Necroptosis suppresses inflammation via termination of TNF- or LPS-induced cytokine and chemokine production
暂无分享,去创建一个
S. Cullen | C. Kearney | S. Martin | E. Lavelle | C. M. Henry | D. Clancy | S J Martin | E C Lavelle | Sean P. Cullen | G. A. Tynan | C J Kearney | S P Cullen | G A Tynan | C M Henry | D Clancy | Danielle M. Clancy | S. Martin | Conor M. Henry | Graham A. Tynan | Conor J. Kearney
[1] R. Dudani,et al. Type I interferon induces necroptosis in macrophages during infection with Salmonella enterica serovar Typhimurium , 2012, Nature Immunology.
[2] P. Matzinger. Tolerance, danger, and the extended family. , 1994, Annual review of immunology.
[3] N. D. Di Paolo,et al. The transcription factor IRF3 triggers "defensive suicide" necrosis in response to viral and bacterial pathogens. , 2013, Cell reports.
[4] Iain B McInnes,et al. Evidence that cytokines play a role in rheumatoid arthritis. , 2008, The Journal of clinical investigation.
[5] W. Kaiser,et al. Virus inhibition of RIP3-dependent necrosis. , 2010, Cell host & microbe.
[6] M. Hickey,et al. Perivascular macrophages mediate neutrophil recruitment during bacterial skin infection , 2013, Nature Immunology.
[7] Grace Y Chen,et al. Sterile inflammation: sensing and reacting to damage , 2010, Nature Reviews Immunology.
[8] V. Dixit,et al. Death receptors: signaling and modulation. , 1998, Science.
[9] F. Chan,et al. Phosphorylation-Driven Assembly of the RIP1-RIP3 Complex Regulates Programmed Necrosis and Virus-Induced Inflammation , 2009, Cell.
[10] J. Tschopp,et al. Induction of TNF Receptor I-Mediated Apoptosis via Two Sequential Signaling Complexes , 2003, Cell.
[11] David Baltimore,et al. An Essential Role for NF-κB in Preventing TNF-α-Induced Cell Death , 1996, Science.
[12] D. Lawrence,et al. A specific role of integrin Mac-1 in accelerated macrophage efflux to the lymphatics. , 2005, Blood.
[13] D. Baltimore,et al. An essential role for NF-kappaB in preventing TNF-alpha-induced cell death. , 1996, Science.
[14] Xiaodong Wang,et al. TNF-α Induces Two Distinct Caspase-8 Activation Pathways , 2008, Cell.
[15] Vinay Tergaonkar,et al. IAP antagonists target cIAP1 to induce TNFalpha-dependent apoptosis. , 2007, Cell.
[16] William J. Kaiser,et al. Viral infection and the evolution of caspase 8-regulated apoptotic and necrotic death pathways , 2011, Nature Reviews Immunology.
[17] T. Vanden Berghe,et al. RIP kinase-dependent necrosis drives lethal systemic inflammatory response syndrome. , 2011, Immunity.
[18] T. Vanden Berghe,et al. Many stimuli pull the necrotic trigger, an overview , 2011, Cell Death and Differentiation.
[19] G. Sutter,et al. Serp2, an Inhibitor of the Interleukin-1β-Converting Enzyme, Is Critical in the Pathobiology of Myxoma Virus , 1998, Journal of Virology.
[20] Tao Wang,et al. Receptor Interacting Protein Kinase-3 Determines Cellular Necrotic Response to TNF-α , 2009, Cell.
[21] U. V. von Andrian,et al. Chemokines in innate and adaptive host defense: basic chemokinese grammar for immune cells. , 2004, Annual review of immunology.
[22] D. Green,et al. Dichotomy between RIP1- and RIP3-Mediated Necroptosis in Tumor Necrosis Factor-α-Induced Shock , 2012, Molecular medicine.
[23] S. Nedospasov,et al. Tumor Necrosis Factor-induced Nonapoptotic Cell Death Requires Receptor-interacting Protein-mediated Cellular Reactive Oxygen Species Accumulation* , 2004, Journal of Biological Chemistry.
[24] The nature and action of granulocyte-macrophage colony stimulating factors. , 1980 .
[25] L. Herzenberg,et al. Division and differentiation of natural antibody-producing cells in mouse spleen , 2007, Proceedings of the National Academy of Sciences.
[26] F. Chan,et al. RIP3: a molecular switch for necrosis and inflammation. , 2013, Genes & development.
[27] Guy S. Salvesen,et al. Catalytic activity of the caspase-8-FLIPL complex inhibits RIPK3-dependent necrosis , 2011, Nature.
[28] David L. Vaux,et al. IAP Antagonists Target cIAP1 to Induce TNFα-Dependent Apoptosis , 2007, Cell.
[29] W. Junger,et al. Circulating Mitochondrial DAMPs Cause Inflammatory Responses to Injury , 2009, Nature.
[30] Yuqiong Liang,et al. Toll-like receptors activate programmed necrosis in macrophages through a receptor-interacting kinase-3–mediated pathway , 2011, Proceedings of the National Academy of Sciences.
[31] L. Moldawer,et al. The role of the cowpox virus crmA gene during intratracheal and intradermal infection of C57BL/6 mice. , 2009, Virology.
[32] L. Beck,et al. Chemokines: key players in innate and adaptive immunity. , 2005, The Journal of investigative dermatology.
[33] K. Rock,et al. How dying cells alert the immune system to danger , 2008, Nature Reviews Immunology.
[34] D. Lauffenburger,et al. The Response of Human Epithelial Cells to TNF Involves an Inducible Autocrine Cascade , 2006, Cell.
[35] D. Green,et al. Survival function of the FADD-CASPASE-8-cFLIP(L) complex. , 2012, Cell reports.
[36] Peter Vandenabeele,et al. Necroptosis: the release of damage-associated molecular patterns and its physiological relevance. , 2013, Immunity.
[37] Seamus J. Martin,et al. Suppression of TNF-α-Induced Apoptosis by NF-κB , 1996, Science.
[38] V. Dixit,et al. RIP3, a Novel Apoptosis-inducing Kinase* , 1999, The Journal of Biological Chemistry.
[39] W. Fiers,et al. Inhibition of Caspases Increases the Sensitivity of L929 Cells to Necrosis Mediated by Tumor Necrosis Factor , 1998, The Journal of experimental medicine.
[40] R. Hakem,et al. RIP3 mediates the embryonic lethality of caspase-8-deficient mice , 2011, Nature.