Characterization of metabolic health in mouse models of fibrillin-1 perturbation.
暂无分享,去创建一个
[1] F. Ramirez,et al. Fibrillin microfibrils in bone physiology. , 2016, Matrix biology : journal of the International Society for Matrix Biology.
[2] S. Rane,et al. SMAD3 Negatively Regulates Serum Irisin and Skeletal Muscle FNDC5 and Peroxisome Proliferator-activated Receptor γ Coactivator 1-α (PGC-1α) during Exercise* , 2015, The Journal of Biological Chemistry.
[3] D. Cai,et al. Obesity- and aging-induced excess of central transforming growth factor-β potentiates diabetic development via an RNA stress response , 2014, Nature Medicine.
[4] S. Klein,et al. The Extracellular Matrix Protein MAGP1 Supports Thermogenesis and Protects Against Obesity and Diabetes Through Regulation of TGF-β , 2014, Diabetes.
[5] A. Zwinderman,et al. Circulating transforming growth factor-β as a prognostic biomarker in Marfan syndrome. , 2013, International journal of cardiology.
[6] R. Mecham,et al. Oophorectomy‐induced bone loss is attenuated in MAGP1‐deficient mice , 2012, Journal of cellular biochemistry.
[7] P. Sun,et al. Protection from obesity and diabetes by blockade of TGF-β/Smad3 signaling. , 2011, Cell metabolism.
[8] W. Wahli,et al. Smad3 Deficiency in Mice Protects Against Insulin Resistance and Obesity Induced by a High-Fat Diet , 2011, Diabetes.
[9] C. Baldock,et al. Assembly of fibrillin microfibrils governs extracellular deposition of latent TGFβ , 2010, Journal of Cell Science.
[10] D. Keene,et al. In Vivo Studies of Mutant Fibrillin-1 Microfibrils* , 2010, The Journal of Biological Chemistry.
[11] Justin S. Weinbaum,et al. Microfibril-associated Glycoprotein-1, an Extracellular Matrix Regulator of Bone Remodeling* , 2010, The Journal of Biological Chemistry.
[12] H. Dietz,et al. Circulating Transforming Growth Factor-&bgr; in Marfan Syndrome , 2009, Circulation.
[13] C. Meisinger,et al. Transforming growth factor-beta1 and incident type 2 diabetes: results from the MONICA/KORA case-cohort study, 1984-2002. , 2009, Diabetes care.
[14] Takako Sasaki,et al. Latent Transforming Growth Factor β-binding Proteins and Fibulins Compete for Fibrillin-1 and Exhibit Exquisite Specificities in Binding Sites* , 2009, The Journal of Biological Chemistry.
[15] Justin S. Weinbaum,et al. Deficiency in Microfibril-associated Glycoprotein-1 Leads to Complex Phenotypes in Multiple Organ Systems* , 2008, Journal of Biological Chemistry.
[16] Nicholas H. Putnam,et al. The Trichoplax genome and the nature of placozoans , 2008, Nature.
[17] L. Carta,et al. Fibrillin-Rich Microfibrils—Structural and Instructive Determinants of Mammalian Development and Physiology , 2008, Connective tissue research.
[18] Marc K. Halushka,et al. Losartan, an AT1 Antagonist, Prevents Aortic Aneurysm in a Mouse Model of Marfan Syndrome , 2006, Science.
[19] D. Keene,et al. Fibrillins 1 and 2 Perform Partially Overlapping Functions during Aortic Development* , 2006, Journal of Biological Chemistry.
[20] D. Judge,et al. TGF-β–dependent pathogenesis of mitral valve prolapse in a mouse model of Marfan syndrome , 2004 .
[21] Jessica Geubtner,et al. Evidence for a critical contribution of haploinsufficiency in the complex pathogenesis of Marfan syndrome. , 2004, The Journal of clinical investigation.
[22] D. Keene,et al. Differential expression of fibrillin-3 adds to microfibril variety in human and avian, but not rodent, connective tissues. , 2004, Genomics.
[23] D. Arking,et al. Dysregulation of TGF-β activation contributes to pathogenesis in Marfan syndrome , 2003, Nature Genetics.
[24] D. Rifkin,et al. Latent Transforming Growth Factor β-binding Protein 1 Interacts with Fibrillin and Is a Microfibril-associated Protein* , 2003, The Journal of Biological Chemistry.
[25] P. Morange,et al. Plasminogen activator inhibitor 1, transforming growth factor-beta1, and BMI are closely associated in human adipose tissue during morbid obesity. , 2000, Diabetes.
[26] M. Gibson,et al. Microfibril-associated Glycoprotein-1 (MAGP-1) Binds to the Pepsin-resistant Domain of the α3(VI) Chain of Type VI Collagen* , 1997, The Journal of Biological Chemistry.
[27] J. Spring,et al. An extracellular matrix protein of jellyfish homologous to mammalian fibrillins forms different fibrils depending on the life stage of the animal. , 1995, Developmental biology.
[28] W. Hu,et al. Developmental expression of fibrillin genes suggests heterogeneity of extracellular microfibrils , 1995, The Journal of cell biology.
[29] R. Mecham,et al. Structure and expression of fibrillin-2, a novel microfibrillar component preferentially located in elastic matrices , 1994, The Journal of cell biology.
[30] E. Engvall,et al. Fibrillin, a new 350-kD glycoprotein, is a component of extracellular microfibrils , 1986, The Journal of cell biology.
[31] K. Nath,et al. Marfan's syndrome. , 1959, Journal of the Indian Medical Association.
[32] D. Harlan,et al. Transforming Growth Factor-β/Smad3 Signaling Regulates Insulin Gene Transcription and Pancreatic Islet β-Cell Function* , 2009, Journal of Biological Chemistry.
[33] D. Judge,et al. TGF-beta-dependent pathogenesis of mitral valve prolapse in a mouse model of Marfan syndrome. , 2004, The Journal of clinical investigation.
[34] D. Arking,et al. Dysregulation of TGF-beta activation contributes to pathogenesis in Marfan syndrome. , 2003, Nature genetics.