Multi-pronged inhibition of airway hyper-responsiveness and inflammation by lipoxin A4
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Eugene Y. Kim | J. Drazen | B. Levy | C. Serhan | G. T. Sanctis | P. Devchand | K. Ackerman | Birgitta A Schmidt | W. Szczeklik | Birgitta A. Schmidt
[1] D. Corry,et al. The Th2 lymphocyte products IL-4 and IL-13 rapidly induce airway hyperresponsiveness through direct effects on resident airway cells. , 2002, American journal of respiratory cell and molecular biology.
[2] Ji Ming Wang,et al. The fibrinolytic receptor for urokinase activates the G protein-coupled chemotactic receptor FPRL1/LXA4R , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[3] A. Sher,et al. Lipoxin-mediated inhibition of IL-12 production by DCs: a mechanism for regulation of microbial immunity , 2002, Nature Immunology.
[4] S. Mitchell,et al. Lipoxins: revelations on resolution. , 2001, Trends in pharmacological sciences.
[5] J. Kinet,et al. Direct effects of interleukin-13 on signaling pathways for physiological responses in cultured human airway smooth muscle cells. , 2001, American journal of respiratory and critical care medicine.
[6] Charles N. Serhan,et al. Lipid mediator class switching during acute inflammation: signals in resolution , 2001, Nature Immunology.
[7] C. Serhan,et al. Selectivity of recombinant human leukotriene D(4), leukotriene B(4), and lipoxin A(4) receptors with aspirin-triggered 15-epi-LXA(4) and regulation of vascular and inflammatory responses. , 2001, The American journal of pathology.
[8] Stephen T Holgate,et al. Effects of recombinant human interleukin-12 on eosinophils, airway hyper-responsiveness, and the late asthmatic response , 2000, The Lancet.
[9] K. Chung,et al. Effects of an interleukin-5 blocking monoclonal antibody on eosinophils, airway hyper-responsìveness, and the late asthmatic response , 2000, The Lancet.
[10] M. Sanak,et al. Aspirin-tolerant asthmatics generate more lipoxins than aspirin-intolerant asthmatics. , 2000, The European respiratory journal.
[11] S. Hazen,et al. Eosinophils generate brominating oxidants in allergen-induced asthma. , 2000, The Journal of clinical investigation.
[12] J Bousquet,et al. Asthma. From bronchoconstriction to airways inflammation and remodeling. , 2000, American journal of respiratory and critical care medicine.
[13] C. Serhan,et al. Cutting Edge: Lipoxin (LX) A4 and Aspirin-Triggered 15-Epi-LXA4 Block Allergen-Induced Eosinophil Trafficking1 , 2000, The Journal of Immunology.
[14] Hugh R. Brady,et al. Cutting Edge: Lipoxins Rapidly Stimulate Nonphlogistic Phagocytosis of Apoptotic Neutrophils by Monocyte-Derived Macrophages1 , 2000, The Journal of Immunology.
[15] A. Leff. Role of leukotrienes in bronchial hyperresponsiveness and cellular responses in airways. , 2000, Thorax.
[16] C. Serhan,et al. Cyclooxygenase-2-Derived Prostaglandin E2 and Lipoxin A4 Accelerate Resolution of Allergic Edema in Angiostrongylus costaricensis-Infected Rats: Relationship with Concurrent Eosinophilia1 , 2000, The Journal of Immunology.
[17] E. Silverman,et al. Heterogeneity of therapeutic responses in asthma. , 2000, British medical bulletin.
[18] C. Clish,et al. Local and systemic delivery of a stable aspirin-triggered lipoxin prevents neutrophil recruitment in vivo. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[19] S. Holgate. The epidemic of allergy and asthma , 1999, Nature.
[20] E. Israel,et al. Treatment of asthma with drugs modifying the leukotriene pathway. , 1999, The New England journal of medicine.
[21] Y. Nasuhara,et al. Cysteinyl-leukotrienes partly mediate eotaxin-induced bronchial hyperresponsiveness and eosinophilia in IL-5 transgenic mice. , 1999, American journal of respiratory and critical care medicine.
[22] M. Freeman,et al. Leukotriene B4 receptor transgenic mice reveal novel protective roles for lipoxins and aspirin-triggered lipoxins in reperfusion. , 1999, The Journal of clinical investigation.
[23] S. Holgate,et al. IL-5 increases expression of 5-lipoxygenase-activating protein and translocates 5-lipoxygenase to the nucleus in human blood eosinophils. , 1999, Journal of immunology.
[24] J. Drazen,et al. Contribution of Nitric Oxide Synthases 1, 2, and 3 to Airway Hyperresponsiveness and Inflammation in a Murine Model of Asthma , 1999, The Journal of experimental medicine.
[25] J. Drazen,et al. Interleukin-8 receptor modulates IgE production and B-cell expansion and trafficking in allergen-induced pulmonary inflammation. , 1999, The Journal of clinical investigation.
[26] J. Drazen. Leukotrienes as mediators of airway obstruction. , 1998, American journal of respiratory and critical care medicine.
[27] T. Takano,et al. Aspirin-triggered 15-Epi-Lipoxin A4 (LXA4) and LXA4 Stable Analogues Are Potent Inhibitors of Acute Inflammation: Evidence for Anti-inflammatory Receptors , 1997, The Journal of experimental medicine.
[28] J. Haeggström,et al. Lipid mediator networks in cell signaling: update and impact of cytokines 1 , 1996, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[29] B. Spur,et al. Effects of lipoxin A4 on chemotaxis and degranulation of human eosinophils stimulated by platelet‐activating factor and N‐formyl‐L‐methionyl‐L‐leucyl‐L‐phenylalanine , 1994, Allergy.
[30] B. Spur,et al. The effects of lipoxin A4 on airway responses in asthmatic subjects. , 1992, The American review of respiratory disease.
[31] D. Broide,et al. Cytokines in symptomatic asthma airways. , 1992, The Journal of allergy and clinical immunology.
[32] S. Durham,et al. Predominant TH2-like bronchoalveolar T-lymphocyte population in atopic asthma. , 1992, The New England journal of medicine.
[33] V. Gant,et al. Identification of lipoxin A4 and its relationship to the sulfidopeptide leukotrienes C4, D4, and E4 in the bronchoalveolar lavage fluids obtained from patients with selected pulmonary diseases. , 1990, The American review of respiratory disease.
[34] K. Badr,et al. Lipoxin A4 antagonizes cellular and in vivo actions of leukotriene D4 in rat glomerular mesangial cells: evidence for competition at a common receptor. , 1989, Proceedings of the National Academy of Sciences of the United States of America.
[35] S. Dahlén,et al. Actions of lipoxin A4 and related compounds in smooth muscle preparations and on the microcirculation in vivo. , 1988, Advances in experimental medicine and biology.
[36] B. Samuelsson. FROM STUDIES OF BIOCHEMICAL MECHANISMS TO NEW BIOLOGICAL MEDIATORS: PROSTAGLANDIN ENDOPEROXIDES, THROMBOXANES, AND LEUKOTRIENES , 1984 .
[37] B. Samuelsson. From studies of biochemical mechanism to novel biological mediators: Prostaglandin endoperoxides, thromboxanes, and leukotriences , 1983 .