of Internal Medicine.
[15] J. Peiris,et al. Academic Editor: Sherif Zaki, Centers for Disease Control, United States of America , 2005 .
[16] G. Gao,et al. SARS coronavirus induces apoptosis in Vero E6 Cells , 2004, Journal of medical virology.
[17] Yao-Hsu Yang,et al. Autoantibodies against human epithelial cells and endothelial cells after severe acute respiratory syndrome (SARS)‐associated coronavirus infection , 2005, Journal of medical virology.
[18] Michelle M. Packard,et al. Immunohistochemical, in situ hybridization, and ultrastructural localization of SARS-associated coronavirus in lung of a fatal case of severe acute respiratory syndrome in Taiwan , 2005, Human Pathology.
[19] Malik Peiris,et al. Aetiology: Koch's postulates fulfilled for SARS virus , 2003, Nature.
[20] D. Dwyer,et al. Effects of severe acute respiratory syndrome (SARS) coronavirus infection on peripheral blood lymphocytes and their subsets , 2005, International Journal of Infectious Diseases.
[21] W. Travis,et al. Analysis of deaths during the severe acute respiratory syndrome (SARS) epidemic in Singapore: challenges in determining a SARS diagnosis. , 2004, Archives of pathology & laboratory medicine.
[22] K. Wong,et al. Myopathic changes associated with severe acute respiratory syndrome: a postmortem case series. , 2005, Archives of neurology.
[23] J. Sung,et al. Association of Human-Leukocyte-Antigen Class I (B*0703) and Class II (DRB1*0301) Genotypes with Susceptibility and Resistance to the Development of Severe Acute Respiratory Syndrome , 2004, The Journal of infectious diseases.
[24] Jenq-Wen Huang,et al. Acute renal failure in SARS patients: more than rhabdomyolysis , 2004, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.
[25] X. L. Liu,et al. Isolation and Characterization of Viruses Related to the SARS Coronavirus from Animals in Southern China , 2003, Science.
[26] Pan‐Chyr Yang,et al. Evolution of pulmonary pathology in severe acute respiratory syndrome. , 2005, Journal of the Formosan Medical Association = Taiwan yi zhi.
[27] K. Kain,et al. Fatal Severe Acute Respiratory Syndrome Is Associated with Multiorgan Involvement by Coronavirus , 2005, The Journal of infectious diseases.
[28] J. Sung,et al. Detection of SARS Coronavirus RNA in the Cerebrospinal Fluid of a Patient with Severe Acute Respiratory Syndrome , 2003, Clinical chemistry.
[29] K. To,et al. The comparative pathology of severe acute respiratory syndrome and avian influenza A subtype H5N1—a review , 2006, Human Pathology.
[30] J. Peiris,et al. Mannose-Binding Lectin in Severe Acute Respiratory Syndrome Coronavirus Infection , 2005, The Journal of infectious diseases.
[31] Y. Lau,et al. Chemokine up-regulation in SARS-coronavirus–infected, monocyte-derived human dendritic cells , 2005, Blood.
[32] L. Poon,et al. Clinical progression and viral load in a community outbreak of coronavirus-associated SARS pneumonia : a prospective study , 2003 .
[33] J. Chan,et al. The spectrum of pathological changes in severe acute respiratory syndrome (SARS) , 2004, Histopathology.
[34] G. Navis,et al. Tissue distribution of ACE2 protein, the functional receptor for SARS coronavirus. A first step in understanding SARS pathogenesis , 2004, The Journal of pathology.
[35] Andreas Pichlmair,et al. Inhibition of Beta Interferon Induction by Severe Acute Respiratory Syndrome Coronavirus Suggests a Two-Step Model for Activation of Interferon Regulatory Factor 3 , 2005, Journal of Virology.
[36] J. Sung,et al. Retrospective analysis of liver function derangement in severe acute respiratory syndrome , 2004, The American Journal of Medicine.
[37] Ralph S. Baric,et al. Severe Acute Respiratory Syndrome Coronavirus Infection of Human Ciliated Airway Epithelia: Role of Ciliated Cells in Viral Spread in the Conducting Airways of the Lungs , 2005, Journal of Virology.
[38] Jonathan H. Epstein,et al. Bats Are Natural Reservoirs of SARS-Like Coronaviruses , 2005, Science.
[39] Christian Drosten,et al. Identification of a novel coronavirus in patients with severe acute respiratory syndrome. , 2003, The New England journal of medicine.
[40] C. Fraser,et al. Epidemiological determinants of spread of causal agent of severe acute respiratory syndrome in Hong Kong , 2003, The Lancet.
[41] J. Sung,et al. Plasma inflammatory cytokines and chemokines in severe acute respiratory syndrome , 2004, Clinical and experimental immunology.
[42] J. Gu,et al. Orchitis: A Complication of Severe Acute Respiratory Syndrome (SARS)1 , 2006, Biology of reproduction.
[43] J. A. Comer,et al. Ultrastructural Characterization of SARS Coronavirus , 2004, Emerging infectious diseases.
[44] Elizabeth Rea,et al. Clinical features and short-term outcomes of 144 patients with SARS in the greater Toronto area. , 2003, JAMA.
[45] Y. Guan,et al. Cytokine Responses in Severe Acute Respiratory Syndrome Coronavirus-Infected Macrophages In Vitro: Possible Relevance to Pathogenesis , 2005, Journal of Virology.
[46] Y. Guan,et al. Detection of SARS Coronavirus in Patients with Suspected SARS , 2004, Emerging infectious diseases.
[47] N. Nagata,et al. Laboratory and Epidemiology Communications SARS Coronavirus-Infected Cells in Lung Detected by New In Situ Hybridization Technique , 2003 .
[48] Xin Li,et al. The clinical pathology of severe acute respiratory syndrome (SARS): a report from China , 2003, The Journal of pathology.
[49] S. Perlman,et al. ACE2 Receptor Expression and Severe Acute Respiratory Syndrome Coronavirus Infection Depend on Differentiation of Human Airway Epithelia , 2005, Journal of Virology.
[50] N. Zhong,et al. Detection of Severe Acute Respiratory Syndrome Coronavirus in the Brain: Potential Role of the Chemokine Mig in Pathogenesis , 2005, Clinical infectious diseases : an official publication of the Infectious Diseases Society of America.
[51] C. Hsiao,et al. Modeling the Early Events of Severe Acute Respiratory Syndrome Coronavirus Infection In Vitro , 2006, Journal of Virology.
[52] A. Steinkasserer,et al. DC-SIGN and DC-SIGNR Interact with the Glycoprotein of Marburg Virus and the S Protein of Severe Acute Respiratory Syndrome Coronavirus , 2004, Journal of Virology.
[53] Mark Chappell,et al. A crucial role of angiotensin converting enzyme 2 (ACE2) in SARS coronavirus–induced lung injury , 2005, Nature Medicine.
[54] Bor-Luen Chiang,et al. Patient data, early SARS epidemic, Taiwan. , 2004, Emerging infectious diseases.
[55] M. McNutt,et al. Pathology of the thyroid in severe acute respiratory syndrome☆ , 2006, Human Pathology.
[56] Z. Lang,et al. A clinicopathological study of three cases of severe acute respiratory syndrome (SARS) , 2003, Pathology.
[57] Jun Xu,et al. Characterization of cytokine/chemokine profiles of severe acute respiratory syndrome. , 2005, American journal of respiratory and critical care medicine.
[58] A. Danchin,et al. The Severe Acute Respiratory Syndrome , 2003 .
[59] K. Subbarao,et al. pH-Dependent Entry of Severe Acute Respiratory Syndrome Coronavirus Is Mediated by the Spike Glycoprotein and Enhanced by Dendritic Cell Transfer through DC-SIGN , 2004, Journal of Virology.
[60] S. Zhang,et al. Bcl-xL inhibits T-cell apoptosis induced by expression of SARS coronavirus E protein in the absence of growth factors. , 2005, The Biochemical journal.
[61] F. Lai,et al. Acute renal impairment in coronavirus-associated severe acute respiratory syndrome , 2005, Kidney International.
[62] Martin L Hibberd,et al. A human in vitro model system for investigating genome-wide host responses to SARS coronavirus infection , 2004, BMC infectious diseases.
[63] Z. Li,et al. Expression of elevated levels of pro‐inflammatory cytokines in SARS‐CoV‐infected ACE2+ cells in SARS patients: relation to the acute lung injury and pathogenesis of SARS† , 2006, The Journal of pathology.
[64] P. Openshaw. Potential therapeutic implications of new insights into respiratory syncytial virus disease , 2002, Respiratory research.
[65] Larissa B. Thackray,et al. CD209L (L-SIGN) is a receptor for severe acute respiratory syndrome coronavirus. , 2004, Proceedings of the National Academy of Sciences of the United States of America.
[66] C. Schwegmann-Wessels,et al. Analysis of ACE2 in polarized epithelial cells: surface expression and function as receptor for severe acute respiratory syndrome-associated coronavirus. , 2006, The Journal of general virology.
[67] J. Sung,et al. The 3a protein of severe acute respiratory syndrome-associated coronavirus induces apoptosis in Vero E6 cells. , 2005, The Journal of general virology.
[68] P. Liao,et al. Antibody to severe acute respiratory syndrome (SARS)-associated coronavirus spike protein domain 2 cross-reacts with lung epithelial cells and causes cytotoxicity , 2005, Clinical and experimental immunology.
[69] S. Pillai,et al. Innate immunity. , 1996, Current opinion in immunology.
[70] M. McNutt,et al. The spleen as a target in severe acute respiratory syndrome , 2006, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[71] J. Sung,et al. Pulmonary pathological features in coronavirus associated severe acute respiratory syndrome (SARS) , 2004, Journal of Clinical Pathology.
[72] Tzei-Yi Lin,et al. Detection of Severe Acute Respiratory Syndrome–Associated Coronavirus in Pneumocytes of the Lung , 2004, American journal of clinical pathology.
[73] John L. Sullivan,et al. Angiotensin-converting enzyme 2 is a functional receptor for the SARS coronavirus , 2003, Nature.
[74] D. Weissman,et al. Constitutive and induced expression of DC‐SIGN on dendritic cell and macrophage subpopulations in situ and in vitro , 2002, Journal of leukocyte biology.
[75] J. Sung,et al. Haematological manifestations in patients with severe acute respiratory syndrome: retrospective analysis , 2003, BMJ : British Medical Journal.
[76] J. Sung,et al. Persistent infection of SARS coronavirus in colonic cells in vitro , 2004, Journal of medical virology.
[77] Paroma Basu. Iraq's public health infrastructure a casualty of war , 2004, Nature Medicine.
[78] J. Sung,et al. Clinical significance of hepatic derangement in severe acute respiratory syndrome. , 2005, World journal of gastroenterology.
[79] Arthur S Slutsky,et al. Angiotensin-converting enzyme 2 protects from severe acute lung failure , 2005, Nature.
[80] Lucy A. Perrone,et al. Severe Acute Respiratory Syndrome and the Innate Immune Responses: Modulation of Effector Cell Function without Productive Infection1 , 2005, The Journal of Immunology.
[81] G. Tse,et al. Tissue and cellular tropism of the coronavirus associated with severe acute respiratory syndrome: an in‐situ hybridization study of fatal cases , 2004, The Journal of pathology.
[82] J. Sung,et al. Early Enhanced Expression of Interferon-Inducible Protein-10 (CXCL-10) and Other Chemokines Predicts Adverse Outcome in Severe Acute Respiratory Syndrome , 2005, Clinical chemistry.
[83] Lanjuan Li,et al. SARS-coronavirus replicates in mononuclear cells of peripheral blood (PBMCs) from SARS patients , 2003, Journal of Clinical Virology.
[84] V. ter meulen,et al. Measles virus induced immunosuppression: targets and effector mechanisms. , 2001, Current molecular medicine.
[85] K. Yuen,et al. Possible Central Nervous System Infection by SARS Coronavirus , 2004, Emerging infectious diseases.
[86] S. Asa,et al. Pulmonary pathology of severe acute respiratory syndrome in Toronto , 2005, Modern Pathology.
[87] Obi L. Griffith,et al. The Genome Sequence of the SARS-Associated Coronavirus , 2003, Science.
[88] Peter B Jahrling,et al. Exotic emerging viral diseases: progress and challenges , 2004, Nature Medicine.
[89] W. Mak,et al. Homozygous L-SIGN (CLEC4M) plays a protective role in SARS coronavirus infection , 2005, Nature Genetics.
[90] J. Sung,et al. Enteric involvement of severe acute respiratory syndrome-associated coronavirus infection , 2003, Gastroenterology.
[91] Michel Theron,et al. An interferon‐γ‐related cytokine storm in SARS patients† , 2004, Journal of medical virology.
[92] M. McNutt,et al. Molecular Pathology in the Lungs of Severe Acute Respiratory Syndrome Patients , 2007, The American Journal of Pathology.
[93] Ching-lung Lai,et al. SARS‐associated viral hepatitis caused by a novel coronavirus: Report of three cases , 2004, Hepatology.
[94] Z. Zou,et al. Prognostic Factors for Severe Acute Respiratory Syndrome: A Clinical Analysis of 165 Cases , 2004, Clinical infectious diseases : an official publication of the Infectious Diseases Society of America.