Coupled left-shift of Nav channels: modeling the Na+-loading and dysfunctional excitability of damaged axons
暂无分享,去创建一个
[1] C. Morris,et al. Activation of mechanosensitive currents in traumatized membrane. , 1999, American journal of physiology. Cell physiology.
[2] C. Morris,et al. Impaired stretch modulation in potentially lethal cardiac sodium channel mutants , 2010, Channels.
[3] O. Andersen,et al. Docosahexaenoic acid alters bilayer elastic properties , 2007, Proceedings of the National Academy of Sciences.
[4] M. Devor. Ectopic discharge in Aβ afferents as a source of neuropathic pain , 2009, Experimental Brain Research.
[5] Devin K. Binder,et al. Analysis of Astroglial K+ Channel Expression in the Developing Hippocampus Reveals a Predominant Role of the Kir4.1 Subunit , 2009, The Journal of Neuroscience.
[6] Maarten H. P. Kole,et al. First Node of Ranvier Facilitates High-Frequency Burst Encoding , 2011, Neuron.
[7] Pierre-Alexandre Boucher,et al. Erratum to: Coupled left-shift of Nav channels: modeling the Na+-loading and dysfunctional excitability of damaged axons , 2012, Journal of Computational Neuroscience.
[8] K. Thorneloe,et al. The voltage-gated sodium channel Nav1.9 is required for inflammation-based urinary bladder dysfunction , 2009, Neuroscience Letters.
[9] Douglas H. Smith,et al. Dendritic alterations after dynamic axonal stretch injury in vitro , 2010, Experimental Neurology.
[10] Catherine E. Morris,et al. Voltage-Gated Channel Mechanosensitivity: Fact or Friction? , 2011, Front. Physio..
[11] J. S. Coggan,et al. Imbalance of ionic conductances contributes to diverse symptoms of demyelination , 2010, Proceedings of the National Academy of Sciences.
[12] Steven Petrou,et al. Heat opens axon initial segment sodium channels: A febrile seizure mechanism? , 2009, Annals of neurology.
[13] K. Sakurai,et al. Mexiletine suppresses nodal persistent sodium currents in sensory axons of patients with neuropathic pain , 2010, Clinical Neurophysiology.
[14] Marcello Massimini,et al. A perturbational approach for evaluating the brain's capacity for consciousness. , 2009, Progress in brain research.
[15] Massimo Mantegazza,et al. Voltage-gated sodium channels as therapeutic targets in epilepsy and other neurological disorders , 2010, The Lancet Neurology.
[16] Maxim Bazhenov,et al. Pattern of trauma determines the threshold for epileptic activity in a model of cortical deafferentation , 2011, Proceedings of the National Academy of Sciences.
[17] C. Morris,et al. Membrane stretch affects gating modes of a skeletal muscle sodium channel. , 1999, Biophysical journal.
[18] D. Saint,et al. Riluzole protects against cardiac ischaemia and reperfusion damage via block of the persistent sodium current , 2010, British journal of pharmacology.
[19] D. Allen,et al. Stretch-induced membrane damage in muscle: comparison of wild-type and mdx mice. , 2010, Advances in experimental medicine and biology.
[20] G. Somjen,et al. Simulated seizures and spreading depression in a neuron model incorporating interstitial space and ion concentrations. , 2000, Journal of neurophysiology.
[21] A. Hodgkin. The local electric changes associated with repetitive action in a non‐medullated axon , 1948, The Journal of physiology.
[22] M. Bazhenov,et al. Ionic Dynamics Mediate Spontaneous Termination of Seizures and Postictal Depression State , 2011, The Journal of Neuroscience.
[23] R. Keynes,et al. ELECTROGENIC ION PUMPS , 1974, Annals of the New York Academy of Sciences.
[24] Frederick Sachs,et al. Mechanosensitivity of Nav1.5, a voltage‐sensitive sodium channel , 2010, The Journal of physiology.
[25] C. Morris,et al. Voltage oscillations in the barnacle giant muscle fiber. , 1981, Biophysical journal.
[26] E. Vizi,et al. Binding of sodium channel inhibitors to hyperpolarized and depolarized conformations of the channel , 2011, Neuropharmacology.
[27] Z. Nusser,et al. Molecular Identity of Dendritic Voltage-Gated Sodium Channels , 2010, Science.
[28] B. Kelley,et al. Biochemical, Structural, and Biomarker Evidence for Calpain-Mediated Cytoskeletal Change After Diffuse Brain Injury Uncomplicated by Contusion , 2009, Journal of neuropathology and experimental neurology.
[29] I. Soltesz,et al. Selective depolarization of interneurons in the early posttraumatic dentate gyrus: involvement of the Na(+)/K(+)-ATPase. , 2000, Journal of neurophysiology.
[30] M. Devor,et al. Simulation in sensory neurons reveals a key role for delayed Na+ current in subthreshold oscillations and ectopic discharge: implications for neuropathic pain. , 2009, Journal of neurophysiology.
[31] R. MacKinnon,et al. Voltage-dependent K+ channel gating and voltage sensor toxin sensitivity depend on the mechanical state of the lipid membrane , 2008, Proceedings of the National Academy of Sciences.
[32] Riyi Shi 史日异,et al. Potassium channel blockers as an effective treatment to restore impulse conduction in injured axons , 2011, Neuroscience Bulletin.
[33] Eugene M. Izhikevich,et al. Resonate-and-fire neurons , 2001, Neural Networks.
[34] C. Morris. Chapter 27 – Why are So Many Ion Channels Mechanosensitive? , 2011 .
[35] Andrew J Powell,et al. Molecular cloning, distribution and functional analysis of the NA(V)1.6. Voltage-gated sodium channel from human brain. , 2002, Brain research. Molecular brain research.
[36] K. Monastyrskaya,et al. Plasma membrane repair and cellular damage control: the annexin survival kit. , 2011, Biochemical pharmacology.
[37] K. Sakurai,et al. Neuropathic pain is associated with increased nodal persistent Na+ currents in human diabetic neuropathy , 2009, Journal of the peripheral nervous system : JPNS.
[38] U. Ruegg,et al. Nav1.4 Deregulation in Dystrophic Skeletal Muscle Leads to Na+ Overload and Enhanced Cell Death , 2008, The Journal of general physiology.
[39] K. Fried,et al. The paradox of pain from tooth pulp: Low-threshold “algoneurons”? , 2011, PAIN.
[40] David C Viano,et al. Concussion in Professional Football: Comparison with Boxing Head Impacts—Part 10 , 2005, Neurosurgery.
[41] Peter Hänggi,et al. Noise-assisted spike propagation in myelinated neurons. , 2008, Physical review. E, Statistical, nonlinear, and soft matter physics.
[42] J. Bazarian,et al. Traumatic alterations in consciousness: traumatic brain injury. , 2010, Emergency medicine clinics of North America.
[43] W. Maxwell. Histopathological changes at central nodes of Ranvier after stretch‐injury , 1996, Microscopy research and technique.
[44] B. Bean,et al. Subthreshold Sodium Current from Rapidly Inactivating Sodium Channels Drives Spontaneous Firing of Tuberomammillary Neurons , 2002, Neuron.
[45] Fudong Liu,et al. Disruption of the Axon Initial Segment Cytoskeleton Is a New Mechanism for Neuronal Injury , 2009, The Journal of Neuroscience.
[46] Wei Wu,et al. Real-Time CARS Imaging Reveals a Calpain-Dependent Pathway for Paranodal Myelin Retraction during High-Frequency Stimulation , 2011, PloS one.
[47] A. Hodgkin,et al. A quantitative description of membrane current and its application to conduction and excitation in nerve , 1952, The Journal of physiology.
[48] Amandine Duflocq,et al. Characterization of the axon initial segment (AIS) of motor neurons and identification of a para-AIS and a juxtapara-AIS, organized by protein 4.1B , 2011, BMC Biology.
[49] Douglas H. Smith,et al. Sodium channelopathy induced by mild axonal trauma worsens outcome after a repeat injury , 2009, Journal of neuroscience research.
[50] Yuanzheng Gu,et al. Clustering and Activity Tuning of Kv1 Channels in Myelinated Hippocampal Axons* , 2011, The Journal of Biological Chemistry.
[51] J. Rothwell,et al. Latent addition in motor and sensory fibres of human peripheral nerve. , 1997, The Journal of physiology.
[52] C. Morris,et al. Membrane trauma and Na+ leak from Nav1.6 channels. , 2009, American journal of physiology. Cell physiology.
[53] Guillaume Charras,et al. Blebs lead the way: how to migrate without lamellipodia , 2008, Nature Reviews Molecular Cell Biology.
[54] Steven A Prescott,et al. Explaining pathological changes in axonal excitability through dynamical analysis of conductance-based models , 2011, Journal of neural engineering.
[55] C. Morris. Pacemaker, potassium, calcium, sodium: stretch modulation of the voltage-gated channels , 2011 .
[56] H. Sullivan. Electrogenic Ion Pumps (Distinguished Lecture Series of the Society of General Physiologists, Vol. 5) , 1992, Neurology.
[57] D. Meaney,et al. Diffuse Axonal Injury in Head Trauma , 2003, The Journal of head trauma rehabilitation.
[58] W. Wadman,et al. Kinetic changes and modulation by carbamazepine on voltage-gated sodium channels in rat CA1 neurons after epilepsy , 2006, Acta Pharmacologica Sinica.
[59] C. Morris,et al. Modulation of KvAP unitary conductance and gating by 1-alkanols and other surface active agents. , 2010, Biophysical Journal.
[60] R. Bullock,et al. Mechanical injury alters volume activated ion channels in cortical astrocytes. , 2000, Acta neurochirurgica. Supplement.
[61] P. Stys. White matter injury mechanisms. , 2004, Current molecular medicine.
[62] S G Waxman,et al. Noninactivating, tetrodotoxin-sensitive Na+ conductance in rat optic nerve axons. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[63] S. Waxman,et al. Noninactivating, tetrodotoxin‐sensitive Na+ conductance in peripheral axons , 2003, Muscle & nerve.
[64] Fredrik Elinder,et al. Electrostatic tuning of cellular excitability. , 2010, Biophysical journal.
[65] F. Roberge,et al. Modeling the dynamic features of the electrogenic Na,K pump of cardiac cells. , 1992, Journal of theoretical biology.
[66] H E M. Journal of Neurophysiology , 1938, Nature.
[67] David L. Worcester,et al. Structure and hydration of membranes embedded with voltage-sensing domains , 2009, Nature.
[68] M. Kiernan,et al. Upregulation of persistent sodium conductances in familial ALS , 2009, Journal of Neurology, Neurosurgery & Psychiatry.
[69] S. Kuwabara,et al. Latent addition in human motor and sensory axons: Different site-dependent changes across the carpal tunnel related to persistent Na+ currents , 2006, Clinical Neurophysiology.
[70] J. Wolf,et al. Traumatic Axonal Injury Induces Calcium Influx Modulated by Tetrodotoxin-Sensitive Sodium Channels , 2001, The Journal of Neuroscience.
[71] H. Kimelberg. Volume activated anion channel and astrocytic cellular edema in traumatic brain injury and stroke. , 2004, Advances in experimental medicine and biology.
[72] Tania Hanekom,et al. Modelled temperature-dependent excitability behaviour of a single ranvier node for a human peripheral sensory nerve fibre , 2009, Biological Cybernetics.