A Myocardial Slice Culture Model Reveals Alpha-1A-Adrenergic Receptor Signaling in the Human Heart

[1]  B. Gerber,et al.  Histological Validation of measurement of diffuse interstitial myocardial fibrosis by myocardial extravascular volume fraction from Modified Look-Locker imaging (MOLLI) T1 mapping at 3 T , 2015, Journal of Cardiovascular Magnetic Resonance.

[2]  A. Vickers,et al.  Isoproterenol effects evaluated in heart slices of human and rat in comparison to rat heart in vivo. , 2014, Toxicology and applied pharmacology.

[3]  Jürgen Hescheler,et al.  Organotypic slice culture from human adult ventricular myocardium. , 2012, Cardiovascular research.

[4]  D. Nishimura,et al.  A molecular MRI probe to detect treatment of cardiac apoptosis in vivo , 2011, Magnetic resonance in medicine.

[5]  P. Simpson Where are the new drugs to treat heart failure? Introduction to the special issue on "key signaling molecules in hypertrophy and heart failure". , 2011, Journal of molecular and cellular cardiology.

[6]  Patrizia Camelliti,et al.  Adult human heart slices are a multicellular system suitable for electrophysiological and pharmacological studies. , 2011, Journal of molecular and cellular cardiology.

[7]  T. Carrel,et al.  S100A1 genetically targeted therapy reverses dysfunction of human failing cardiomyocytes. , 2011, Journal of the American College of Cardiology.

[8]  A. Baker,et al.  Distinctive ERK and p38 signaling in remote and infarcted myocardium during post‐MI remodeling in the mouse , 2010, Journal of cellular biochemistry.

[9]  B. Jensen,et al.  &agr;1-Adrenergic Receptor Subtypes in Nonfailing and Failing Human Myocardium , 2009, Circulation. Heart failure.

[10]  S. Douglas,et al.  Direct Inotropic Effects of Exogenous and Endogenous Urotensin-II: Divergent Actions in Failing and Nonfailing Human Myocardium , 2009, Circulation. Heart failure.

[11]  S. Douglas,et al.  Direct Inotropic Effects of Exogenous and Endogenous Urotensin-IICLINICAL PERSPECTIVE , 2009 .

[12]  C. D. Wright,et al.  An α1A-Adrenergic–Extracellular Signal-Regulated Kinase Survival Signaling Pathway in Cardiac Myocytes , 2007, Circulation.

[13]  Paul A Bottomley,et al.  ATP flux through creatine kinase in the normal, stressed, and failing human heart. , 2005, Proceedings of the National Academy of Sciences of the United States of America.

[14]  R. Starling,et al.  Human myocardial ATP content and in vivo contractile function , 1998, Molecular and Cellular Biochemistry.

[15]  K. Weber,et al.  Effects of angiotensin II and aldosterone on collagen gene expression and protein turnover in cardiac fibroblasts , 1996, Molecular and Cellular Biochemistry.

[16]  P. Karczewski,et al.  Protein phosphorylation in isolated trabeculae from nonfailing and failing human hearts , 2004, Molecular and Cellular Biochemistry.

[17]  Stefan Neubauer,et al.  Absolute concentrations of high-energy phosphate metabolites in normal, hypertrophied, and failing human myocardium measured noninvasively with (31)P-SLOOP magnetic resonance spectroscopy. , 2002, Journal of the American College of Cardiology.

[18]  P. Simpson,et al.  Knockout of the α1A/C-adrenergic receptor subtype: The α1A/C is expressed in resistance arteries and is required to maintain arterial blood pressure , 2002, Proceedings of the National Academy of Sciences of the United States of America.

[19]  N. Aiyar,et al.  Bucindolol Displays Intrinsic Sympathomimetic Activity in Human Myocardium , 2002, Circulation.

[20]  S. Matsuo,et al.  Mechanism of specific nuclear transport of adriamycin: the mode of nuclear translocation of adriamycin-proteasome complex. , 2001, Cancer research.

[21]  U. Schmidt,et al.  Restoration of contractile function in isolated cardiomyocytes from failing human hearts by gene transfer of SERCA2a. , 1999, Circulation.

[22]  S. Houser,et al.  The sarcoplasmic reticulum and the Na+/Ca2+ exchanger both contribute to the Ca2+ transient of failing human ventricular myocytes. , 1999, Circulation research.

[23]  S. Houser,et al.  Myocyte recovery after mechanical circulatory support in humans with end-stage heart failure. , 1998, Circulation.

[24]  M. Böhm,et al.  Endothelin receptors in the failing and nonfailing human heart. , 1998, Circulation.

[25]  A. J. Gandolfi,et al.  Precision-cut tissue slices: applications in pharmacology and toxicology. , 1995, Life sciences.

[26]  P A Poole-Wilson The dimensions of human cardiac myocytes; confusion caused by methodology and pathology. , 1995, Journal of molecular and cellular cardiology.

[27]  P. Simpson,et al.  Differentiation of Rat Myocytes in Single Cell Cultures with and without Proliferating Nonmyocardial Cells: Cross‐Striations, infrastructure, and Chronotropic Response to Isoproterenol , 1982, Circulation research.

[28]  T. Spray,et al.  Transmural gradient in high-energy phosphate content in patients with coronary artery disease. , 1981, The Annals of thoracic surgery.

[29]  G. Langer,et al.  THE MYOCARDIAL INTERSTITIUM: ITS STRUCTURE AND ITS ROLE IN IONIC EXCHANGE , 1974, The Journal of cell biology.

[30]  C. Chidsey,et al.  Biochemical studies of energy production in the failing human heart. , 1966, The Journal of clinical investigation.