Effect of Short-Term Aspirin Use on C-Reactive Protein

[1]  J. Muller,et al.  Effect of aspirin dosage and enteric coating on platelet reactivity. , 1997, The American journal of cardiology.

[2]  P. Ridker,et al.  Inflammation, aspirin, and the risk of cardiovascular disease in apparently healthy men. , 1997, The New England journal of medicine.

[3]  C. Serhan.  Lipoxins and novel aspirin-triggered 15-epi-lipoxins (ATL): a jungle of cell-cell interactions or a therapeutic opportunity? , 1997, Prostaglandins.

[4]  S. Thompson,et al.  Hemostatic factors and the risk of myocardial infarction or sudden death in patients with angina pectoris. European Concerted Action on Thrombosis and Disabilities Angina Pectoris Study Group. , 1995, The New England journal of medicine.

[5]  T. Krusius,et al.  Short-term aspirin treatment does not reduce plasma fibrinogen concentration in young healthy adults. , 1994, Thrombosis research.

[6]  A. Rebuzzi,et al.  The prognostic value of C-reactive protein and serum amyloid a protein in severe unstable angina. , 1994, The New England journal of medicine.

[7]  V. Fuster,et al.  Macrophage Infiltration in Acute Coronary Syndromes: Implications for Plaque Rupture , 1994, Circulation.

[8]  A. Whitehead,et al.  The major acute phase reactants: C-reactive protein, serum amyloid P component and serum amyloid A protein. , 1994, Immunology today.

[9]  R. Goldberg,et al.  Triggering of acute myocardial infarction by heavy physical exertion. Protection against triggering by regular exertion. Determinants of Myocardial Infarction Onset Study Investigators. , 1993, The New England journal of medicine.

[10]  L. M. Weight,et al.  Strenuous exercise: analogous to the acute-phase response? , 1991, Clinical science.

[11]  A. Henney,et al.  Localization of stromelysin gene expression in atherosclerotic plaques by in situ hybridization. , 1991, Proceedings of the National Academy of Sciences of the United States of America.

[12]  J. Manson,et al.  Low-dose aspirin therapy for chronic stable angina. A randomized, placebo-controlled clinical trial. , 1991, Annals of internal medicine.

[13]  M. Davies,et al.  Atherosclerotic plaque caps are locally weakened when macrophages density is increased. , 1991, Atherosclerosis.

[14]  A. Eisen,et al.  Neutral metalloproteinases produced by human mononuclear phagocytes. Enzyme profile, regulation, and expression during cellular development. , 1990, The Journal of clinical investigation.

[15]  W. Weintraub,et al.  Elevation of C-reactive protein in "active" coronary artery disease. , 1990, The American journal of cardiology.

[16]  G. V. R. Born,et al.  INFLUENCE OF PLAQUE CONFIGURATION AND STRESS DISTRIBUTION ON FISSURING OF CORONARY ATHEROSCLEROTIC PLAQUES , 1989, The Lancet.

[17]  H. Baumann,et al.  IL-6 modulates the synthesis of a specific set of acute phase plasma proteins in vivo. , 1989, Journal of immunology.

[18]  B. Hazenberg,et al.  The effect of interleukin-1, interleukin-6 and its interrelationship on the synthesis of serum amyloid A and C-reactive protein in primary cultures of adult human hepatocytes. , 1988, Biochemical and biophysical research communications.

[19]  H. Gewurz,et al.  Stimulation of human neutrophils, monocytes, and platelets by modified C-reactive protein (CRP) expressing a neoantigenic specificity , 1988, Inflammation.

[20]  C. Hawkey,et al.  Effect in man of aspirin, standard indomethacin, and sustained release indomethacin preparations on gastric bleeding. , 1988, British journal of clinical pharmacology.

[21]  G. Rogers,et al.  Hematologic, iron-related, and acute-phase protein responses to sustained strenuous exercise. , 1987, Journal of applied physiology.

[22]  T. Lint,et al.  Enhancement of Human Peripheral Blood Monocyte Respiratory Burst Activity by Aggregated C‐Reactive Protein , 1986, Journal of leukocyte biology.

[23]  C. Savage,et al.  Objective monitoring of disease activity in polyarteritis by measurement of serum C reactive protein concentration. , 1984, British medical journal.

[24]  M. LeWinter,et al.  Protective effects of aspirin against acute myocardial infarction and death in men with unstable angina. Results of a Veterans Administration Cooperative Study. , 1983, The New England journal of medicine.

[25]  R. Parker,et al.  A study of the influence of various antirheumatic drug regimens on serum acute-phase proteins, plasma tryptophan, and erythrocyte sedimentation rate in rheumatoid arthritis. , 1975, Rheumatology and rehabilitation.

[26]  B E Statland,et al.  Factors contributing to intra-individual variation of serum constituents. 2. Effects of exercise and diet on variation of serum constituents in healthy subjects. , 1973, Clinical chemistry.

[27]  B E Statland,et al.  Factors contributing to intra-individual variation of serum constituents. 1. Within-day variation of serum constituents in healthy subjects. , 1973, Clinical chemistry.

[28]  B. McConkey,et al.  The effects of some anti-inflammatory drugs on the acute-phase proteins in rheumatoid arthritis. , 1973, The Quarterly journal of medicine.

[29]  G. Cumming,et al.  Effects of exercise in the cold on blood clotting and platelets. , 1965, Journal of applied physiology.

[30]  R. Tracy,et al.  Variability in the measurement of C-reactive protein in healthy subjects: implications for reference intervals and epidemiological applications. , 1997, Clinical chemistry.

[31]  A. Becker,et al.  Site of intimal rupture or erosion of thrombosed coronary atherosclerotic plaques is characterized by an inflammatory process irrespective of the dominant plaque morphology. , 1994, Circulation.

[32]  Harry,et al.  C-reactive protein induces human peripheral blood monocytes to synthesize tissue factor. , 1993, Blood.