Disturbed connexin43 gap junction distribution correlates with the location of reentrant circuits in the epicardial border zone of healing canine infarcts that cause ventricular tachycardia.
暂无分享,去创建一个
N S Peters | N. Peters | J. Coromilas | N. Severs | A. L. Wit | A L Wit | J Coromilas | N J Severs
[1] J. Francis Heidlage,et al. Influence of the Passive Anisotropic Properties on Directional Differences in Propagation Following Modification of the Sodium Conductance in Human Atrial Muscle: A Model of Reentry Based on Anisotropic Discontinuous Propagation , 1988, Circulation research.
[2] M S Spach,et al. Structure of canine Bachmann's bundle related to propagation of excitation. , 1989, The American journal of physiology.
[3] C. Green,et al. Altered patterns of gap junction distribution in ischemic heart disease. An immunohistochemical study of human myocardium using laser scanning confocal microscopy. , 1991, The American journal of pathology.
[4] N. El-Sherif,et al. Reentrant Ventricular Arrhythmias in the Late Myocardial Infarction Period 9. Electrophysiologic-Anatomic Correlation of Reentrant Circuits , 1983, Circulation.
[5] A. L. Wit,et al. Action potentials of cardiac muscle in healing infarcts: response to norepinephrine and caffeine. , 1988, Journal of molecular and cellular cardiology.
[6] J. Spear,et al. Effects of Cellular Uncoupling on Conduction in Anisotropic Canine Ventricular Myocardium , 1988, Circulation research.
[7] M. Yacoub,et al. Spatiotemporal Relation Between Gap Junctions and Fascia Adherens Junctions During Postnatal Development of Human Ventricular Myocardium , 1994, Circulation.
[8] R. W. Joyner,et al. Effects of the Discrete Pattern of Electrical Coupling on Propagation through an Electrical Syncytium , 1982, Circulation research.
[9] P. Ursell,et al. Electrophysiologic and anatomic basis for fractionated electrograms recorded from healed myocardial infarcts. , 1985, Circulation.
[10] J E Saffitz,et al. Remodeling of ventricular conduction pathways in healed canine infarct border zones. , 1991, The Journal of clinical investigation.
[11] J E Saffitz,et al. Alterations of intercellular junctions induced by hypoxia in canine myocardium. , 1990, The American journal of physiology.
[12] J. Saffitz,et al. Cardiac myocytes express multiple gap junction proteins. , 1992, Circulation research.
[13] P. Poole‐Wilson,et al. Reduced content of connexin43 gap junctions in ventricular myocardium from hypertrophied and ischemic human hearts. , 1993, Circulation.
[14] M. Allessie,et al. Influences of anisotropic tissue structure on reentrant circuits in the epicardial border zone of subacute canine infarcts. , 1988, Circulation research.
[15] R. P. Thompson,et al. The spatial distribution and relative abundance of gap-junctional connexin40 and connexin43 correlate to functional properties of components of the cardiac atrioventricular conduction system. , 1993, Journal of cell science.
[16] N. Peters. Myocardial gap junction organization in ischemia and infarction , 1995, Microscopy research and technique.
[17] C. Green,et al. Gap junction distribution in adult mammalian myocardium revealed by an anti-peptide antibody and laser scanning confocal microscopy. , 1991, Journal of cell science.
[18] J. Spear,et al. Gap junctional conductance in ventricular myocyte pairs isolated from postischemic rabbit myocardium. , 1992, Circulation research.
[19] A. Nathan. The ventricular arrhythmias of ischemia and infarction: A.L. Wit and M.J. Janse Futura, Mount Kisco, NY, 1992; 648 pp.; US$150.00; ISBN: 0-87993-576-0 , 1993 .
[20] J E Saffitz,et al. Distribution and Three‐Dimensional Structure of Intercellular Junctions in Canine Myocardium , 1989, Circulation research.
[21] A. Kleber,et al. Electrical uncoupling and increase of extracellular resistance after induction of ischemia in isolated, arterially perfused rabbit papillary muscle. , 1987, Circulation research.
[22] D. Ganten,et al. Gap junction protein connexin40 is preferentially expressed in vascular endothelium and conductive bundles of rat myocardium and is increased under hypertensive conditions. , 1993, Circulation research.
[23] M. Allessie,et al. Electrophysiologic mapping to determine the mechanism of experimental ventricular tachycardia initiated by premature impulses. Experimental approach and initial results demonstrating reentrant excitation. , 1982, The American journal of cardiology.
[24] M. Rosen,et al. Lethal Arrhythmias Resulting from Myocardial Ischemia and Infarction , 2011, Developments in Cardiovascular Medicine.
[25] N. Severs,et al. Intercellular junctions and the application of microscopical techniques: the cardiac gap junction as a case model , 1993, Journal of microscopy.
[26] E. Haber,et al. The heart and cardiovascular system , 1986 .
[27] M S Spach,et al. The Functional Role of Structural Complexities in the Propagation of Depolarization in the Atrium of the Dog: Cardiac Conduction Disturbances Due to Discontinuities of Effective Axial Resistivity , 1982, Circulation research.
[28] P. Boyden,et al. Abnormal Electrical Properties of Myocytes From Chronically Infarcted Canine Heart: Alterations in &OV0312;max and the Transient Outward Current , 1992, Circulation.
[29] Y. Rudy,et al. Unidirectional block and reentry of cardiac excitation: a model study. , 1990, Circulation research.
[30] José Jalife,et al. Cardiac electrophysiology and arrhythmias , 1985 .
[31] J. Saffitz,et al. Multiple connexins colocalize in canine ventricular myocyte gap junctions. , 1993, Circulation research.
[32] J. Coromilas,et al. Electrophysiological effects of flecainide on anisotropic conduction and reentry in infarcted canine hearts. , 1995, Circulation.
[33] C. Green,et al. Gap junction connexon configuration in rapidly frozen myocardium and isolated intercalated disks , 1984, The Journal of cell biology.
[34] C. Green,et al. Cardiac myocyte gap junctions: evidence for a major connexon protein with an apparent relative molecular mass of 70,000. , 1990, Journal of cell science.
[35] C. Green,et al. Validation of immunohistochemical quantification in confocal scanning laser microscopy: a comparative assessment of gap junction size with confocal and ultrastructural techniques. , 1993, The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society.
[36] J. Wojtczak. Contractures and Increase in Internal Longitudinal Resistance of Cow Ventricular Muscle Induced by Hypoxia , 1979, Circulation research.
[37] P. Ursell,et al. Structural and Electrophysiological Changes in the Epicardial Border Zone of Canine Myocardial Infarcts during Infarct Healing , 1985, Circulation research.
[38] C. Green,et al. Cardiac gap junctions in rat ventricle: localization using site-directed antibodies and laser scanning confocal microscopy. , 1990, Cardioscience.
[39] J. Brachmann,et al. Mechanisms of spontaneous and induced ventricular arrhythmias in the 24-hour infarcted dog heart. , 1983, The American journal of cardiology.
[40] M Delmar,et al. Effects of increasing intercellular resistance on transverse and longitudinal propagation in sheep epicardial muscle. , 1987, Circulation research.
[41] C. Peracchia. Structural correlates of gap junction permeation. , 1980, International review of cytology.
[42] P. Cranefield. The conduction of the cardiac impulse : the slow response and cardiac arrhythmias , 1975 .
[43] M. Spach,et al. Relating Extracellular Potentials and Their Derivatives to Anisotropic Propagation at a Microscopic Level in Human Cardiac Muscle: Evidence for Electrical Uncoupling of Side‐to‐Side Fiber Connections with Increasing Age , 1986, Circulation research.
[44] A. L. Wit. Anisotropic Reentry: A Model of Arrhythmias that may Necessitate a New Approach to Antiarrhythmic Drug Development , 1989 .
[45] E. Harlow,et al. Antibodies: A Laboratory Manual , 1988 .