Expression of kidney injury molecule-1 (Kim-1) in relation to necrosis and apoptosis during the early stages of Cd-induced proximal tubule injury.
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Jie Liu | J. Bonventre | V. Vaidya | W. Prozialeck | P. Lamar | J. Edwards
[1] Gunnar F. Nordberg,et al. Cadmium and Health: A Toxicological and Epidemiological Appraisal , 2019 .
[2] Wing-Kee Lee,et al. Novel roles for ceramides, calpains and caspases in kidney proximal tubule cell apoptosis: lessons from in vitro cadmium toxicity studies. , 2008, Biochemical pharmacology.
[3] Joseph V Bonventre,et al. Kidney injury molecule-1 is a phosphatidylserine receptor that confers a phagocytic phenotype on epithelial cells. , 2008, The Journal of clinical investigation.
[4] Joseph V Bonventre,et al. Biomarkers of nephrotoxic acute kidney injury. , 2008, Toxicology.
[5] J. Bonventre,et al. Biomarkers of acute kidney injury. , 2008, Annual review of pharmacology and toxicology.
[6] J. Bonventre,et al. Kidney injury molecule-1 is an early biomarker of cadmium nephrotoxicity. , 2007, Kidney international.
[7] I. Sabolić,et al. Common Mechanisms in Nephropathy Induced by Toxic Metals , 2006, Nephron Physiology.
[8] D. Templeton,et al. Cadmium inhibits both intrinsic and extrinsic apoptotic pathways in renal mesangial cells. , 2006, American journal of physiology. Renal physiology.
[9] Joseph V Bonventre,et al. Urinary kidney injury molecule-1: a sensitive quantitative biomarker for early detection of kidney tubular injury. , 2006, American journal of physiology. Renal physiology.
[10] Rudolfs K. Zalups,et al. Molecular and ionic mimicry and the transport of toxic metals. , 2005, Toxicology and applied pharmacology.
[11] J. Bonventre,et al. Biologic markers for the early detection of acute kidney injury , 2004, Current opinion in critical care.
[12] A. Bernard. Renal dysfunction induced by cadmium: biomarkers of critical effects , 2004, Biometals.
[13] R. Tennant,et al. Identification of putative gene based markers of renal toxicity. , 2004, Environmental health perspectives.
[14] Z. Endre,et al. Cell death in toxic nephropathies. , 2003, Seminars in nephrology.
[15] S. Lynch,et al. Cadmium alters the localization of N-cadherin, E-cadherin, and beta-catenin in the proximal tubule epithelium. , 2003, Toxicology and applied pharmacology.
[16] T. Aoyagi,et al. Cadmium nephrotoxicity and evacuation from the body in a rat modeled subchronic intoxication , 2003, International journal of urology : official journal of the Japanese Urological Association.
[17] B. Padanilam. Cell death induced by acute renal injury: a perspective on the contributions of apoptosis and necrosis. , 2003, American journal of physiology. Renal physiology.
[18] F. Thévenod. Nephrotoxicity and the Proximal Tubule , 2003, Nephron Physiology.
[19] K. Zwierz,et al. Changes in the structure and function of the kidney of rats chronically exposed to cadmium. I. Biochemical and histopathological studies , 2003, Archives of Toxicology.
[20] J. Bonventre,et al. Shedding of Kidney Injury Molecule-1, a Putative Adhesion Protein Involved in Renal Regeneration* , 2002, The Journal of Biological Chemistry.
[21] Z. Shaikh,et al. Oxidative stress as a mechanism of chronic cadmium-induced hepatotoxicity and renal toxicity and protection by antioxidants. , 1999, Toxicology and applied pharmacology.
[22] Yuan Zhang,et al. The Human Homolog of HAVcr-1 Codes for a Hepatitis A Virus Cellular Receptor , 1998, Journal of Virology.
[23] E. Kharasch,et al. Role of the renal cysteine conjugate beta-lyase pathway in inhaled compound A nephrotoxicity in rats. , 1998, Anesthesiology.
[24] P. Mueller,et al. New approaches for detecting thresholds of human nephrotoxicity using cadmium as an example. , 1998, Environmental health perspectives.
[25] L. Teppo,et al. Health effects of cadmium exposure - a review of the literature and a risk estimate , 1998 .
[26] J. Johnson,et al. Cadmium-induced apoptosis in the urogenital organs of the male rat and its suppression by chelation. , 1997, Journal of toxicology and environmental health.
[27] S. Kimura,et al. Mechanism of nephrotoxicity induced by repeated administration of cadmium chloride in rats. , 1996, Journal of toxicology and environmental health.
[28] R. Lauwerys,et al. Urinary protein 1 or Clara cell protein: a new sensitive marker of proximal tubular dysfunction. , 1994, Kidney international. Supplement.
[29] R. Nilsson,et al. Glutathione transferases in the urine: sensitive methods for detection of kidney damage induced by nephrotoxic agents in humans. , 1994, Environmental health perspectives.
[30] A. Tanimoto,et al. Cell Death and Regeneration of Renal Proximal Tubular Cells in Rats with Subchronic Cadmium Intoxication , 1993, Toxicologic pathology.
[31] A. Tanimoto,et al. Pathological Study on Beagles after Long-term Oral Administration of Cadmium , 1991, Toxicologic pathology.
[32] K. S. Subramanian,et al. Biological monitoring for occupational cadmium exposure: the urinary metallothionein. , 1990, Toxicology.
[33] C. Miller,et al. Non-metallothionein-bound cadmium in the pathogenesis of cadmium nephrotoxicity in the rat. , 1989, Toxicology and applied pharmacology.
[34] A. Vyskočil,et al. Assessment of urinary retinol-binding protein as an index of proximal tubular injury. , 1987, Clinical chemistry.
[35] C. Klaassen,et al. Cadmium-induced hepatic and renal injury in chronically exposed rats: likely role of hepatic cadmium-metallothionein in nephrotoxicity. , 1985, Toxicology and applied pharmacology.
[36] C. Viau,et al. Characterization of cadmium proteinuria in man and rat. , 1984, Environmental health perspectives.
[37] L. Friberg,et al. Cadmium and the kidney. , 1984, Environmental health perspectives.
[38] R. Gibey,et al. Predictive value of urinary N-acetyl-beta-D-glucosaminidase (NAG), alanine-aminopeptidase (AAP) and beta-2-microglobulin (beta 2M) in evaluating nephrotoxicity of gentamicin. , 1981, Clinica chimica acta; international journal of clinical chemistry.
[39] Y. Suzuki,et al. Cadmium metabolism and toxicity in rats after long-term subcutaneous administration. , 1980, Journal of toxicology and environmental health.
[40] J. Peereboom,et al. Influence of chronic Cd intoxication on the alkaline phosphatase activity of liver and kidney; biochemical, histochemical and histological investigations. , 1979, Toxicology.
[41] Ronald P. Brown,et al. Comparison of kidney injury molecule-1 and other nephrotoxicity biomarkers in urine and kidney following acute exposure to gentamicin, mercury, and chromium. , 2008, Toxicological sciences : an official journal of the Society of Toxicology.
[42] J. Bonventre,et al. Mineralocorticoid receptor blockade confers renoprotection in preexisting chronic cyclosporine nephrotoxicity. , 2007, American journal of physiology. Renal physiology.
[43] O. Barbier,et al. Zinc protects renal function during cadmium intoxication in the rat. , 2006, American journal of physiology. Renal physiology.
[44] B. Fauconneau,et al. Nephrotoxicity of gentamicin and vancomycin given alone and in combination as determined by enzymuria and cortical antibiotic levels in rats. , 1997, Renal failure.
[45] C. Klaassen,et al. Role of metallothionein in cadmium-induced hepatotoxicity and nephrotoxicity. , 1997, Drug metabolism reviews.
[46] M. Piscator. The Nephropathy of Chronic Cadmium Poisoning , 1986 .