LncRNA HOTAIR down-expression inhibits the invasion and tumorigenicity of epithelial ovarian cancer cells by suppressing TGF-β1 and ZEB1

[1]  G. Nagaraju,et al.  HOTAIR: a potential metastatic, drug-resistant and prognostic regulator of breast cancer , 2023, Molecular Cancer.

[2]  Howard Y. Chang,et al.  Inducible lncRNA transgenic mice reveal continual role of HOTAIR in promoting breast cancer metastasis , 2022, eLife.

[3]  Lixia Xiong,et al.  HOTAIR/miR-203/CAV1 Crosstalk Influences Proliferation, Migration, and Invasion in the Breast Cancer Cell , 2022, International journal of molecular sciences.

[4]  Jiajia Wang,et al.  Long noncoding RNA HOTAIR regulates the stemness of breast cancer cells via activation of the NF-κB signaling pathway , 2022, The Journal of biological chemistry.

[5]  Dongmei Zhang,et al.  LncRNA-Dependent Mechanisms of Transforming Growth Factor-β: From Tissue Fibrosis to Cancer Progression , 2022, Non-coding RNA.

[6]  M. Miyoshi,et al.  Pancreatic cancer cell-derived exosomes induce epithelial-mesenchymal transition in human pancreatic cancer cells themselves partially via transforming growth factor β1 , 2022, Medical Molecular Morphology.

[7]  M. Shahin,et al.  Surgery in Advanced Ovary Cancer: Primary versus Interval Cytoreduction , 2022, Diagnostics.

[8]  Y. Niida,et al.  A comparison of mRNA sequencing (RNA-Seq) library preparation methods for transcriptome analysis , 2021, BMC genomics.

[9]  R. Jaksik,et al.  RNA-seq library preparation for comprehensive transcriptome analysis in cancer cells. , 2021, Genomics.

[10]  U. Demirci,et al.  Malignant Ascites in Ovarian Cancer: Cellular, Acellular, and Biophysical Determinants of Molecular Characteristics and Therapy Response , 2021, Cancers.

[11]  Shuling Guo,et al.  HOTAIR lncRNA promotes epithelial–mesenchymal transition by redistributing LSD1 at regulatory chromatin regions , 2021, EMBO reports.

[12]  G. Botti,et al.  Functional Interaction among lncRNA HOTAIR and MicroRNAs in Cancer and Other Human Diseases , 2021, Cancers.

[13]  Christian Schmidl,et al.  Genome‐wide cooperation of EMT transcription factor ZEB1 with YAP and AP‐1 in breast cancer , 2020, The EMBO journal.

[14]  Dehua Wu,et al.  HOTAIR expands the population of prostatic cancer stem-like cells and causes Docetaxel resistance via activating STAT3 signaling , 2020, Aging.

[15]  Taejung Kim,et al.  Ginsenosides Rk1 and Rg5 inhibit transforming growth factor-β1-induced epithelial-mesenchymal transition and suppress migration, invasion, anoikis resistance, and development of stem-like features in lung cancer , 2020, Journal of ginseng research.

[16]  Cuiping Yang,et al.  miR‐200c overexpression inhibits the invasion and tumorigenicity of epithelial ovarian cancer cells by suppressing lncRNA HOTAIR in mice , 2020, Journal of cellular biochemistry.

[17]  G. Wei,et al.  LncRNA HOTAIR regulates the expression of E-cadherin to affect nasopharyngeal carcinoma progression by recruiting histone methylase EZH2 to mediate H3K27 trimethylation. , 2019, Genomics.

[18]  C.-C. Wei,et al.  LncRNA HOTAIR promotes colon cancer development by down-regulating miRNA-34a. , 2019, European review for medical and pharmacological sciences.

[19]  I. Bure,et al.  Roles of E-cadherin and Noncoding RNAs in the Epithelial–mesenchymal Transition and Progression in Gastric Cancer , 2019, International journal of molecular sciences.

[20]  Chunfang Wang,et al.  Long non-coding RNA HOTAIR promotes cervical cancer progression through regulating BCL2 via targeting miR-143-3p , 2018, Cancer biology & therapy.

[21]  Mei Mei,et al.  Paracrine and epigenetic control of CAF-induced metastasis: the role of HOTAIR stimulated by TGF-ß1 secretion , 2018, Molecular Cancer.

[22]  Fan Yang,et al.  Missing Links in Epithelial-Mesenchymal Transition: Long Non-Coding RNAs Enter the Arena , 2017, Cellular Physiology and Biochemistry.

[23]  J. Dou,et al.  Downregulated lincRNA HOTAIR expression in ovarian cancer stem cells decreases its tumorgeniesis and metastasis by inhibiting epithelial-mesenchymal transition , 2015, Cancer Cell International.

[24]  J. Dou,et al.  MicroRNA-200c overexpression inhibits tumorigenicity and metastasis of CD117+CD44+ ovarian cancer stem cells by regulating epithelial-mesenchymal transition , 2013, Journal of Ovarian Research.