Aberrant methylation of GTPase regulator associated with the focal adhesion kinase (GRAF) promoter is an adverse prognostic factor in myelodysplastic syndrome
暂无分享,去创建一个
Qin Chen | J. Qian | Jian-yong Li | Jiang Lin | Zhao-hui Zhu | Dong-ming Yao | Gao-fei Xiao | Z. Qian | Dong‐ming Yao | Zhen Qian
[1] C. O'keefe,et al. Aberrant DNA methylation is a dominant mechanism in MDS progression to AML. , 2009, Blood.
[2] J. Boultwood,et al. Gene silencing by DNA methylation in haematological malignancies , 2007, British journal of haematology.
[3] S. Bojesen,et al. Characterisation of the GRAF gene promoter and its methylation in patients with acute myeloid leukaemia and myelodysplastic syndrome , 2006, British Journal of Cancer.
[4] N. Harris,et al. The World Health Organization (WHO) classification of the myeloid neoplasms. , 2002, Blood.
[5] E. Sahai,et al. Cross‐talk between Ras and Rho signalling pathways in transformation favours proliferation and increased motility , 2001, The EMBO journal.
[6] U. Jaeger,et al. The human GRAF gene is fused to MLL in a unique t(5;11)(q31;q23) and both alleles are disrupted in three cases of myelodysplastic syndrome/acute myeloid leukemia with a deletion 5q. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[7] J. Taylor,et al. An SH3 domain-containing GTPase-activating protein for Rho and Cdc42 associates with focal adhesion kinase , 1996, Molecular and cellular biology.