TOLLIP gene variant is associated with Plasmodium vivax malaria in the Brazilian Amazon

[1]  Q. Bassat,et al.  Declining malaria transmission in rural Amazon: changing epidemiology and challenges to achieve elimination , 2016, Malaria Journal.

[2]  Richard D. Wells,et al.  Genetic Variation in Toll-Interacting Protein Is Associated With Leprosy Susceptibility and Cutaneous Expression of Interleukin 1 Receptor Antagonist. , 2016, The Journal of infectious diseases.

[3]  S. Talhari,et al.  Polymorphisms in the TOLLIP Gene Influence Susceptibility to Cutaneous Leishmaniasis Caused by Leishmania guyanensis in the Amazonas State of Brazil , 2015, PLoS neglected tropical diseases.

[4]  A. Debrabant,et al.  Aptamer-Based Detection of Disease Biomarkers in Mouse Models for Chagas Drug Discovery , 2015, PLoS neglected tropical diseases.

[5]  J. Muñóz-Valle,et al.  Role of Toll-Interacting Protein Gene Polymorphisms in Leprosy Mexican Patients , 2013, BioMed research international.

[6]  M. U. Ferreira,et al.  Cytokine Balance in Human Malaria: Does Plasmodium vivax Elicit More Inflammatory Responses than Plasmodium falciparum? , 2012, PloS one.

[7]  J. Farrar,et al.  Human TOLLIP Regulates TLR2 and TLR4 Signaling and Its Polymorphisms Are Associated with Susceptibility to Tuberculosis , 2012, The Journal of Immunology.

[8]  M. Póvoa,et al.  Increased interleukin-10 and interferon-γ levels in Plasmodium vivax malaria suggest a reciprocal regulation which is not altered by IL-10 gene promoter polymorphism , 2011, Malaria Journal.

[9]  M. Lacerda,et al.  Malaria in Brazil: an overview , 2010, Malaria Journal.

[10]  D. Hansen,et al.  Natural Regulatory T Cells in Malaria: Host or Parasite Allies? , 2010, PLoS pathogens.

[11]  B. Andrade,et al.  Severe Plasmodium vivax malaria exhibits marked inflammatory imbalance , 2010, Malaria Journal.

[12]  J. Baird,et al.  Key gaps in the knowledge of Plasmodium vivax, a neglected human malaria parasite. , 2009, The Lancet. Infectious diseases.

[13]  E. Riley,et al.  IL-10: The Master Regulator of Immunity to Infection , 2008, The Journal of Immunology.

[14]  I. Clark,et al.  Human malarial disease: a consequence of inflammatory cytokine release , 2006, Malaria Journal.

[15]  T. Hawn,et al.  Towards subtlety: understanding the role of Toll-like receptor signaling in susceptibility to human infections. , 2006, Clinical immunology.

[16]  Z. Şimşek,et al.  Parasite density and serum cytokine levels in Plasmodium vivax malaria in Turkey , 2006, Parasite immunology.

[17]  S. Akira,et al.  Induction of Proinflammatory Responses in Macrophages by the Glycosylphosphatidylinositols of Plasmodium falciparum , 2005, Journal of Biological Chemistry.

[18]  R. Schumann,et al.  Single nucleotide polymorphisms of Toll-like receptors and susceptibility to infectious disease. , 2005, The Lancet. Infectious diseases.

[19]  O. Doumbo,et al.  Serum Levels of the Proinflammatory Cytokines Interleukin-1 Beta (IL-1β), IL-6, IL-8, IL-10, Tumor Necrosis Factor Alpha, and IL-12(p70) in Malian Children with Severe Plasmodium falciparum Malaria and Matched Uncomplicated Malaria or Healthy Controls , 2004, Infection and Immunity.

[20]  G. Snounou,et al.  Development of a Real-Time PCR Assay for Detection of Plasmodium falciparum, Plasmodium vivax, and Plasmodium ovale for Routine Clinical Diagnosis , 2004, Journal of Clinical Microbiology.

[21]  K. Mendis,et al.  The paroxysm of Plasmodium vivax malaria. , 2003, Trends in parasitology.

[22]  P. Seeberger,et al.  Synthetic GPI as a candidate anti-toxic vaccine in a model of malaria , 2002, Nature.

[23]  Sankar Ghosh,et al.  Negative Regulation of Toll-like Receptor-mediated Signaling by Tollip* , 2002, The Journal of Biological Chemistry.

[24]  F. Martinon,et al.  Tollip, a new component of the IL-1RI pathway, links IRAK to the IL-1 receptor , 2000, Nature Cell Biology.

[25]  N. Day,et al.  The prognostic and pathophysiologic role of pro- and antiinflammatory cytokines in severe malaria. , 1999, The Journal of infectious diseases.

[26]  P. Gerold,et al.  Glycosylphosphatidylinositol toxin of Plasmodium up-regulates intercellular adhesion molecule-1, vascular cell adhesion molecule-1, and E-selectin expression in vascular endothelial cells and increases leukocyte and parasite cytoadherence via tyrosine kinase-dependent signal transduction. , 1996, Journal of immunology.

[27]  J. Banchereau,et al.  High levels of circulating IL‐10 in human malaria , 1994, Clinical and experimental immunology.

[28]  D. Kwiatkowski,et al.  Tumour necrosis factor‐dependent parasite‐killing effects during paroxysms in non‐immune Plasmodium vivax malaria patients , 1992, Clinical and experimental immunology.